about
'Miracle stents'--a future without restenosisRisk factors in coronary atherosclerosis athero-inflammation: the meeting pointEverolimus-eluting stent platforms in percutaneous coronary intervention: comparative effectiveness and outcomesInflammation and vascular injury: basic discovery to drug developmentRecent advances in drug eluting stentsNanoparticulate carriers for the treatment of coronary restenosisEffects of an alpha-4 integrin inhibitor on restenosis in a new porcine model combining endothelial denudation and stent placementPlasma microRNAs as potential noninvasive biomarkers for in-stent restenosisPrognostic Impact of 9-Month High-Sensitivity C-Reactive Protein Levels on Long-Term Clinical Outcomes and In-Stent Restenosis in Patients at 9 Months after Drug-Eluting Stent ImplantationPDGF-induced proliferation of smooth muscular cells is related to the regulation of CREB phosphorylation and Nur77 expressionEstrogen modulates TNF-alpha-induced inflammatory responses in rat aortic smooth muscle cells through estrogen receptor-beta activationIntegrative genomics identifies DSCR1 (RCAN1) as a novel NFAT-dependent mediator of phenotypic modulation in vascular smooth muscle cells.Increased smooth muscle cell activation and neointima formation in response to injury in AIF-1 transgenic miceCD40 ligand promotes Mac-1 expression, leukocyte recruitment, and neointima formation after vascular injury.Computational simulation methodologies for mechanobiological modelling: a cell-centred approach to neointima development in stentsEvaluation of wrought Zn-Al alloys (1, 3, and 5 wt % Al) through mechanical and in vivo testing for stent applications.ACE (I/D) polymorphism and response to treatment in coronary artery disease: a comprehensive database and meta-analysis involving study quality evaluationMatrine inhibits vascular smooth muscle cell proliferation by modulating the expression of cell cycle regulatory genes.Stenting and angioplasty of small cerebral arteries in symptomatic intracranial atherosclerotic disease.Impact of C-reactive protein on in-stent restenosis: a meta-analysis.A novel mouse model of in-stent restenosisNeutrophils alter the inflammatory milieu by signal-dependent translation of constitutive messenger RNAs.Cathepsin L promotes Vascular Intimal Hyperplasia after Arterial InjuryIL-19 reduces ligation-mediated neointimal hyperplasia by reducing vascular smooth muscle cell activation.Frictional Behavior of Individual Vascular Smooth Muscle Cells Assessed By Lateral Force MicroscopyInflammation and in-stent restenosis: the role of serum markers and stent characteristics in carotid artery stenting.Inhibition of STAT3 signaling prevents vascular smooth muscle cell proliferation and neointima formation.Bisphosphonate-mediated gene vector delivery from the metal surfaces of stents.Estrogen modulates NFκB signaling by enhancing IκBα levels and blocking p65 binding at the promoters of inflammatory genes via estrogen receptor-β.Paclitaxel/sirolimus combination coated drug-eluting stent: in vitro and in vivo drug release studies.Cyclooxygenase-2-derived prostaglandin E₂ promotes injury-induced vascular neointimal hyperplasia through the E-prostanoid 3 receptor.Reduction of endoplasmic reticulum stress inhibits neointima formation after vascular injury.Association between serum sulfatide and carotid intima media thickness in patients with familial hypercholesterolemia.Plaque morphology detected with Duplex ultrasound before carotid angioplasty and stenting (CAS) is not a predictor of carotid artery in-stent restenosis, a case control study.Soluble CD40 ligand stimulates CD40-dependent activation of the β2 integrin Mac-1 and protein kinase C zeda (PKCζ) in neutrophils: implications for neutrophil-platelet interactions and neutrophil oxidative burstAnti-inflammatory cytokine interleukin-19 inhibits smooth muscle cell migration and activation of cytoskeletal regulators of VSMC motility.Genetic polymorphism of angiotensin converting enzyme and risk of coronary restenosis after percutaneous transluminal coronary angioplasties: evidence from 33 cohort studiesDetermining the prevalence and predictors of sleep disordered breathing in patients with chronic heart failure: rationale and design of the SCHLA-HF registry.An agent-based model of the response to angioplasty and bare-metal stent deployment in an atherosclerotic blood vessel.Smooth muscle specific overexpression of p22phox potentiates carotid artery wall thickening in response to injury.
P2860
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P2860
description
2002 nî lūn-bûn
@nan
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Inflammation and restenosis in the stent era.
@ast
Inflammation and restenosis in the stent era.
@en
Inflammation and restenosis in the stent era.
@nl
type
label
Inflammation and restenosis in the stent era.
@ast
Inflammation and restenosis in the stent era.
@en
Inflammation and restenosis in the stent era.
@nl
prefLabel
Inflammation and restenosis in the stent era.
@ast
Inflammation and restenosis in the stent era.
@en
Inflammation and restenosis in the stent era.
@nl
P1476
Inflammation and restenosis in the stent era.
@en
P2093
Campbell Rogers
Frederick G P Welt
P304
P356
10.1161/01.ATV.0000037100.44766.5B
P407
P577
2002-11-01T00:00:00Z