Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44. - Test2 - elhamkhani - TriplyDB

Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44.

Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44.

http://www.wikidata.org/entity/Q35102315

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Roles of Proteoglycans and Glycosaminoglycans in Wound Healing and Fibrosis Size Matters: Molecular Weight Specificity of Hyaluronan Effects in Cell Biology Always cleave up your mess: targeting collagen degradation to treat tissue fibrosis Matrix biology of idiopathic pulmonary fibrosis: a workshop report of the national heart, lung, and blood institute Autotaxin-LPA receptor axis in the pathogenesis of lung diseases Cellular mechanisms of tissue fibrosis. 7. New insights into the cellular mechanisms of pulmonary fibrosis The wound healing, chronic fibrosis, and cancer progression triad Interactions between Hyaluronan and Its Receptors (CD44, RHAMM) Regulate the Activities of Inflammation and Cancer Mechanisms of alveolar epithelial injury, repair, and fibrosis Pulmonary fibrosis: patterns and perpetrators Validated prediction of pro-invasive growth factors using a transcriptome-wide invasion signature derived from a complex 3D invasion assay Targeting the isoprenoid pathway to abrogate progression of pulmonary fibrosis Mechanisms of fibrosis: therapeutic translation for fibrotic disease FAK-related nonkinase is a multifunctional negative regulator of pulmonary fibrosis.Hsp90 regulation of fibroblast activation in pulmonary fibrosis.Interaction of Src and Alpha-V Integrin Regulates Fibroblast Migration and Modulates Lung Fibrosis in A Preclinical Model of Lung Fibrosis Urokinase-type plasminogen activator receptor (uPAR) ligation induces a raft-localized integrin signaling switch that mediates the hypermotile phenotype of fibrotic fibroblasts The effect of NF-κB antisense oligonucleotide on transdifferentiation of fibroblast in lung tissue of mice injured by bleomycin.Adenosine A2B receptor and hyaluronan modulate pulmonary hypertension associated with chronic obstructive pulmonary disease Bone marrow-derived stromal cells are invasive and hyperproliferative and alter transforming growth factor-α-induced pulmonary fibrosis Pathogenesis of idiopathic pulmonary fibrosis High and low molecular weight hyaluronic acid differentially regulate human fibrocyte differentiation.Prostaglandin E2 reduces Toll-like receptor 4 expression in alveolar macrophages by inhibition of translation.Therapeutic targeting of SRC kinase in myofibroblast differentiation and pulmonary fibrosis.Hydrogel scaffolds as in vitro models to study fibroblast activation in wound healing and disease.Lymphatics in lymphangioleiomyomatosis and idiopathic pulmonary fibrosis CD44 plays a critical role in regulating diet-induced adipose inflammation, hepatic steatosis, and insulin resistance Mechanosignaling through YAP and TAZ drives fibroblast activation and fibrosis Integrated genomic analyses in bronchopulmonary dysplasia.MicroRNA-328 inhibits renal tubular cell epithelial-to-mesenchymal transition by targeting the CD44 in pressure-induced renal fibrosis.Hyaluronan in intestinal homeostasis and inflammation: implications for fibrosis Gene expression profiling of extrahepatic ducts in children with biliary atresia.Methylation-mediated BMPER expression in fibroblast activation in vitro and lung fibrosis in mice in vivo.Down-regulation of USP13 mediates phenotype transformation of fibroblasts in idiopathic pulmonary fibrosis Osteopontin is an endogenous modulator of the constitutively activated phenotype of pulmonary adventitial fibroblasts in hypoxic pulmonary hypertension.Group B Streptococcus Evades Host Immunity by Degrading Hyaluronan.Association between MUC5B polymorphism and susceptibility and severity of idiopathic pulmonary fibrosis.Focal Adhesion Kinase Regulates Fibroblast Migration via Integrin beta-1 and Plays a Central Role in Fibrosis.Hyaluronan as a therapeutic target in human diseases Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension

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Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44.

http://www.wikidata.org/entity/Q35102315

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2011年の論文

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Severe lung fibrosis requires ...... gulated by hyaluronan and CD44

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Alice Gray

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Eric B Meltzer

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Lise Wogensen

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Paul W Noble

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Riu Miura

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Yuejuan Li

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P2860

Cell surface-localized matrix metalloproteinase-9 proteolytically activates TGF-beta and promotes tumor invasion and angiogenesis

ADAMTS-1: a metalloproteinase-disintegrin essential for normal growth, fertility, and organ morphology and function

Disruption of hyaluronan synthase-2 abrogates normal cardiac morphogenesis and hyaluronan-mediated transformation of epithelium to mesenchyme

The myofibroblast: one function, multiple origins

Idiopathic pulmonary fibrosis: an epithelial/fibroblastic cross-talk disorder

Genetic ablation of Bcl-x attenuates invasiveness without affecting apoptosis or tumor growth in a mouse model of pancreatic neuroendocrine cancer

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis

Prognostic significance of histopathologic subsets in idiopathic pulmonary fibrosis

Resolution of lung inflammation by CD44

Three isoforms of mammalian hyaluronan synthases have distinct enzymatic properties

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10.1084/JEM.20102510

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7

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pulmonary fibrosis

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