Reductions in claudin-1 may enhance susceptibility to herpes simplex virus 1 infections in atopic dermatitis - Test2 - elhamkhani - TriplyDB

Reductions in claudin-1 may enhance susceptibility to herpes simplex virus 1 infections in atopic dermatitis

Reductions in claudin-1 may enhance susceptibility to herpes simplex virus 1 infections in atopic dermatitis

http://www.wikidata.org/entity/Q35085856

about

Molecular Mechanisms of Cutaneous Inflammatory Disorder: Atopic Dermatitis The Pathogenetic Effect of Natural and Bacterial Toxins on Atopic Dermatitis Genetic and epigenetic studies of atopic dermatitis Skin barrier defects in atopic dermatitis Advances in atopic dermatitis Epithelial barrier function: at the front line of asthma immunology and allergic airway inflammation Atopic dermatitis: a disease of altered skin barrier and immune dysregulation.Comparable mRNA expression of inflammatory markers but lower claudin-1 mRNA levels in foreskin tissue of HSV-2 seropositive versus seronegative asymptomatic Kenyan young men.Progesterone-based intrauterine device use is associated with a thinner apical layer of the human ectocervical epithelium and a lower ZO-1 mRNA expression.Mannan-binding lectin levels and activity are not altered in atopic dermatitis patients with a history of eczema herpeticum Cutaneous microbiome effects of fluticasone propionate cream and adjunctive bleach baths in childhood atopic dermatitis.Staphylococcus aureus α-toxin modulates skin host response to viral infection.Comorbidity in Atopic Dermatitis.Why is eczema herpeticum unexpectedly rare?Epidermal tight junctions in health and disease.The Genetics and Epigenetics of Atopic Dermatitis-Filaggrin and Other Polymorphisms.Ectodysplasin A regulates epithelial barrier function through sonic hedgehog signalling pathway.Skin barrier in atopic dermatitis: beyond filaggrin.Chronic systemic inflammation originating from epithelial tissues.Tight junctions in skin inflammation.Differential effects of topical corticosteroid and calcineurin inhibitor on the epidermal tight junction.Atopic dermatitis complicated by eczema herpeticum is associated with HLA B7 and reduced interferon-γ-producing CD8+ T cells.What is the role of Staphylococcus aureus and herpes virus infections in the pathogenesis of atopic dermatitis?IL-4 impairs wound healing potential in the skin by repressing fibronectin expression.Allergen Delivery Inhibitors: A Rationale for Targeting Sentinel Innate Immune Signaling of Group 1 House Dust Mite Allergens through Structure-Based Protease Inhibitor Design

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Reductions in claudin-1 may enhance susceptibility to herpes simplex virus 1 infections in atopic dermatitis

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2011 nî lūn-bûn

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2011年の論文

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J.JACI.2011.02.014

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The Journal of Allergy and Clinical Immunology

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Reductions in claudin-1 may en ...... nfections in atopic dermatitis

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Anna De Benedetto

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David C Johnson

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Donald Y M Leung

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I-Hsin Kuo

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Jon M Hanifin

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Mark K Slifka

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P2860

Entry of alphaherpesviruses mediated by poliovirus receptor-related protein 1 and poliovirus receptor

Claudin-based tight junctions are crucial for the mammalian epidermal barrier: a lesson from claudin-1-deficient mice

Recent insights into atopic dermatitis and implications for management of infectious complications.

Tight junction defects in patients with atopic dermatitis.

Phenotype of atopic dermatitis subjects with a history of eczema herpeticum.

Tight junctions/adherens junctions: basic structure and function.

Filaggrin mutations that confer risk of atopic dermatitis confer greater risk for eczema herpeticum.

Virus interaction with the apical junctional complex.

Filaggrin in atopic dermatitis.

Herpes simplex virus type 1 exhibits a tropism for basal entry in polarized epithelial cells.

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