Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus. - Test2 - elhamkhani - TriplyDB

Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus.

Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus.

http://www.wikidata.org/entity/Q35008584

about

Fracture healing: mechanisms and interventions Biomimetic strategies for fracture repair: Engineering the cell microenvironment for directed tissue formation.IL-17-producing γδ T cells enhance bone regeneration.Effects of Aging on Fracture Healing.The Role of the Immune Cells in Fracture Healing.Identification of key gene networks associated with fracture healing using αSMA‑labeled progenitor cells.

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P2860

Role of Fas and Treg cells in fracture healing as characterized in the fas-deficient (lpr) mouse model of lupus.

http://www.wikidata.org/entity/Q35008584

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Role of Fas and Treg cells in ...... nt (lpr) mouse model of lupus.

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Role of Fas and Treg cells in ...... nt (lpr) mouse model of lupus.

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Role of Fas and Treg cells in ...... nt (lpr) mouse model of lupus.

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Role of Fas and Treg cells in ...... nt (lpr) mouse model of lupus.

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Journal of Bone and Mineral Research

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Role of Fas and Treg cells in ...... ent (lpr) mouse model of lupus

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Dana Graves

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Dana M Daukss

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Daniel Cusher

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Elise Morgan

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Maisa O Al-Sebaei

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Nathan A Wigner

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Sanjeev Kakar

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P2860

Estrogen protects bone by inducing Fas ligand in osteoblasts to regulate osteoclast survival

A proinflammatory role for Fas in joints of mice with collagen-induced arthritis

Bone histomorphometry: standardization of nomenclature, symbols, and units. Report of the ASBMR Histomorphometry Nomenclature Committee

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

Tumor necrosis factor alpha (TNF-alpha) coordinately regulates the expression of specific matrix metalloproteinases (MMPS) and angiogenic factors during fracture healing.

Micro-computed tomography assessment of fracture healing: relationships among callus structure, composition, and mechanical function.

Tumor necrosis factor receptor and Fas signaling mechanisms.

Phosphate-induced apoptosis of hypertrophic chondrocytes is associated with a decrease in mitochondrial membrane potential and is dependent upon Erk1/2 phosphorylation.

The role of interleukin-17 in bone metabolism and inflammatory skeletal diseases.

T-lymphocytes enable osteoblast maturation via IL-17F during the early phase of fracture repair

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1478-1491

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scholarly article

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10.1002/JBMR.2169

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6

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29

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Thomas A Einhorn

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2014-06-01T00:00:00Z

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24677136

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