TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis. - Test2 - elhamkhani - TriplyDB

TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis.

TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis.

http://www.wikidata.org/entity/Q35606977

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Bleomycin induces molecular changes directly relevant to idiopathic pulmonary fibrosis: a model for "active" disease Genetic variants associated with idiopathic pulmonary fibrosis susceptibility and mortality: a genome-wide association study.Lung microbiome and disease progression in idiopathic pulmonary fibrosis: an analysis of the COMET study Targeting interleukin-13 with tralokinumab attenuates lung fibrosis and epithelial damage in a humanized SCID idiopathic pulmonary fibrosis model.Pathogenesis of idiopathic pulmonary fibrosis: review of recent findings.High and low molecular weight hyaluronic acid differentially regulate human fibrocyte differentiation.The nucleic acid scavenger polyamidoamine third-generation dendrimer inhibits fibroblast activation and granulation tissue contraction Lung fibroblasts from patients with idiopathic pulmonary fibrosis exhibit genome-wide differences in DNA methylation compared to fibroblasts from nonfibrotic lung Mechanosignaling through YAP and TAZ drives fibroblast activation and fibrosis New insights into the mechanisms of innate immune receptor signalling in fibrosis.Toll like receptors in diseases of the lung.Elevated expression of NEU1 sialidase in idiopathic pulmonary fibrosis provokes pulmonary collagen deposition, lymphocytosis, and fibrosis.TLR9 expression is required for the development of cigarette smoke-induced emphysema in mice.Animal models of fibrotic lung disease.HIF-1α regulates EMT via the Snail and β-catenin pathways in paraquat poisoning-induced early pulmonary fibrosis.Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report.Regulation of wound healing and organ fibrosis by toll-like receptors.Heterogeneity in fibroblast proliferation and survival in idiopathic pulmonary fibrosis.Translational models of lung disease.TLR9-induced interferon β is associated with protection from gammaherpesvirus-induced exacerbation of lung fibrosis.Emerging roles of innate immune signaling and toll-like receptors in fibrosis and systemic sclerosis.Regulation and Relevance of Myofibroblast Responses in Idiopathic Pulmonary Fibrosis.Modeling DNA damage-induced pneumopathy in mice: insight from danger signaling cascades.Microbiome in interstitial lung disease: from pathogenesis to treatment target.Senescent stromal cell-induced divergence and therapeutic resistance in T cell acute lymphoblastic leukemia/lymphoma The Lung Microbiome, Immunity, and the Pathogenesis of Chronic Lung Disease.Lung Pericytes and Resident Fibroblasts: Busy Multitaskers.Microbes Are Associated with Host Innate Immune Response in Idiopathic Pulmonary Fibrosis.Exploring Animal Models That Resemble Idiopathic Pulmonary Fibrosis.Aberrant innate immune sensing leads to the rapid progression of idiopathic pulmonary fibrosis.A Dataset of 26 Candidate Gene and Pro-Inflammatory Cytokine Variants for Association Studies in Idiopathic Pulmonary Fibrosis: Frequency Distribution in Normal Czech Population.Increased toll-like receptor 9 expression is associated with the severity of paraquat-induced lung injury in mice.Epigenetic mechanisms through which Toll-like receptor-9 drives idiopathic pulmonary fibrosis progression.Host-Microbial Interactions: Idiopathic Pulmonary Fibrosis in Technicolor.Toll-like receptor 9 in systemic sclerosis patients: relation to modified Rodnan skin score, disease severity, and functional status.Extracellular Mitochondrial DNA Is Generated by Fibroblasts and Predicts Death in Idiopathic Pulmonary Fibrosis.Protective Effect of Atazanavir Sulphate Against Pulmonary Fibrosis In Vivo and In Vitro.Modeling Idiopathic Pulmonary Fibrosis in Humanized SCID Mice.Anti-fibrotic Effects of CXCR4-Targeting i-body AD-114 in Preclinical Models of Pulmonary Fibrosis.Revealing the pathogenic and aging-related mechanisms of the enigmatic idiopathic pulmonary fibrosis. an integral model.

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P2860

TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis.

http://www.wikidata.org/entity/Q35606977

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2010年论文

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name

TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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type

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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Science Translational Medicine

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TLR9 differentiates rapidly fr ...... idiopathic pulmonary fibrosis.

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Alessia Meneghin

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Amrita D Joshi

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Ana Lucia Coelho

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Barry H Gross

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Beatrice Waters

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Cory M Hogaboam

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Elizabeth Day

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Ella A Kazerooni

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Fernando J Martinez

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Gabor Jarai

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P2860

Inhibition of fibrocyte differentiation by serum amyloid P

Cellular and molecular mechanisms of fibrosis

Human lung cancer cells express functionally active Toll-like receptor 9

TGF-beta1 induces human alveolar epithelial to mesenchymal cell transition (EMT)

Idiopathic interstitial pneumonia: what is the effect of a multidisciplinary approach to diagnosis?

Evidence that fibroblasts derive from epithelium during tissue fibrosis

Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

Circulating peripheral blood fibrocytes in human fibrotic interstitial lung disease.

Functional Wnt signaling is increased in idiopathic pulmonary fibrosis

Liver cancer-derived hepatitis C virus core proteins shift TGF-beta responses from tumor suppression to epithelial-mesenchymal transition

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57

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47742686

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21068441

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pulmonary fibrosis

Toll-like receptor

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3235647

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