The inflammatory response in myocardial injury, repair, and remodelling.
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Targeting the Innate Immune Response to Improve Cardiac Graft Recovery after Heart Transplantation: Implications for the Donation after Cardiac DeathUnfolding the Role of Large Heat Shock Proteins: New Insights and Therapeutic ImplicationsFrom C-Reactive Protein to Interleukin-6 to Interleukin-1: Moving Upstream To Identify Novel Targets for AtheroprotectionModulators of Macrophage Polarization Influence Healing of the Infarcted MyocardiumThe circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarctionMechanobiology of myofibroblast adhesion in fibrotic cardiac diseaseGetting to the heart of intracellular glucocorticoid regeneration: 11β-HSD1 in the myocardiumCardiac fibrosis in myocardial infarction-from repair and remodeling to regenerationThe role of notch in the cardiovascular system: potential adverse effects of investigational notch inhibitorsPhysiological Implications of Myocardial Scar StructureDevelopment of target-specific liposomes for delivering small molecule drugs after reperfused myocardial infarction.Telomerase Is Essential for Zebrafish Heart RegenerationA strategy for the identification of combinatorial bioactive compounds contributing to the holistic effect of herbal medicines.Human cardiac fibroblasts adaptive responses to controlled combined mechanical strain and oxygen changes in vitro.Methotrexate carried in lipid core nanoparticles reduces myocardial infarction size and improves cardiac function in rats.Relationships of growth factors, proinflammatory cytokines, and anti-inflammatory cytokines with long-term clinical results of autologous bone marrow mononuclear cell transplantation in STEMITime course of VCAM-1 expression in reperfused myocardial infarction in swine and its relation to retention of intracoronary administered bone marrow-derived mononuclear cells.Sestrin2 Suppresses Classically Activated Macrophages-Mediated Inflammatory Response in Myocardial Infarction through Inhibition of mTORC1 Signaling.Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.Protective Effects of Berberine on Isoproterenol-Induced Acute Myocardial Ischemia in Rats through Regulating HMGB1-TLR4 Axis.Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.Bisphenol A alters autonomic tone and extracellular matrix structure and induces sex-specific effects on cardiovascular function in male and female CD-1 mice.Cardiac autoimmunity as a novel biomarker, mediator, and therapeutic target of heart disease in type 1 diabetes.The evaluation of plasma and leukocytic IL-37 expression in early inflammation in patients with acute ST-segment elevation myocardial infarction after PCI.Identification of NF-κB inhibitors in Qishenyiqi dropping pills for myocardial infarction treatment based on bioactivity-integrated UPLC-Q/TOF MS.TAK-242, a Toll-Like Receptor 4 Antagonist, Protects against Aldosterone-Induced Cardiac and Renal InjuryBone Marrow Mononuclear Cell Transplantation Restores Inflammatory Balance of Cytokines after ST Segment Elevation Myocardial Infarction.Extracellular mtDNA activates NF-κB via toll-like receptor 9 and induces cell death in cardiomyocytes.Macrophages mediate cardioprotective cellular postconditioning in acute myocardial infarctionWnt10b Gain-of-Function Improves Cardiac Repair by Arteriole Formation and Attenuation of Fibrosis.Cardiac-Restricted IGF-1Ea Overexpression Reduces the Early Accumulation of Inflammatory Myeloid Cells and Mediates Expression of Extracellular Matrix Remodelling Genes after Myocardial InfarctionCardiosphere-Derived Cells Facilitate Heart Repair by Modulating M1/M2 Macrophage Polarization and Neutrophil Recruitment.Targeting Interleukin-1β Reduces Leukocyte Production After Acute Myocardial InfarctionThe role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction.Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities.Reversible cardiac hypertrophy induced by PEG-coated gold nanoparticles in mice.The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarctionTranscriptional control of cardiac fibroblast plasticity.Innate immune cells in ischaemic heart disease: does myocardial infarction beget myocardial infarction?Prospective Evaluation of 18F-Fluorodeoxyglucose Uptake in Postischemic Myocardium by Simultaneous Positron Emission Tomography/Magnetic Resonance Imaging as a Prognostic Marker of Functional Outcome.
P2860
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P2860
The inflammatory response in myocardial injury, repair, and remodelling.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
The inflammatory response in myocardial injury, repair, and remodelling.
@ast
The inflammatory response in myocardial injury, repair, and remodelling.
@en
type
label
The inflammatory response in myocardial injury, repair, and remodelling.
@ast
The inflammatory response in myocardial injury, repair, and remodelling.
@en
prefLabel
The inflammatory response in myocardial injury, repair, and remodelling.
@ast
The inflammatory response in myocardial injury, repair, and remodelling.
@en
P2860
P356
P1476
The inflammatory response in myocardial injury, repair, and remodelling
@en
P2093
Nikolaos G Frangogiannis
P2860
P304
P356
10.1038/NRCARDIO.2014.28
P407
P577
2014-03-25T00:00:00Z