Gene knockout mice establish a primary protective role for major histocompatibility complex class II-restricted responses in Chlamydia trachomatis genital tract infection.
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The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged SwordIntravaginal Chlamydia trachomatis Challenge Infection Elicits TH1 and TH17 Immune Responses in Mice That Promote Pathogen Clearance and Genital Tract DamageCollateral Damage: Detrimental Effect of Antibiotics on the Development of Protective Immune MemoryTissue-Resident T Cells as the Central Paradigm of Chlamydia ImmunityRole for inducible nitric oxide synthase in protection from chronic Chlamydia trachomatis urogenital disease in mice and its regulation by oxygen free radicalsInduction of protective immunity against Chlamydia muridarum intravaginal infection with a chlamydial glycogen phosphorylaseA T cell epitope-based vaccine protects against chlamydial infection in HLA-DR4 transgenic mice.CD8+ T cells recognize an inclusion membrane-associated protein from the vacuolar pathogen Chlamydia trachomatisImmunobiological outcomes of repeated chlamydial infection from two models of within-host population dynamics.Expression of mucosal homing receptor alpha4beta7 is associated with enhanced migration to the Chlamydia-infected murine genital mucosa in vivo.Differential sensitivities of Chlamydia trachomatis strains to inhibitory effects of gamma interferonHeat shock protein expression and immunity in chlamydial infectionsImmunization with the attenuated plasmidless Chlamydia trachomatis L2(25667R) strain provides partial protection in a murine model of female genitourinary tract infectionSimultaneous Subcutaneous and Intranasal Administration of a CAF01-Adjuvanted Chlamydia Vaccine Elicits Elevated IgA and Protective Th1/Th17 Responses in the Genital Tract.Pathogenesis of Chlamydia induced pelvic inflammatory disease.Inducible nitric oxide synthase does not affect resolution of murine chlamydial genital tract infections or eradication of chlamydiae in primary murine cell culture.Neither interleukin-6 nor inducible nitric oxide synthase is required for clearance of Chlamydia trachomatis from the murine genital tract epithelium.Protection against ascending infection of the genital tract by Chlamydia trachomatis is associated with recruitment of major histocompatibility complex class II antigen-presenting cells into uterine tissueA vaccine and monoclonal antibodies that enhance mouse resistance to Candida albicans vaginal infection.Role of NK cells in early host response to chlamydial genital infectionThe Role of Non-Cognate T Cell Stimulation during Intracellular Bacterial Infection.The effect of infectious dose on humoral and cellular immune responses in Chlamydophila caviae primary ocular infectionChlamydial colonization of multiple mucosae following infection by any mucosal route.Clearance of Chlamydia trachomatis from the murine genital mucosa does not require perforin-mediated cytolysis or Fas-mediated apoptosis.Immunity to Chlamydia trachomatis mouse pneumonitis induced by vaccination with live organisms correlates with early granulocyte-macrophage colony-stimulating factor and interleukin-12 production and with dendritic cell-like maturation.Interleukin-12 production is required for chlamydial antigen-pulsed dendritic cells to induce protection against live Chlamydia trachomatis infection.Subclinical chlamydial infection of the female mouse genital tract generates a potent protective immune response: implications for development of live attenuated chlamydial vaccine strains.Differential regulation of CD4 lymphocyte recruitment between the upper and lower regions of the genital tract during Chlamydia trachomatis infectionPolymorphonuclear neutrophils are necessary for the recruitment of CD8(+) T cells in the liver in a pregnant mouse model of Chlamydophila abortus (Chlamydia psittaci serotype 1) infection.In situ analysis of the evolution of the primary immune response in murine Chlamydia trachomatis genital tract infection.Priming with Chlamydia trachomatis major outer membrane protein (MOMP) DNA followed by MOMP ISCOM boosting enhances protection and is associated with increased immunoglobulin A and Th1 cellular immune responses.Immunity to murine Chlamydia trachomatis genital tract reinfection involves B cells and CD4(+) T cells but not CD8(+) T cells.Inflammation and clearance of Chlamydia trachomatis in enteric and nonenteric mucosae.Resolution of secondary Chlamydia trachomatis genital tract infection in immune mice with depletion of both CD4+ and CD8+ T cells.Early local cytokine profiles in strains of mice with different outcomes from chlamydial genital tract infectionChlamydia trachomatis persistence in the female mouse genital tract: inducible nitric oxide synthase and infection outcomeImmunization with the Chlamydia trachomatis mouse pneumonitis major outer membrane protein can elicit a protective immune response against a genital challengeThe quantity of nitric oxide released by macrophages regulates Chlamydia-induced disease.The protective effect of antibody in immunity to murine chlamydial genital tract reinfection is independent of immunoglobulin A.Enhanced upper genital tract pathologies by blocking Tim-3 and PD-L1 signaling pathways in mice intravaginally infected with Chlamydia muridarum.
P2860
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P2860
Gene knockout mice establish a primary protective role for major histocompatibility complex class II-restricted responses in Chlamydia trachomatis genital tract infection.
description
1995 nî lūn-bûn
@nan
1995 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Gene knockout mice establish a ...... matis genital tract infection.
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Gene knockout mice establish a ...... matis genital tract infection.
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type
label
Gene knockout mice establish a ...... matis genital tract infection.
@ast
Gene knockout mice establish a ...... matis genital tract infection.
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prefLabel
Gene knockout mice establish a ...... matis genital tract infection.
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Gene knockout mice establish a ...... matis genital tract infection.
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P2093
P2860
P1476
Gene knockout mice establish a ...... matis genital tract infection.
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P2093
P2860
P304
P407
P577
1995-12-01T00:00:00Z