Choice of antiretroviral therapy differentially impacts survival of HIV-infected CD4 T cells - Test2 - elhamkhani - TriplyDB

Choice of antiretroviral therapy differentially impacts survival of HIV-infected CD4 T cells

Choice of antiretroviral therapy differentially impacts survival of HIV-infected CD4 T cells

http://www.wikidata.org/entity/Q35111879

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Tuning of AKT-pathway by Nef and its blockade by protease inhibitors results in limited recovery in latently HIV infected T-cell line.Enhanced MAPK signaling drives ETS1-mediated induction of miR-29b leading to downregulation of TET1 and changes in epigenetic modifications in a subset of lung SCC HIV-1 Frameshift RNA-Targeted Triazoles Inhibit Propagation of Replication-Competent and Multi-Drug-Resistant HIV in Human Cells.Maintenance of the HIV Reservoir Is Antagonized by Selective BCL2 Inhibition.Limited HIV-1 Reactivation in Resting CD4+ T cells from Aviremic Patients under Protease Inhibitors.3' UTR G-quadruplexes regulate miRNA binding.Counteracting Akt Activation by HIV Protease Inhibitors in Monocytes/Macrophages.

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Choice of antiretroviral therapy differentially impacts survival of HIV-infected CD4 T cells

http://www.wikidata.org/entity/Q35111879

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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type

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

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Molecular and cellular therapies

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Choice of antiretroviral thera ...... al of HIV-infected CD4 T cells

@en

P2093

Amy M Sainski

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Andrew D Badley

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Gary D Bren

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Nathan W Cummins

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Sekar Natesampillai

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P2860

HIV-1 protease processes procaspase 8 to cause mitochondrial release of cytochrome c, caspase cleavage and nuclear fragmentation

In vitro and ex vivo inhibition of human telomerase by anti-HIV nucleoside reverse transcriptase inhibitors (NRTIs) but not by non-NRTIs

Intensification of antiretroviral therapy with a CCR5 antagonist in patients with chronic HIV-1 infection: effect on T cells latently infected

Prospects for treatment of latent HIV.

Homeostatic proliferation fails to efficiently reactivate HIV-1 latently infected central memory CD4+ T cells.

Infected cell killing by HIV-1 protease promotes NF-kappaB dependent HIV-1 replication.

Patients with discordant responses to antiretroviral therapy have impaired killing of HIV-infected T cells.

Elite suppressors harbor low levels of integrated HIV DNA and high levels of 2-LTR circular HIV DNA compared to HIV+ patients on and off HAART.

Inhibition of adenine nucleotide translocator pore function and protection against apoptosis in vivo by an HIV protease inhibitor

The immunopathogenesis of human immunodeficiency virus infection.

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1

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scholarly article

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10.1186/2052-8426-2-1

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2

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2014-01-03T00:00:00Z

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270459733

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1015221623

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26057236

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antiretroviral drug

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4337831

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