NADPH oxidases in cardiovascular disease: insights from in vivo models and clinical studies.
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NOX signaling in molecular cardiovascular mechanisms involved in the blood pressure homeostasisNeuroprotection after stroke by targeting NOX4 as a source of oxidative stressBiochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular systemNADPH oxidases in heart failure: poachers or gamekeepers?Heart failure and mitochondrial dysfunction: the role of mitochondrial fission/fusion abnormalities and new therapeutic strategiesHypertension and chronic ethanol consumption: What do we know after a century of study?Antioxidant-based therapies for angiotensin II-associated cardiovascular diseasesAction mechanism and cardiovascular effect of anthocyanins: a systematic review of animal and human studiesMitochondrial KATP channel involvement in angiotensin II-induced autophagy in vascular smooth muscle cells.Crossroads between peripheral atherosclerosis, western-type diet and skeletal muscle pathophysiology: emphasis on apolipoprotein E deficiency and peripheral arterial disease.P21-activated kinase in inflammatory and cardiovascular disease.Mechanisms of Myofascial PainDetraining differentially preserved beneficial effects of exercise on hypertension: effects on blood pressure, cardiac function, brain inflammatory cytokines and oxidative stress.Activation of volume-sensitive outwardly rectifying chloride channel by ROS contributes to ER stress and cardiac contractile dysfunction: involvement of CHOP through Wnt.Oxidative DNA damage in kidneys and heart of hypertensive mice is prevented by blocking angiotensin II and aldosterone receptors.Mitochondrial regulation of NADPH oxidase in hindlimb unweighting rat cerebral arteries.Endothelial dysfunction in the apolipoprotein E-deficient mouse: insights into the influence of diet, gender and aging.Chronic exercise modulates RAS components and improves balance between pro- and anti-inflammatory cytokines in the brain of SHRRegulation of the human coronary microcirculation.Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.Proteostasis and REDOX state in the heart.Regulation of NAD(P)H oxidases by AMPK in cardiovascular systemsEndothelin-1 impairs coronary arteriolar dilation: Role of p38 kinase-mediated superoxide production from NADPH oxidase.Activation of Stat3 in endothelial cells following hypoxia-reoxygenation is mediated by Rac1 and protein Kinase C.Serum levels of nicotinamide-adenine dinucleotide phosphate oxidase 4 are associated with non-valvular atrial fibrillation.Redox-sensitive oxidation and phosphorylation of PTEN contribute to enhanced activation of PI3K/Akt signaling in rostral ventrolateral medulla and neurogenic hypertension in spontaneously hypertensive ratsEvidence of the Importance of Nox4 in Production of Hypertension in Dahl Salt-Sensitive Rats.Oxidative stress, redox signaling, and metal chelation in anthracycline cardiotoxicity and pharmacological cardioprotection.Endothelial NADPH oxidase 4 protects ApoE-/- mice from atherosclerotic lesionsEndothelial NOS (NOS3) impairs myocardial function in developing sepsis.Type II diabetes increases mitochondrial DNA mutations in the left ventricle of the Goto-Kakizaki diabetic rat.The CYBA Gene (⁎)49A>G Polymorphism (rs7195830) Is Associated with Hypertension in Patients with Coronary Artery Disease.Genetic Variants of Cytochrome b-245, Alpha Polypeptide Gene and Premature Acute Myocardial Infarction Risk in an Iranian Population.Angiotensin receptor-mediated oxidative stress is associated with impaired cardiac redox signaling and mitochondrial function in insulin-resistant rats.Demethoxycurcumin Preserves Renovascular Function by Downregulating COX-2 Expression in Hypertension.PKC/NADPH oxidase are involved in the protective effect of pioglitazone in high homocysteine-induced paracrine dyfunction in endothelial progenitor cells.Opportunity nox: the future of NADPH oxidases as therapeutic targets in cardiovascular disease.Oxidative stress, Nox isoforms and complications of diabetes--potential targets for novel therapies.Redox regulation of sodium and calcium handlingNADPH oxidases as a source of oxidative stress and molecular target in ischemia/reperfusion injury.
P2860
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P2860
NADPH oxidases in cardiovascular disease: insights from in vivo models and clinical studies.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@ast
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@en
type
label
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@ast
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@en
prefLabel
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@ast
NADPH oxidases in cardiovascul ...... o models and clinical studies.
@en
P2860
P921
P1476
NADPH oxidases in cardiovascul ...... vo models and clinical studies
@en
P2093
Alexander Sirker
P2860
P2888
P304
P356
10.1007/S00395-011-0190-Z
P50
P577
2011-05-20T00:00:00Z