Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.
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NF-κB in inflammation and renal diseasesSoluble Fn14 Is Detected and Elevated in Mouse and Human Kidney DiseaseAnti-Double-Stranded DNA IgG Participates in Renal Fibrosis through Suppressing the Suppressor of Cytokine Signaling 1 Signals.TWEAK/Fn14 Activation Participates in Ro52-Mediated Photosensitization in Cutaneous Lupus ErythematosusBiomarkers for kidney involvement in pediatric lupus.TWEAK/Fn14 Signaling Involvement in the Pathogenesis of Cutaneous Disease in the MRL/lpr Model of Spontaneous Lupus.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Accelerates the Progression of Renal Fibrosis in Lupus Nephritis by Activating SMAD and p38 MAPK in TGF-β1 Signaling PathwayRole of the TWEAK/Fn14 pathway in autoimmune diseases.Beyond TNF: TNF superfamily cytokines as targets for the treatment of rheumatic diseases.Roles of tumour necrosis factor-related weak inducer of apoptosis/fibroblast growth factor-inducible 14 pathway in lupus nephritis.Fn14 deficiency protects lupus-prone mice from histological lupus erythematosus-like skin inflammation induced by ultraviolet light.Distinct effects of mycophenolate mofetil and cyclophosphamide on renal fibrosis in NZBWF1/J mice.Non-canonical NFκB activation promotes chemokine expression in podocytesTranscription factor 7-like 1 dysregulates keratinocyte differentiation through upregulating lipocalin 2.TWEAK/Fn14 Activation Participates in Skin Inflammation.TWEAK-Fn14 Signaling Activates Myofibroblasts to Drive Progression of Fibrotic Kidney Disease.CSF-1R inhibition attenuates renal and neuropsychiatric disease in murine lupus.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Activates Type I Interferon Signals in Lupus Nephritis.NF-κB inducing kinase is a therapeutic target for systemic lupus erythematosus.TWEAK is not elevated in patients with newly diagnosed inflammatory bowel disease.TWEAK and RIPK1 mediate a second wave of cell death during AKI.TWEAK/Fn14 activation induces keratinocyte proliferation under psoriatic inflammation.Tnfrsf12a-Mediated Atherosclerosis Signaling and Inflammatory Response as a Common Protection Mechanism of Shuxuening Injection Against Both Myocardial and Cerebral Ischemia-Reperfusion Injuries.Repeated administration of alpha-galactosylceramide ameliorates experimental lupus nephritis in mice.Topical TWEAK Accelerates Healing of Experimental Burn Wounds in Mice.TWEAK increases CD74 expression and sensitizes to DDT proinflammatory actions in tubular cells.
P2860
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P2860
Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@ast
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@en
type
label
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@ast
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@en
prefLabel
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@ast
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@en
P2093
P2860
P356
P1476
Deficiency of fibroblast growt ...... d ameliorates lupus nephritis.
@en
P2093
Alexander Misharin
Chaim Putterman
Harris Perlman
Jennifer S Michaelson
Leal C Herlitz
Linda C Burkly
Rahul D Pawar
P2860
P304
P356
10.1681/ASN.2014030233
P577
2014-09-30T00:00:00Z