microRNA-210 is upregulated in hypoxic cardiomyocytes through Akt- and p53-dependent pathways and exerts cytoprotective effects - Test2 - elhamkhani - TriplyDB

microRNA-210 is upregulated in hypoxic cardiomyocytes through Akt- and p53-dependent pathways and exerts cytoprotective effects

microRNA-210 is upregulated in hypoxic cardiomyocytes through Akt- and p53-dependent pathways and exerts cytoprotective effects

http://www.wikidata.org/entity/Q35395850

about

Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and Apoptosis microRNAs as Potential Biomarkers in Adrenocortical Cancer: Progress and Challenges HypoxamiRs and cancer: from biology to targeted therapy Small molecules, big effects: the role of microRNAs in regulation of cardiomyocyte death Role of microRNAs in the reperfused myocardium towards post-infarct remodelling OxymiRs in cutaneous development, wound repair and regeneration miRNA Deregulation in Cancer Cells and the Tumor Microenvironment The role of microRNAs in regulating myocardial ischemia reperfusion injury Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.Evaluation of Plasma MicroRNAs as Diagnostic and Prognostic Biomarkers in Pancreatic Adenocarcinoma: miR-196a and miR-210 Could Be Negative and Positive Prognostic Markers, Respectively.Multiple functions of hypoxia-regulated miR-210 in cancer.miR-210 promotes IPF fibroblast proliferation in response to hypoxia.HypoxamiR regulation and function in ischemic cardiovascular diseases.Hypoxia-induced miR-210 modulates tissue response to acute peripheral ischemia.MicroRNA-377 regulates mesenchymal stem cell-induced angiogenesis in ischemic hearts by targeting VEGF ROD1 is a seedless target gene of hypoxia-induced miR-210.Global effect of inauhzin on human p53-responsive transcriptome Gestational hypoxia and epigenetic programming of brain development disorders.Differential expression of microRNAs in ischemic heart disease.Up-regulation of microRNA-21 mediates isoflurane-induced protection of cardiomyocytes.Integromics network meta-analysis on cardiac aging offers robust multi-layer modular signatures and reveals micronome synergism.Exosomes/microvesicles from induced pluripotent stem cells deliver cardioprotective miRNAs and prevent cardiomyocyte apoptosis in the ischemic myocardium.Down-regulation of ARNT promotes cancer metastasis by activating the fibronectin/integrin β1/FAK axis.Olmesartan restores the protective effect of remote ischemic perconditioning against myocardial ischemia/reperfusion injury in spontaneously hypertensive rats.miR-210: fine-tuning the hypoxic response.Coronary Artery-Bypass-Graft Surgery Increases the Plasma Concentration of Exosomes Carrying a Cargo of Cardiac MicroRNAs: An Example of Exosome Trafficking Out of the Human Heart with Potential for Cardiac Biomarker Discovery.MiR-1246: a new link of the p53 family with cancer and Down syndrome.Function and clinical potential of microRNAs in hepatocellular carcinoma.Hypoxia-inducible factor 1-α induces miR-210 in normoxic differentiating myoblasts.Reactive oxygen species-responsive miR-210 regulates proliferation and migration of adipose-derived stem cells via PTPN2 MicroRNA-210 decreases heme levels by targeting ferrochelatase in cardiomyocytes.Effects of knockdown of miR-210 in combination with ionizing radiation on human hepatoma xenograft in nude mice.Hypoxia-upregulated microRNA-630 targets Dicer, leading to increased tumor progression.Mitochondrial p38β and manganese superoxide dismutase interaction mediated by estrogen in cardiomyocytes.RNAs that make a heart beat.Protection of Human Umbilical Vein Endothelial Cells against Oxidative Stress by MicroRNA-210.Suppression of microRNA-16 protects against acute myocardial infarction by reversing beta2-adrenergic receptor down-regulation in rats.miR-210: the master hypoxamir.miRNAs as potential therapeutic targets for age-related macular degeneration.miRNAs in ESC differentiation.

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microRNA-210 is upregulated in hypoxic cardiomyocytes through Akt- and p53-dependent pathways and exerts cytoprotective effects

http://www.wikidata.org/entity/Q35395850

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

microRNA-210 is upregulated in ...... exerts cytoprotective effects

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Hossein Ardehali

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R Kannan Mutharasan

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Varun Nagpal

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Yoshihiko Ichikawa

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P2860

MicroRNA-210 regulates mitochondrial free radical response to hypoxia and krebs cycle in cancer cells by targeting iron sulfur cluster protein ISCU

Molecular cloning and characterization of a human AIF-like gene with ability to induce apoptosis

miR-210 is overexpressed in late stages of lung cancer and mediates mitochondrial alterations associated with modulation of HIF-1 activity

MicroRNA-210 modulates endothelial cell response to hypoxia and inhibits the receptor tyrosine kinase ligand Ephrin-A3

MicroRNA-210 regulates cancer cell proliferation through targeting fibroblast growth factor receptor-like 1 (FGFRL1)

MicroRNA-210 as a novel therapy for treatment of ischemic heart disease

Ischemic preconditioning augments survival of stem cells via miR-210 expression by targeting caspase-8-associated protein 2

Hypoxia-inducible mir-210 regulates normoxic gene expression involved in tumor initiation.

Most mammalian mRNAs are conserved targets of microRNAs

MicroRNA-210 controls mitochondrial metabolism during hypoxia by repressing the iron-sulfur cluster assembly proteins ISCU1/2

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3197368

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