Lipid-induced NOX2 activation inhibits autophagic flux by impairing lysosomal enzyme activity
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Biliverdin reductase isozymes in metabolismInsulin Signaling and Heart Failure.Lysosome acidification by photoactivated nanoparticles restores autophagy under lipotoxicity.Cluster Differentiating 36 (CD36) Deficiency Attenuates Obesity-Associated Oxidative Stress in the Heart.Loss of long-chain acyl-CoA synthetase isoform 1 impairs cardiac autophagy and mitochondrial structure through mechanistic target of rapamycin complex 1 activation.Sexual dimorphism in activation of placental autophagy in obese women with evidence for fetal programming from a placenta-specific mouse model.Discordant signaling and autophagy response to fasting in hearts of obese mice: Implications for ischemia tolerance.Inhibition of autophagosome-lysosome fusion by ginsenoside Ro via the ESR2-NCF1-ROS pathway sensitizes esophageal cancer cells to 5-fluorouracil-induced cell death via the CHEK1-mediated DNA damage checkpoint.Adipocyte Fatty Acid Binding Protein Potentiates Toxic Lipids-Induced Endoplasmic Reticulum Stress in Macrophages via Inhibition of Janus Kinase 2-dependent Autophagy.Active Site Hydrophobicity and the Convergent Evolution of Paraoxonase Activity in Structurally Divergent Enzymes: The Case of Serum Paraoxonase 1.Boosting autophagy in the diabetic heart: a translational perspective.Autophagy in cardiac metabolic control: Novel mechanisms for cardiovascular disorders.Lipids, lysosomes, and autophagy.Autophagy as a regulator of cardiovascular redox homeostasis.Myocardial stress and autophagy: mechanisms and potential therapies.Oxidative Stress and Cardiovascular Risk: Obesity, Diabetes, Smoking, and Pollution: Part 3 of a 3-Part Series.A new pathway regulating autophagy.Inhibition of NADPH oxidase 2 (NOX2) prevents sepsis-induced cardiomyopathy by improving calcium handling and mitochondrial function.Electron Paramagnetic Resonance Measurements of Reactive Oxygen Species by Cyclic Hydroxylamine Spin Probes.Endothelial Dysfunction in Obesity.What Is Lipotoxicity?Lipotoxic very-long-chain ceramides cause mitochondrial dysfunction, oxidative stress, and cell death in cardiomyocytes.Manifestations and mechanisms of myocardial lipotoxicity in obesity.Activation of IGF-1 receptors and Akt signaling by systemic hyperinsulinemia contributes to cardiac hypertrophy but does not regulate cardiac autophagy in obese diabetic mice.Increased expression of sterol regulatory element binding protein‑2 alleviates autophagic dysfunction in NAFLD.
P2860
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P2860
Lipid-induced NOX2 activation inhibits autophagic flux by impairing lysosomal enzyme activity
description
2014 nî lūn-bûn
@nan
2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@ast
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@en
type
label
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@ast
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@en
prefLabel
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@ast
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@en
P2093
P2860
P356
P1476
Lipid-induced NOX2 activation ...... ring lysosomal enzyme activity
@en
P2093
Christian Riehle
E Dale Abel
Heaseung S Chung
Jamie Soto
Jennifer E Van Eyk
L Ashley Cowart
Patrick Abel
Stephen Jenkins
P2860
P304
P356
10.1194/JLR.M055152
P577
2014-12-21T00:00:00Z