Suppression by antisense mRNA demonstrates a requirement for the glial fibrillary acidic protein in the formation of stable astrocytic processes in response to neurons.
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The function of intermediate filaments in cell shape and cytoskeletal integrityTargeted deletion in astrocyte intermediate filament (Gfap) alters neuronal physiologySchwann cells of the myelin-forming phenotype express neurofilament protein NF-MComparative anatomy of the cerebellar cortex in mice lacking vimentin, GFAP, and both vimentin and GFAPIn situ hybridization analysis of glial fibrillary acidic protein mRNA reveals evidence of biphasic astrocyte activation during acute experimental autoimmune encephalomyelitis.Vimentin organization modulates the formation of lamellipodia.Integrative proteomic analysis of the nucleus accumbens in rhesus monkeys following cocaine self-administrationGFAP isoforms control intermediate filament network dynamics, cell morphology, and focal adhesions.Cytosolic proteomic alterations in the nucleus accumbens of cocaine overdose victimsGliotoxin-induced swelling of astrocytes hinders diffusion in brain extracellular space via formation of dead-space microdomainsVimentin induces changes in cell shape, motility, and adhesion during the epithelial to mesenchymal transition.Endocytosis is a critical step in entry of subgroup B avian leukosis viruses.Inroads into the structure and function of intermediate filament networksEffects of growth factors and basement membrane proteins on the phenotype of U-373 MG glioblastoma cells as determined by the expression of intermediate filament proteins.Fatal encephalopathy with astrocyte inclusions in GFAP transgenic miceInhibition of desmin expression blocks myoblast fusion and interferes with the myogenic regulators MyoD and myogenin.Reexpression of glial fibrillary acidic protein rescues the ability of astrocytoma cells to form processes in response to neuronsA function for keratins and a common thread among different types of epidermolysis bullosa simplex diseasesExpression of 300-kilodalton intermediate filament-associated protein distinguishes human glioma cells from normal astrocytes.Characterization of a Novel Chromatin Sorting Tool Reveals Importance of Histone Variant H3.3 in Contextual Fear Memory and Motor LearningCortical expression of glial fibrillary acidic protein and glutamine synthetase is decreased in schizophreniaAstrocytic plasticity and patterned oxytocin neuronal activity: dynamic interactions.Suppression of villin expression by antisense RNA impairs brush border assembly in polarized epithelial intestinal cellsMice lacking glial fibrillary acidic protein display astrocytes devoid of intermediate filaments but develop and reproduce normally.Role of GFAP in CNS injuries.E804 induces growth arrest, differentiation and apoptosis of glioblastoma cells by blocking Stat3 signaling.Overexpression of p21WAF1/CIP1 induces cell differentiation and growth inhibition in a human glioma cell line.Astrocytoma and Schwann cells in coculture.Vinculin-deficient PC12 cell lines extend unstable lamellipodia and filopodia and have a reduced rate of neurite outgrowth.Modulation of apoptosis by adenosine in the central nervous system: a possible role for the A3 receptor. Pathophysiological significance and therapeutic implications for neurodegenerative disorders.Abnormal reaction to central nervous system injury in mice lacking glial fibrillary acidic protein and vimentin.Engrafted peripheral blood-derived mesenchymal stem cells promote locomotive recovery in adult rats after spinal cord injury.Suppression of vinculin expression by antisense transfection confers changes in cell morphology, motility, and anchorage-dependent growth of 3T3 cells.Neurite outgrowth in peripherin-depleted PC12 cells.Molecular dissection of the Rho-associated protein kinase (p160ROCK)-regulated neurite remodeling in neuroblastoma N1E-115 cells.Administration of FGF-2 to embryonic mouse brain induces hydrocephalic brain morphology and aberrant differentiation of neurons in the postnatal cerebral cortex.Effect of elevated K(+), hypotonic stress, and cortical spreading depression on astrocyte swelling in GFAP-deficient mice.Forebrain astroglial plasticity is induced following withdrawal from repeated cocaine administration.Loss of GFAP expression in high-grade astrocytomas does not contribute to tumor development or progression.Dehydration and rehydration selectively and reversibly alter glial fibrillary acidic protein immunoreactivity in the rat supraoptic nucleus and subjacent glial limitans.
P2860
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P2860
Suppression by antisense mRNA demonstrates a requirement for the glial fibrillary acidic protein in the formation of stable astrocytic processes in response to neurons.
description
1991 nî lūn-bûn
@nan
1991 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի մարտին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
name
Suppression by antisense mRNA ...... cesses in response to neurons.
@ast
Suppression by antisense mRNA ...... cesses in response to neurons.
@en
type
label
Suppression by antisense mRNA ...... cesses in response to neurons.
@ast
Suppression by antisense mRNA ...... cesses in response to neurons.
@en
prefLabel
Suppression by antisense mRNA ...... cesses in response to neurons.
@ast
Suppression by antisense mRNA ...... cesses in response to neurons.
@en
P2093
P2860
P356
P1476
Suppression by antisense mRNA ...... cesses in response to neurons.
@en
P2093
D E Weinstein
M L Shelanski
P2860
P304
P356
10.1083/JCB.112.6.1205
P407
P577
1991-03-01T00:00:00Z