Fragmentation of the Golgi apparatus induced by the overexpression of wild-type and mutant human tau forms in neurons.
about
Golgi fragmentation in Alzheimer's diseaseTangles, Toxicity, and Tau Secretion in AD - New Approaches to a Vexing ProblemEssential role of cyclin-G-associated kinase (Auxilin-2) in developing and mature miceCo-expression of truncated and full-length tau induces severe neurotoxicityTau interacts with Golgi membranes and mediates their association with microtubules.Mechanisms of tau-induced neurodegeneration.NMDA receptor mediates tau-induced neurotoxicity by calpain and ERK/MAPK activation.Tau in Alzheimer disease and related tauopathies.Functional genomic screen and network analysis reveal novel modifiers of tauopathy dissociated from tau phosphorylation.Studies of optineurin, a glaucoma gene: Golgi fragmentation and cell death from overexpression of wild-type and mutant optineurin in two ocular cell types.Golgi fragmentation in amyotrophic lateral sclerosis, an overview of possible triggers and consequences.Alteration of Golgi Structure by Stress: A Link to Neurodegeneration?Tau secretion is correlated to an increase of Golgi dynamics.The study of Golgi apparatus in Alzheimer's disease.A rapid gene delivery-based mouse model for early-stage Alzheimer disease-type tauopathyMisfolded tau protein and disease modifying pathways in transgenic rodent models of human tauopathies.Cell-cycle markers in a transgenic mouse model of human tauopathy: increased levels of cyclin-dependent kinase inhibitors p21Cip1 and p27Kip1.Morphometric alterations of Golgi apparatus in Alzheimer's disease are related to tau hyperphosphorylation.Changes in the Golgi Apparatus of Neocortical and Hippocampal Neurons in the Hibernating Hamster.Phospho-Tau Accumulation and Structural Alterations of the Golgi Apparatus of Cortical Pyramidal Neurons in the P301S Tauopathy Mouse Model.Hyperphosphorylation-induced tau oligomersIncreased association between rough endoplasmic reticulum membranes and mitochondria in transgenic mice that express P301L tau.SOD1 Mutations Causing Familial Amyotrophic Lateral Sclerosis Induce Toxicity in Astrocytes: Evidence for Bystander Effects in a Continuum of Astrogliosis.
P2860
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P2860
Fragmentation of the Golgi apparatus induced by the overexpression of wild-type and mutant human tau forms in neurons.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@ast
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@en
type
label
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@ast
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@en
prefLabel
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@ast
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@en
P2093
P2860
P1476
Fragmentation of the Golgi app ...... nt human tau forms in neurons.
@en
P2093
Dalinda Liazoghli
Mylène Desjardins
Nicole Leclerc
Sebastien Perreault
P2860
P304
P356
10.1016/S0002-9440(10)62366-8
P407
P577
2005-05-01T00:00:00Z