Attaching and effacing bacterial effector NleC suppresses epithelial inflammatory responses by inhibiting NF-κB and p38 mitogen-activated protein kinase activation.
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Inhibition of TLR signaling by a bacterial protein containing immunoreceptor tyrosine-based inhibitory motifsBacterial Toxins as Pathogen Weapons Against PhagocytesEffector triggered manipulation of host immune response elicited by different pathotypes of Escherichia coliThe Structure and Specificity of the Type III Secretion System Effector NleC Suggest a DNA Mimicry Mechanism of Substrate RecognitionStructure and mechanism of a type III secretion protease, NleCModulation of the host innate immune and inflammatory response by translocated bacterial proteinsEnteropathogenic Escherichia coli inhibits type I interferon- and RNase L-mediated host defense to disrupt intestinal epithelial cell barrier functionRecent advances in understanding enteric pathogenic Escherichia coli.The apoptogenic toxin AIP56 is a metalloprotease A-B toxin that cleaves NF-κb P65SIGIRR, a negative regulator of TLR/IL-1R signalling promotes Microbiota dependent resistance to colonization by enteric bacterial pathogens.Metalloprotease NleC suppresses host NF-κB/inflammatory responses by cleaving p65 and interfering with the p65/RPS3 interactionA novel mouse model of Campylobacter jejuni gastroenteritis reveals key pro-inflammatory and tissue protective roles for Toll-like receptor signaling during infection.The type III system-secreted effector EspZ localizes to host mitochondria and interacts with the translocase of inner mitochondrial membrane 17bInvolvement of the mannose receptor and p38 mitogen-activated protein kinase signaling pathway of the microdomain of the integral membrane protein after enteropathogenic Escherichia coli infection.SseK3 Is a Salmonella Effector That Binds TRIM32 and Modulates the Host's NF-κB Signalling ActivityNovel Host Proteins and Signaling Pathways in Enteropathogenic E. coli Pathogenesis Identified by Global Phosphoproteome Analysis.The Type III Effector NleD from Enteropathogenic Escherichia coli Differentiates between Host Substrates p38 and JNK.Enterotoxigenic Escherichia coli prevents host NF-κB activation by targeting IκBα polyubiquitination.Prolonged NF-κB activation by a macrophage inhibitory cytokine 1-linked signal in enteropathogenic Escherichia coli-infected epithelial cells.A Modular, Tn7-Based System for Making Bioluminescent or Fluorescent Salmonella and Escherichia coli StrainsInterference with nuclear factor kappaB signaling pathway by pathogen-encoded proteases: global and selective inhibition.Escherichia coli Type III Secretion System 2 ATPase EivC Is Involved in the Motility and Virulence of Avian Pathogenic Escherichia coli.Pathogenesis of human enterovirulent bacteria: lessons from cultured, fully differentiated human colon cancer cell linesEscherichia coli virulence protein NleH1 interaction with the v-Crk sarcoma virus CT10 oncogene-like protein (CRKL) governs NleH1 inhibition of the ribosomal protein S3 (RPS3)/nuclear factor κB (NF-κB) pathway.Apoptosis in response to microbial infection induces autoreactive TH17 cellsInnate host responses to enteric bacterial pathogens: a balancing act between resistance and tolerance.The impact of Toll-like receptors on bacterial virulence strategies.Hijacking of death receptor signaling by bacterial pathogen effectors.Bringing down the host: enteropathogenic and enterohaemorrhagic Escherichia coli effector-mediated subversion of host innate immune pathways.Role of virulence factors on host inflammatory response induced by diarrheagenic Escherichia coli pathotypes.Modulation of host signaling in the inflammatory response by enteropathogenic Escherichia coli virulence proteins.A quantitative proteomic screen of the Campylobacter jejuni flagellar-dependent secretome.Flagellin Induces β-Defensin 2 in Human Colonic Ex vivo Infection with Enterohemorrhagic Escherichia coli.Identification of a Distinct Substrate-binding Domain in the Bacterial Cysteine Methyltransferase Effectors NleE and OspZ.A distinct regulatory sequence is essential for the expression of a subset of nle genes in attaching and effacing Escherichia coli.Model system based proteomics to understand the host response during bacterial infections.Cooperative Immune Suppression by Escherichia coli and Shigella Effector Proteins.Citrobacter rodentium Infection Model for the Analysis of Bacterial Pathogenesis and Mucosal Immunology.A Metalloprotease Homolog Venom Protein From a Parasitoid Wasp Suppresses the Toll Pathway in Host Hemocytes
P2860
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P2860
Attaching and effacing bacterial effector NleC suppresses epithelial inflammatory responses by inhibiting NF-κB and p38 mitogen-activated protein kinase activation.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Attaching and effacing bacteri ...... ted protein kinase activation.
@ast
Attaching and effacing bacteri ...... ted protein kinase activation.
@en
type
label
Attaching and effacing bacteri ...... ted protein kinase activation.
@ast
Attaching and effacing bacteri ...... ted protein kinase activation.
@en
prefLabel
Attaching and effacing bacteri ...... ted protein kinase activation.
@ast
Attaching and effacing bacteri ...... ted protein kinase activation.
@en
P2093
P2860
P50
P356
P1476
Attaching and effacing bacteri ...... ated protein kinase activation
@en
P2093
B Brett Finlay
Ho Pan Sham
Justin M Chan
Mohammed A Khan
Wanyin Deng
P2860
P304
P356
10.1128/IAI.05033-11
P407
P577
2011-07-11T00:00:00Z