Isolation of CD4-independent primary human immunodeficiency virus type 1 isolates that are syncytium inducing and acutely cytopathic for CD8+ lymphocytes.
about
HIV and the chemokine system: 10 years later.Soluble Envelope Glycoprotein Trimers from a CD4-Independent HIV-1 Elicit Antibody-Dependent Cellular Cytotoxicity-Mediating Antibodies in Guinea PigsCKR-L3, a deletion version CCR6-isoform shows coreceptor-activity for limited human and simian immunodeficiency viruses.CD4-independent human immunodeficiency virus infection involves participation of endocytosis and cathepsin B.Contribution of intrinsic reactivity of the HIV-1 envelope glycoproteins to CD4-independent infection and global inhibitor sensitivity.Binding mode characterization of NBD series CD4-mimetic HIV-1 entry inhibitors by X-ray structure and resistance study.HIV-1 utilizes the CXCR4 chemokine receptor to infect multipotent hematopoietic stem and progenitor cells.CD4-independent use of the CCR5 receptor by sequential primary SIVsm isolatesAcquisition of CD4-dependence by CD4-independent SIV passaged in human peripheral blood mononuclear cellsHIV entry inhibitors: a new generation of antiretroviral drugs.Role of the long cytoplasmic domain of the SIV Env glycoprotein in early and late stages of infection.Retrovirus entry by endocytosis and cathepsin proteases.Infection of CD8+CD45RO+ memory T-cells by HIV-1 and their proliferative response.A single amino acid change and truncated TM are sufficient for simian immunodeficiency virus to enter cells using CCR5 in a CD4-independent pathway.Matrix and envelope coevolution revealed in a patient monitored since primary infection with human immunodeficiency virus type 1.High levels of human immunodeficiency virus infection of CD8 lymphocytes expressing CD4 in vivo.CXCR4-dependent infection of CD8+, but not CD4+, lymphocytes by a primary human immunodeficiency virus type 1 isolate.Candidate antibody-based therapeutics against HIV-1."Cytoplasmic domain effects on exposure of co-receptor-binding sites of HIV-1 Env".Activity of the HIV-1 attachment inhibitor BMS-626529, the active component of the prodrug BMS-663068, against CD4-independent viruses and HIV-1 envelopes resistant to other entry inhibitors.HIV gp120 H375 is unique to HIV-1 subtype CRF01_AE and confers strong resistance to the entry inhibitor BMS-599793, a candidate microbicide drug.Down-modulation of CD8αβ is a fundamental activity of primate lentiviral Nef proteins.Homology models of the HIV-1 attachment inhibitor BMS-626529 bound to gp120 suggest a unique mechanism of action.
P2860
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P2860
Isolation of CD4-independent primary human immunodeficiency virus type 1 isolates that are syncytium inducing and acutely cytopathic for CD8+ lymphocytes.
description
2004 nî lūn-bûn
@nan
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@ast
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@en
type
label
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@ast
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@en
prefLabel
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@ast
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@en
P2093
P2860
P1433
P1476
Isolation of CD4-independent p ...... topathic for CD8+ lymphocytes.
@en
P2093
Bouchra Zerhouni
Julie A E Nelson
Kunal Saha
P2860
P304
P356
10.1128/JVI.78.3.1243-1255.2004
P407
P577
2004-02-01T00:00:00Z