Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype - Test2 - elhamkhani - TriplyDB

Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Anti-Tumor Effects of Second Generation β-Hydroxylase Inhibitors on Cholangiocarcinoma Development and Progression.Aspartate β-hydroxylase modulates cellular senescence through glycogen synthase kinase 3β in hepatocellular carcinoma.Aspartate-β-hydroxylase (ASPH): A potential therapeutic target in human malignant gliomas.Role of Notch signaling pathway in pancreatic cancer.Lambda phage-based vaccine induces antitumor immunity in hepatocellular carcinoma.Ethanol-Induced White Matter Atrophy Is Associated with Impaired Expression of Aspartyl-Asparaginyl-β-Hydroxylase (ASPH) and Notch Signaling in an Experimental Rat Model.MiR-200a inhibits cell proliferation and EMT by down-regulating the ASPH expression levels and affecting ERK and PI3K/Akt pathways in human hepatoma cells.Clinically relevant inflammatory breast cancer patient-derived xenograft-derived ex vivo model for evaluation of tumor-specific therapies.Aspartate beta-hydroxylase promotes cholangiocarcinoma progression by modulating RB1 phosphorylation

P2860

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P2860

Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

http://www.wikidata.org/entity/Q35176052

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2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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type

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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prefLabel

Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Aspartate β-Hydroxylase expression promotes a malignant pancreatic cellular phenotype

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Arihiro Aihara

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Chiung-Kuei Huang

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Christina Nadolny

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Gregory Gabriel

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Jack R Wands

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Qi Tang

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Qiushi Lin

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Rolf Carlson

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Waihong Chung

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Xiaoqun Dong

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P2860

Overexpression of human aspartyl(asparaginyl)beta-hydroxylase in hepatocellular carcinoma and cholangiocarcinoma

Cancer statistics, 2013

Overexpression of human aspartyl (asparaginyl) beta-hydroxylase is associated with malignant transformation

Aspartyl beta -hydroxylase (Asph) and an evolutionarily conserved isoform of Asph missing the catalytic domain share exons with junctin

Antisense oligodeoxynucleotides directed against aspartyl (asparaginyl) beta-hydroxylase suppress migration of cholangiocarcinoma cells

Role of aspartyl-(asparaginyl) beta-hydroxylase in placental implantation: Relevance to early pregnancy loss

Notch signalling: a simple pathway becomes complex

Concise review: clinical programs of stem cell therapies for liver and pancreas

WNT7B mediates autocrine Wnt/β-catenin signaling and anchorage-independent growth in pancreatic adenocarcinoma.

Upregulation of Wnt5a promotes epithelial-to-mesenchymal transition and metastasis of pancreatic cancer cells

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1231-1248

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