H2O2 is the transferrable factor mediating flow-induced dilation in human coronary arterioles
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Bang-bang model for regulation of local blood flowDiversity in mechanisms of endothelium-dependent vasodilation in health and diseaseMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesHydrogen peroxide elicits constriction of skeletal muscle arterioles by activating the arachidonic acid pathwayCeramide changes the mediator of flow-induced vasodilation from nitric oxide to hydrogen peroxide in the human microcirculation.Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cellsRenal autoregulation in health and diseaseH2O2-induced dilation in human coronary arterioles: role of protein kinase G dimerization and large-conductance Ca2+-activated K+ channel activation.Vascular TRP channels: performing under pressure and going with the flowα1- and α2-adrenergic responsiveness in human skeletal muscle feed arteries: the role of TRPV ion channels in heat-induced sympatholysisAn acute rise in intraluminal pressure shifts the mediator of flow-mediated dilation from nitric oxide to hydrogen peroxide in human arterioles.The endothelium: influencing vascular smooth muscle in many ways.Oxidative stress in atrial fibrillation: an emerging role of NADPH oxidase.Recent developments in vascular biologyAcute exertion elicits a H2O2-dependent vasodilator mechanism in the microvasculature of exercise-trained but not sedentary adults.Endothelium-derived hyperpolarizing factor and vascular function.Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse heartsHydrogen sulfide as endothelium-derived hyperpolarizing factor sulfhydrates potassium channels.5-hydroxytryptamine has an endothelium-derived hyperpolarizing factor-like effect on coronary flow in isolated rat hearts.Regulation of the human coronary microcirculation.Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Activation of endothelial TRPV4 channels mediates flow-induced dilation in human coronary arterioles: role of Ca2+ entry and mitochondrial ROS signaling.CYP2C29 produces superoxide in response to shear stress.Endothelium-Derived Hyperpolarization and Coronary Vasodilation: Diverse and Integrated Roles of Epoxyeicosatrienoic Acids, Hydrogen Peroxide, and Gap Junctions.Exercise training-enhanced, endothelium-dependent dilation mediated by altered regulation of BK(Ca) channels in collateral-dependent porcine coronary arteriolesCritical Role for Telomerase in the Mechanism of Flow-Mediated Dilation in the Human Microcirculation.Reduced flow-and acetylcholine-induced dilations in visceral compared to subcutaneous adipose arterioles in human morbid obesity.Impact of age on the vasodilatory function of human skeletal muscle feed arteriesDifferential role of endothelial versus neuronal nitric oxide synthase in the regulation of coronary blood flow during pacing-induced increases in cardiac workload.Diverse Functions of Endothelial NO Synthases System: NO and EDH.Modulation of the myogenic mechanism: concordant effects of NO synthesis inhibition and O2- dismutation on renal autoregulation in the time and frequency domains.Arachidonic acid-induced dilation in human coronary arterioles: convergence of signaling mechanisms on endothelial TRPV4-mediated Ca2+ entryDifferential effects of superoxide and hydrogen peroxide on myogenic signaling, membrane potential, and contractions of mouse renal afferent arterioles.Reactive oxygen species facilitate the EDH response in arterioles by potentiating intracellular endothelial Ca(2+) releaseContribution of nitric oxide to brachial artery vasodilation during progressive handgrip exercise in the elderly.Exercise training enhances multiple mechanisms of relaxation in coronary arteries from ischemic hearts.Transcriptional and phenotypic changes in aorta and aortic valve with aging and MnSOD deficiency in mice.Oxidative stress in hypertension: role of the kidney.Characterization of blood pressure and endothelial function in TRPV4-deficient mice with l-NAME- and angiotensin II-induced hypertension.Differential roles of NADPH oxidases in vascular physiology and pathophysiology
P2860
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P2860
H2O2 is the transferrable factor mediating flow-induced dilation in human coronary arterioles
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
H2O2 is the transferrable fact ...... n in human coronary arterioles
@ast
H2O2 is the transferrable fact ...... n in human coronary arterioles
@en
type
label
H2O2 is the transferrable fact ...... n in human coronary arterioles
@ast
H2O2 is the transferrable fact ...... n in human coronary arterioles
@en
prefLabel
H2O2 is the transferrable fact ...... n in human coronary arterioles
@ast
H2O2 is the transferrable fact ...... n in human coronary arterioles
@en
P2093
P2860
P1433
P1476
H2O2 is the transferrable fact ...... n in human coronary arterioles
@en
P2093
Aaron H Bubolz
David D Gutterman
David X Zhang
Suelhem Mendoza
Yanping Liu
P2860
P304
P356
10.1161/CIRCRESAHA.110.237636
P577
2011-01-13T00:00:00Z