I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
about
Cortical HCN channels: function, trafficking and plasticityDifferential contribution of Ih to the integration of excitatory synaptic inputs in substantia nigra pars compacta and ventral tegmental area dopaminergic neurons.Differential triggering of spontaneous glutamate release by P/Q-, N- and R-type Ca2+ channels.A calcium-dependent plasticity rule for HCN channels maintains activity homeostasis and stable synaptic learningDisrupted Cl(-) homeostasis contributes to reductions in the inhibitory efficacy of diazepam during hyperexcited statesAstrocytic GABA transporter activity modulates excitatory neurotransmission.Endogenous zinc depresses GABAergic transmission via T-type Ca(2+) channels and broadens the time window for integration of glutamatergic inputs in dentate granule cells.Towards an integrated view of HCN channel role in epilepsy.Cortical inhibition, pH and cell excitability in epilepsy: what are optimal targets for antiepileptic interventions?Regulation of neuronal chloride homeostasis by neuromodulators.Differentiation and functional incorporation of embryonic stem cell-derived GABAergic interneurons in the dentate gyrus of mice with temporal lobe epilepsy.The Hyperpolarization-Activated Current Determines Synaptic Excitability, Calcium Activity and Specific Viability of Substantia Nigra Dopaminergic Neurons.Tonic GABAA conductance decreases membrane time constant and increases EPSP-spike precision in hippocampal pyramidal neurons.High-conductance states and A-type K+ channels are potential regulators of the conductance-current balance triggered by HCN channels.Degeneracy in the regulation of short-term plasticity and synaptic filtering by presynaptic mechanisms.Layer- and subregion-specific differences in the neurophysiological properties of rat medial prefrontal cortex pyramidal neurons.Synaptic mechanisms of adenosine A2A receptor-mediated hyperexcitability in the hippocampus.Influence fields: a quantitative framework for representation and analysis of active dendrites.Dynamical Mechanism of Hyperpolarization-Activated Non-specific Cation Current Induced Resonance and Spike-Timing Precision in a Neuronal Model.
P2860
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P2860
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
name
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@ast
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@en
type
label
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@ast
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@en
prefLabel
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@ast
I(h)-mediated depolarization enhances the temporal precision of neuronal integration.
@en
P2860
P50
P356
P1476
I(h)-mediated depolarization enhances the temporal precision of neuronal integration
@en
P2093
Leonid Savtchenko
P2860
P2888
P356
10.1038/NCOMMS1202
P407
P577
2011-02-15T00:00:00Z