G protein G(alpha)o is essential for vomeronasal function and aggressive behavior in mice. - Test2 - elhamkhani - TriplyDB

G protein G(alpha)o is essential for vomeronasal function and aggressive behavior in mice.

G protein G(alpha)o is essential for vomeronasal function and aggressive behavior in mice.

http://www.wikidata.org/entity/Q35149601

about

Formyl peptide receptors from immune and vomeronasal system exhibit distinct agonist properties Of pheromones and kairomones: what receptors mediate innate emotional responses?Recognition of bacterial signal peptides by mammalian formyl peptide receptors: a new mechanism for sensing pathogens.Innate Predator Odor Aversion Driven by Parallel Olfactory Subsystems that Converge in the Ventromedial Hypothalamus A family of nonclassical class I MHC genes contributes to ultrasensitive chemodetection by mouse vomeronasal sensory neurons The structure, stability and pheromone binding of the male mouse protein sex pheromone darcin Functional Overexpression of Vomeronasal Receptors Using a Herpes Simplex Virus Type 1 (HSV-1)-Derived Amplicon A juvenile mouse pheromone inhibits sexual behaviour through the vomeronasal system Aggressive behaviour and physiological responses to pheromones are strongly impaired in mice deficient for the olfactory G-protein -subunit G8 Kirrel3 is required for the coalescence of vomeronasal sensory neuron axons into glomeruli and for male-male aggression Mammalian pheromones: emerging properties and mechanisms of detection Deterioration of the Gαo vomeronasal pathway in sexually dimorphic mammals Olfactomedin 1 Deficiency Leads to Defective Olfaction and Impaired Female Fertility Prioritizing the development of mouse models for childhood brain disorders Paradoxical contribution of SK3 and GIRK channels to the activation of mouse vomeronasal organ.Absence of M-Ras modulates social behavior in mice.Glutamate and Opioid Antagonists Modulate Dopamine Levels Evoked by Innately Attractive Male Chemosignals in the Nucleus Accumbens of Female Rats Deconstructing pheromone-mediated behavior one layer at a time.A wide range of pheromone-stimulated sexual and reproductive behaviors in female mice depend on G protein Gαo.Murine pheromone proteins constitute a context-dependent combinatorial code governing multiple social behaviors.Neurogenetics of aggressive behavior: studies in rodents.Evolution of immune chemoreceptors into sensors of the outside world.Physiological characterization of formyl peptide receptor expressing cells in the mouse vomeronasal organ.Signaling mechanisms and behavioral function of the mouse basal vomeronasal neuroepithelium.The molecular evolutionary dynamics of the vomeronasal receptor (class 1) genes in primates: a gene family on the verge of a functional breakdown.Mammalian pheromones.Conditional knockout of TMEM16A/anoctamin1 abolishes the calcium-activated chloride current in mouse vomeronasal sensory neurons.TRICK or TRP? What Trpc2(-/-) mice tell us about vomeronasal organ mediated innate behaviors Pregnancy and estrogen enhance neural progenitor-cell proliferation in the vomeronasal sensory epithelium.Development of the main olfactory system and main olfactory epithelium-dependent male mating behavior are altered in Go-deficient mice Identification of a Novel Gnao-Mediated Alternate Olfactory Signaling Pathway in Murine OSNs.Strain-specific Loss of Formyl Peptide Receptor 3 in the Murine Vomeronasal and Immune Systems Ominous odors: olfactory control of instinctive fear and aggression in mice.Heterotrimeric G protein subunit Gγ13 is critical to olfaction State-dependent responses to sex pheromones in mouse.Whole-genome resequencing of Xishuangbanna fighting chicken to identify signatures of selection A Sensor for Low Environmental Oxygen in the Mouse Main Olfactory Epithelium.The genomic basis of vomeronasal-mediated behaviour.Dichotomous Distribution of Putative Cholinergic Interneurons in Mouse Accessory Olfactory Bulb.Simultaneous deletion of floxed genes mediated by CaMKIIα-Cre in the brain and in male germ cells: application to conditional and conventional disruption of Goα.

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G protein G(alpha)o is essential for vomeronasal function and aggressive behavior in mice.

http://www.wikidata.org/entity/Q35149601

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

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G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

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type

label

G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

@ast

G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

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prefLabel

G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

@ast

G protein G(alpha)o is essenti ...... d aggressive behavior in mice.

@en

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Proceedings of the National Academy of Sciences of the United States of America

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G protein G(alpha)o is essential for vomeronasal function and aggressive behavior in mice

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Bernd Bufe

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Hiroaki Matsunami

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Joel Abramowitz

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Lutz Birnbaumer

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Philipp Hendrix

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Richard Roberts

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Vicky Katsoulidou

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P2860

Species specificity in major urinary proteins by parallel evolution

Dynamic instability of the major urinary protein gene family revealed by genomic and phenotypic comparisons between C57 and 129 strain mice

Structure of the human gene and two rat cDNAs encoding the alpha chain of GTP-binding regulatory protein Go: two different mRNAs are generated by alternative splicing

International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) family

Formyl peptide receptor-like proteins are a novel family of vomeronasal chemosensors

From pheromones to behavior

Identification of protein pheromones that promote aggressive behaviour

Altered sexual and social behaviors in trp2 mutant mice.

G(o) protein-dependent survival of primary accessory olfactory neurons

Sex-specific peptides from exocrine glands stimulate mouse vomeronasal sensory neurons

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108

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