Inactivation of a two-component signal transduction system, SaeRS, eliminates adherence and attenuates virulence of Staphylococcus aureus.
about
Pathogenesis of methicillin-resistant Staphylococcus aureus infectionStaphylococcus aureus RNAIII coordinately represses the synthesis of virulence factors and the transcription regulator Rot by an antisense mechanismThe SaeRS Two-Component System of Staphylococcus aureusThe extracytoplasmic linker peptide of the sensor protein SaeS tunes the kinase activity required for staphylococcal virulence in response to host signalsRole of the SaeRS two-component regulatory system in Staphylococcus epidermidis autolysis and biofilm formationDifferential regulation of staphylococcal virulence by the sensor kinase SaeS in response to neutrophil-derived stimuli.Aureusimines in Staphylococcus aureus are not involved in virulence.Solution insights into the structure of the Efb/C3 complement inhibitory complex as revealed by lysine acetylation and mass spectrometry.The SaeR/S gene regulatory system is essential for innate immune evasion by Staphylococcus aureus.An in vivo reporter assay for sRNA-directed gene control in Gram-positive bacteria: identifying a novel sRNA target in Staphylococcus aureus.The SaeRS Two-Component System Controls Survival of Staphylococcus aureus in Human Blood through Regulation of Coagulase.In the Staphylococcus aureus two-component system sae, the response regulator SaeR binds to a direct repeat sequence and DNA binding requires phosphorylation by the sensor kinase SaeSImportance of the global regulators Agr and SaeRS in the pathogenesis of CA-MRSA USA300 infection.Identification of single nucleotide polymorphisms associated with hyperproduction of alpha-toxin in Staphylococcus aureus.The SaeR/S gene regulatory system induces a pro-inflammatory cytokine response during Staphylococcus aureus infection.Insights into alpha-hemolysin (Hla) evolution and expression among Staphylococcus aureus clones with hospital and community originGolden pigment production and virulence gene expression are affected by metabolisms in Staphylococcus aureusStaphylococcal superantigen-like genes, ssl5 and ssl8, are positively regulated by Sae and negatively by Agr in the Newman strain.Changes in the Staphylococcus aureus transcriptome during early adaptation to the lung.Factors contributing to epidemic MRSA clones replacement in a hospital setting.Staphylococcus aureus ClpC divergently regulates capsule via sae and codY in strain newman but activates capsule via codY in strain UAMS-1 and in strain Newman with repaired saeSThe essential yhcSR two-component signal transduction system directly regulates the lac and opuCABCD operons of Staphylococcus aureus.Significance of four methionine sulfoxide reductases in Staphylococcus aureus.Establishment of a bacterial infection model using the European honeybee, Apis mellifera L.Identification of the P3 promoter and distinct roles of the two promoters of the SaeRS two-component system in Staphylococcus aureus.Silkworm apolipophorin protein inhibits Staphylococcus aureus virulenceThe AirSR two-component system contributes to Staphylococcus aureus survival in human blood and transcriptionally regulates sspABC operonOrganizational requirements of the SaeR binding sites for a functional P1 promoter of the sae operon in Staphylococcus aureusRegulation of bacterial virulence gene expression by cell envelope stress responses.The WalKR system controls major staphylococcal virulence genes and is involved in triggering the host inflammatory responseThe auxiliary protein complex SaePQ activates the phosphatase activity of sensor kinase SaeS in the SaeRS two-component system of Staphylococcus aureus.Staphylococcus epidermidis saeR is an effector of anaerobic growth and a mediator of acute inflammation.The mutated staphylococcal H35A α-toxin inhibits adhesion and invasion of Staphylococcus aureus and group A streptococci.Perturbation of Staphylococcus aureus gene expression by the enoyl-acyl carrier protein reductase inhibitor AFN-1252.Insertional inactivation of branched-chain alpha-keto acid dehydrogenase in Staphylococcus aureus leads to decreased branched-chain membrane fatty acid content and increased susceptibility to certain stressesStaphylococcus aureus Coordinates Leukocidin Expression and Pathogenesis by Sensing Metabolic Fluxes via RpiRc.A secreted bacterial protease tailors the Staphylococcus aureus virulence repertoire to modulate bone remodeling during osteomyelitis.MgrA activates expression of capsule genes, but not the α-toxin gene in experimental Staphylococcus aureus endocarditis.The Staphylococcus aureus AirSR Two-Component System Mediates Reactive Oxygen Species Resistance via Transcriptional Regulation of Staphyloxanthin Production.Correspondence: Spontaneous secondary mutations confound analysis of the essential two-component system WalKR in Staphylococcus aureus.
P2860
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P2860
Inactivation of a two-component signal transduction system, SaeRS, eliminates adherence and attenuates virulence of Staphylococcus aureus.
description
2006 nî lūn-bûn
@nan
2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@ast
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@en
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@nl
type
label
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@ast
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@en
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@nl
prefLabel
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@ast
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@en
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@nl
P2093
P2860
P356
P1476
Inactivation of a two-componen ...... ence of Staphylococcus aureus.
@en
P2093
Christina Landwehr
Chuanxin Yu
David Holmes
Junsong Sun
Xudong Liang
P2860
P304
P356
10.1128/IAI.00322-06
P407
P577
2006-08-01T00:00:00Z