Progesterone inhibition of voltage-gated calcium channels is a potential neuroprotective mechanism against excitotoxicity - Test2 - elhamkhani - TriplyDB

Progesterone inhibition of voltage-gated calcium channels is a potential neuroprotective mechanism against excitotoxicity

Progesterone inhibition of voltage-gated calcium channels is a potential neuroprotective mechanism against excitotoxicity

http://www.wikidata.org/entity/Q35085671

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Embracing failure: What the Phase III progesterone studies can teach about TBI clinical trials Progesterone inhibition of neuronal calcium signaling underlies aspects of progesterone-mediated neuroprotection Novel frontiers in epilepsy treatments: preventing epileptogenesis by targeting inflammation Neuroprotection by Estrogen and Progesterone in Traumatic Brain Injury and Spinal Cord Injury.Water-soluble progesterone analogues are effective, injectable treatments in animal models of traumatic brain injury.The neuroprotective steroid progesterone promotes mitochondrial uncoupling, reduces cytosolic calcium and augments stress resistance in yeast cells Progesterone alleviates neural behavioral deficits and demyelination with reduced degeneration of oligodendroglial cells in cuprizone-induced mice Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures.Imaging the Insertion of Superecliptic pHluorin-Labeled Dopamine D2 Receptor Using Total Internal Reflection Fluorescence Microscopy.Progesterone blocks multiple routes of ion flux Corticotropin-releasing factor and urocortin I activate CREB through functionally selective Gβγ signaling in hippocampal pyramidal neurons.Progesterone increases the release of brain-derived neurotrophic factor from glia via progesterone receptor membrane component 1 (Pgrmc1)-dependent ERK5 signaling.Progesterone and vitamin d hormone as a biologic treatment of traumatic brain injury in the aged.The neuroprotective effects of progesterone on traumatic brain injury: current status and future prospects.Pharmacotherapy of traumatic brain injury: state of the science and the road forward: report of the Department of Defense Neurotrauma Pharmacology Workgroup.Progesterone Provides the Pleiotropic Neuroprotective Effect on Traumatic Brain Injury Through the Nrf2/ARE Signaling Pathway.Progesterone Sharpens Temporal Response Profiles of Sensory Cortical Neurons in Animals Exposed to Traumatic Brain Injury.Beta-Estradiol Regulates Voltage-Gated Calcium Channels and Estrogen Receptors in Telocytes from Human Myometrium.

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Progesterone inhibition of voltage-gated calcium channels is a potential neuroprotective mechanism against excitotoxicity

http://www.wikidata.org/entity/Q35085671

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Progesterone inhibition of vol ...... chanism against excitotoxicity

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Progesterone inhibition of vol ...... chanism against excitotoxicity

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Progesterone inhibition of vol ...... chanism against excitotoxicity

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J.STEROIDS.2011.02.013

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Progesterone inhibition of vol ...... chanism against excitotoxicity

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Brooke G Kelley

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Jessie I Luoma

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Paul G Mermelstein

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P2860

Improved outcomes from the administration of progesterone for patients with acute severe traumatic brain injury: a randomized controlled trial

Role of extracellular signal-regulated protein kinases in apoptosis by asbestos and H2O2

Progesterone receptor A (PRA) and PRB-independent effects of progesterone on gonadotropin-releasing hormone release

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Transcription factors of the NFAT family: regulation and function

Extrasynaptic NMDA receptors couple preferentially to excitotoxicity via calpain-mediated cleavage of STEP

Calcineurin/NFAT-induced up-regulation of the Fas ligand/Fas death pathway is involved in methamphetamine-induced neuronal apoptosis

Deafferentation-induced activation of NFAT (nuclear factor of activated T-cells) in cochlear nucleus neurons during a developmental critical period: a role for NFATc4-dependent apoptosis in the CNS

Allopregnanolone, a progesterone metabolite, is more effective than progesterone in reducing cortical infarct volume after transient middle cerebral artery occlusion.

Acute administration of non-classical estrogen receptor agonists attenuates ischemia-induced hippocampal neuron loss in middle-aged female rats.

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845-855

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scholarly article

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10.1016/J.STEROIDS.2011.02.013

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9

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76

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2011-03-01T00:00:00Z

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50270224

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21371490

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3129396

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