Genome-wide ChIP-Seq reveals a dramatic shift in the binding of the transcription factor erythroid Kruppel-like factor during erythrocyte differentiation.
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The CACCC-binding protein KLF3/BKLF represses a subset of KLF1/EKLF target genes and is required for proper erythroid maturation in vivoKruppel-like factor 1 (KLF1), KLF2, and Myc control a regulatory network essential for embryonic erythropoiesisKrüppeling erythropoiesis: an unexpected broad spectrum of human red blood cell disorders due to KLF1 variantsAnalysis of disease-causing GATA1 mutations in murine gene complementation systemsTMEM14C is required for erythroid mitochondrial heme metabolismRepression by RB1 characterizes genes involved in the penultimate stage of erythroid developmentIdentification of biologically relevant enhancers in human erythroid cells.Extrinsic and intrinsic control by EKLF (KLF1) within a specialized erythroid niche.A crucial role for the ubiquitously expressed transcription factor Sp1 at early stages of hematopoietic specificationHeme-bound iron activates placenta growth factor in erythroid cells via erythroid Krüppel-like factorHistones to the cytosol: exportin 7 is essential for normal terminal erythroid nuclear maturationTissue-specific mitotic bookmarking by hematopoietic transcription factor GATA1.Generation of mice deficient in both KLF3/BKLF and KLF8 reveals a genetic interaction and a role for these factors in embryonic globin gene silencingRegulation of delta-aminolevulinic acid dehydratase by krüppel-like factor 1.Browning of human adipocytes requires KLF11 and reprogramming of PPARγ superenhancers.KLF1-null neonates display hydrops fetalis and a deranged erythroid transcriptome.Nuclear RNA sequencing of the mouse erythroid cell transcriptomeUpstream distal regulatory elements contact the Lmo2 promoter in mouse erythroid cellsDynamic shifts in occupancy by TAL1 are guided by GATA factors and drive large-scale reprogramming of gene expression during hematopoiesis.Alternative splicing of EKLF/KLF1 in murine primary erythroid tissuesKrüppel-like factors are effectors of nuclear receptor signaling.Dynamics of the epigenetic landscape during erythroid differentiation after GATA1 restoration.The mTORC1/4E-BP pathway coordinates hemoglobin production with L-leucine availability.The IKAROS interaction with a complex including chromatin remodeling and transcription elongation activities is required for hematopoiesis.Altered chromatin occupancy of master regulators underlies evolutionary divergence in the transcriptional landscape of erythroid differentiationThe hypersensitive sites of the murine β-globin locus control region act independently to affect nuclear localization and transcriptional elongation.A novel comparative pattern count analysis reveals a chronic ethanol-induced dynamic shift in immediate early NF-κB genome-wide promoter binding during liver regeneration.ATP-dependent mitochondrial porphyrin importer ABCB6 protects against phenylhydrazine toxicity.Functional interactions between erythroid Krüppel-like factor (EKLF/KLF1) and protein phosphatase PPM1B/PP2CβCETCh-seq: CRISPR epitope tagging ChIP-seq of DNA-binding proteins.The DEK Oncoprotein Is a Critical Component of the EKLF/KLF1 Enhancer in Erythroid Cells.GATA-1 utilizes Ikaros and polycomb repressive complex 2 to suppress Hes1 and to promote erythropoiesisCombinatorial assembly of developmental stage-specific enhancers controls gene expression programs during human erythropoiesis.SBR-Blood: systems biology repository for hematopoietic cells.Novel roles for KLF1 in erythropoiesis revealed by mRNA-seq.Manganese superoxide dismutase depletion in murine hematopoietic stem cells perturbs iron homeostasis, globin switching, and epigenetic control in erythrocyte precursor cells.GATA-1 genome-wide occupancy associates with distinct epigenetic profiles in mouse fetal liver erythropoiesis.Insight into GATA1 transcriptional activity through interrogation of cis elements disrupted in human erythroid disordersLdb1-nucleated transcription complexes function as primary mediators of global erythroid gene activation.Regions outside the DNA-binding domain are critical for proper in vivo specificity of an archetypal zinc finger transcription factor
P2860
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P2860
Genome-wide ChIP-Seq reveals a dramatic shift in the binding of the transcription factor erythroid Kruppel-like factor during erythrocyte differentiation.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@ast
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@en
type
label
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@ast
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@en
prefLabel
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@ast
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@en
P2093
P2860
P1433
P1476
Genome-wide ChIP-Seq reveals a ...... g erythrocyte differentiation.
@en
P2093
Andre M Pilon
David M Bodine
Elliott H Margulies
Laurie A Steiner
NISC Comparative Sequencing Center
Patrick G Gallagher
Praveen F Cherukuri
Stacie M Anderson
Stephen Wincovitch
Subramanian S Ajay
P2860
P304
P356
10.1182/BLOOD-2011-05-355107
P407
P577
2011-09-06T00:00:00Z