Neuronal apoptosis and motor deficits in mice with genetic inhibition of GSK-3 are Fas-dependent
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Isoflurane Damages the Developing Brain of Mice and Induces Subsequent Learning and Memory Deficits through FASL-FAS SignalingExtrinsic Apoptosis Pathway Altered by Glycogen Synthase Kinase-3β Inhibitor Influences the Net Drug Effect on NSC-34 Motor Neuron-Like Cell Survival.Decreased glycogen synthase kinase-3 levels and activity contribute to Huntington's disease.Bi-directional genetic modulation of GSK-3β exacerbates hippocampal neuropathology in experimental status epilepticus
P2860
Neuronal apoptosis and motor deficits in mice with genetic inhibition of GSK-3 are Fas-dependent
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2013 nî lūn-bûn
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2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
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2013年論文
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2013年論文
@zh-mo
2013年論文
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2013年论文
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Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@ast
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@en
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@nl
type
label
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@ast
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@en
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@nl
prefLabel
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@ast
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@en
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@nl
P2860
P1433
P1476
Neuronal apoptosis and motor d ...... ion of GSK-3 are Fas-dependent
@en
P2093
Raquel Gómez-Sintes
P2860
P304
P356
10.1371/JOURNAL.PONE.0070952
P577
2013-08-05T00:00:00Z