Effects of non-enzymatic glycation on cancellous bone fragility. - Test2 - elhamkhani - TriplyDB

Effects of non-enzymatic glycation on cancellous bone fragility.

Effects of non-enzymatic glycation on cancellous bone fragility.

http://www.wikidata.org/entity/Q35429093

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Does accumulation of advanced glycation end products contribute to the aging phenotype?Biology of Bone Tissue: Structure, Function, and Factors That Influence Bone Cells Pathogenesis, management and prevention of atypical femoral fractures Advanced glycation end products play adverse proinflammatory activities in osteoporosis American Society of Biomechanics Journal of Biomechanics Award 2013: cortical bone tissue mechanical quality and biological mechanisms possibly underlying atypical fractures.Vibrational spectroscopic imaging for the evaluation of matrix and mineral chemistry Heterogeneous glycation of cancellous bone and its association with bone quality and fragility N-phenacylthiazolium bromide reduces bone fragility induced by nonenzymatic glycation Bone protein extraction without demineralization using principles from hydroxyapatite chromatography Effect of age on mechanical properties of the collagen phase in different orientations of human cortical bone Small animal bone biomechanics.The Role of Water Compartments in the Material Properties of Cortical Bone Relationship of an advanced glycation end product, plasma carboxymethyl-lysine, with slow walking speed in older adults: the InCHIANTI study.Bisphosphonates and bone quality.Effects of trabecular type and orientation on microdamage susceptibility in trabecular bone.Osteopontin deficiency increases bone fragility but preserves bone mass.In situ accumulation of advanced glycation endproducts (AGEs) in bone matrix and its correlation with osteoclastic bone resorption.The effect of antiresorptives on bone quality.Biochemical characterization of major bone-matrix proteins using nanoscale-size bone samples and proteomics methodology.Alterations in T2 relaxation magnetic resonance imaging of the ovine intervertebral disc due to nonenzymatic glycation.Age-related changes in the plasticity and toughness of human cortical bone at multiple length scales.Aging and bone.Dilatational band formation in bone Determinants of microdamage in elderly human vertebral trabecular bone Increasing duration of type 1 diabetes perturbs the strength-structure relationship and increases brittleness of bone.Chronic ingestion of advanced glycation end products induces degenerative spinal changes and hypertrophy in aging pre-diabetic mice.Methods for assessing bone quality: a review.Role of trabecular microarchitecture in the formation, accumulation, and morphology of microdamage in human cancellous bone.Focal therapeutic irradiation induces an early transient increase in bone glycation.Non-enzymatic glycation alters microdamage formation in human cancellous bone.Reduced cortical bone compositional heterogeneity with bisphosphonate treatment in postmenopausal women with intertrochanteric and subtrochanteric fractures.Alterations in intervertebral disc composition, matrix homeostasis and biomechanical behavior in the UCD-T2DM rat model of type 2 diabetes.Lathyrism-induced alterations in collagen cross-links influence the mechanical properties of bone material without affecting the mineral Hyperglycemia increases the expression levels of sclerostin in a reactive oxygen species- and tumor necrosis factor-alpha-dependent manner Differences in non-enzymatic glycation and collagen cross-links between human cortical and cancellous bone Association between non-enzymatic glycation, resorption, and microdamage in human tibial cortices.Effects of bone matrix proteins on fracture and fragility in osteoporosis.Matrix metalloproteinase-13 is required for osteocytic perilacunar remodeling and maintains bone fracture resistance.Multiscale Predictors of Femoral Neck In Situ Strength in Aging Women: Contributions of BMD, Cortical Porosity, Reference Point Indentation, and Nonenzymatic Glycation.Effect of type 2 diabetes-related non-enzymatic glycation on bone biomechanical properties.

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P2860

Effects of non-enzymatic glycation on cancellous bone fragility.

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Contribution of collagen and mineral to the elastic-plastic properties of bone

Accelerated age-related browning of human collagen in diabetes mellitus

Formation of pentosidine during nonenzymatic browning of proteins by glucose. Identification of glucose and other carbohydrates as possible precursors of pentosidine in vivo

Bone indentation recovery time correlates with bond reforming time.

Influence of nonenzymatic glycation on biomechanical properties of cortical bone.

Sacrificial bonds and hidden length dissipate energy as mineralized fibrils separate during bone fracture.

Accumulation of Maillard reaction products in skin collagen in diabetes and aging.

Trabecular microfracture and the influence of pyridinium and non-enzymatic glycation-mediated collagen cross-links.

Pentosidine: a molecular marker for the cumulative damage to proteins in diabetes, aging, and uremia.

Nonenzymatic glycation of collagen in aging and diabetes.

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