Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis. - Test2 - elhamkhani - TriplyDB

Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.

Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.

http://www.wikidata.org/entity/Q35550587

about

NF-κB in inflammation and renal diseases Soluble Fn14 Is Detected and Elevated in Mouse and Human Kidney Disease Anti-Double-Stranded DNA IgG Participates in Renal Fibrosis through Suppressing the Suppressor of Cytokine Signaling 1 Signals.TWEAK/Fn14 Activation Participates in Ro52-Mediated Photosensitization in Cutaneous Lupus Erythematosus Biomarkers for kidney involvement in pediatric lupus.TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Accelerates the Progression of Renal Fibrosis in Lupus Nephritis by Activating SMAD and p38 MAPK in TGF-β1 Signaling Pathway Role of the TWEAK/Fn14 pathway in autoimmune diseases.Beyond TNF: TNF superfamily cytokines as targets for the treatment of rheumatic diseases.Roles of tumour necrosis factor-related weak inducer of apoptosis/fibroblast growth factor-inducible 14 pathway in lupus nephritis.Fn14 deficiency protects lupus-prone mice from histological lupus erythematosus-like skin inflammation induced by ultraviolet light.Distinct effects of mycophenolate mofetil and cyclophosphamide on renal fibrosis in NZBWF1/J mice.Non-canonical NFκB activation promotes chemokine expression in podocytes Transcription factor 7-like 1 dysregulates keratinocyte differentiation through upregulating lipocalin 2.TWEAK/Fn14 Activation Participates in Skin Inflammation.TWEAK-Fn14 Signaling Activates Myofibroblasts to Drive Progression of Fibrotic Kidney Disease.CSF-1R inhibition attenuates renal and neuropsychiatric disease in murine lupus.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Activates Type I Interferon Signals in Lupus Nephritis.NF-κB inducing kinase is a therapeutic target for systemic lupus erythematosus.TWEAK is not elevated in patients with newly diagnosed inflammatory bowel disease.TWEAK and RIPK1 mediate a second wave of cell death during AKI.TWEAK/Fn14 activation induces keratinocyte proliferation under psoriatic inflammation.Tnfrsf12a-Mediated Atherosclerosis Signaling and Inflammatory Response as a Common Protection Mechanism of Shuxuening Injection Against Both Myocardial and Cerebral Ischemia-Reperfusion Injuries.Repeated administration of alpha-galactosylceramide ameliorates experimental lupus nephritis in mice.Topical TWEAK Accelerates Healing of Experimental Burn Wounds in Mice.TWEAK increases CD74 expression and sensitizes to DDT proinflammatory actions in tubular cells.

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P2860

Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.

http://www.wikidata.org/entity/Q35550587

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

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Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

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type

label

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

@ast

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

@en

prefLabel

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

@ast

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

@en

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Journal of the American Society of Nephrology

P1476

Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.

@en

P2093

Alexander Misharin

http://www.w3.org/2001/XMLSchema#string

Chaim Putterman

http://www.w3.org/2001/XMLSchema#string

Harris Perlman

http://www.w3.org/2001/XMLSchema#string

Jennifer S Michaelson

http://www.w3.org/2001/XMLSchema#string

Jing Wen

http://www.w3.org/2001/XMLSchema#string

Lan Wu

http://www.w3.org/2001/XMLSchema#string

Leal C Herlitz

http://www.w3.org/2001/XMLSchema#string

Linda C Burkly

http://www.w3.org/2001/XMLSchema#string

Ping Wu

http://www.w3.org/2001/XMLSchema#string

Rahul D Pawar

http://www.w3.org/2001/XMLSchema#string

P2860

TWEAK induces apoptosis through a death-signaling complex comprising receptor-interacting protein 1 (RIP1), Fas-associated death domain (FADD), and caspase-8

Adipocytes as immune cells: differential expression of TWEAK, BAFF, and APRIL and their receptors (Fn14, BAFF-R, TACI, and BCMA) at different stages of normal and pathological adipose tissue development

TWEAK induces angiogenesis and proliferation of endothelial cells

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

IgG anti-cardiolipin antibodies in murine lupus.

TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.

Measurement of autoantibodies using multiplex methodology in patients with systemic lupus erythematosus.

TWEAK affects keratinocyte G2/M growth arrest and induces apoptosis through the translocation of the AIF protein to the nucleus.

Lupus nephritis: a critical review.

TWEAK-independent Fn14 self-association and NF-κB activation is mediated by the C-terminal region of the Fn14 cytoplasmic domain

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1053-1070

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10.1681/ASN.2014030233

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5

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26

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25270074

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