Hesr1 and Hesr3 are essential to generate undifferentiated quiescent satellite cells and to maintain satellite cell numbers. - Test2 - elhamkhani - TriplyDB

Hesr1 and Hesr3 are essential to generate undifferentiated quiescent satellite cells and to maintain satellite cell numbers.

Hesr1 and Hesr3 are essential to generate undifferentiated quiescent satellite cells and to maintain satellite cell numbers.

http://www.wikidata.org/entity/Q35693118

about

Muscle satellite cell heterogeneity and self-renewal A muscle stem cell for every muscle: variability of satellite cell biology among different muscle groups Lying low but ready for action: the quiescent muscle satellite cell Adult stem cell and mesenchymal progenitor theories of aging.Satellite cells: regenerative mechanisms and applicability in muscular dystrophy Intrinsic and extrinsic mechanisms regulating satellite cell function Target gene analysis by microarrays and chromatin immunoprecipitation identifies HEY proteins as highly redundant bHLH repressors Notch ligands regulate the muscle stem-like state ex vivo but are not sufficient for retaining regenerative capacity.Vitamin D deficiency down-regulates Notch pathway contributing to skeletal muscle atrophy in old wistar rats Muscle stem cells at a glance Hypoxia increases mouse satellite cell clone proliferation maintaining both in vitro and in vivo heterogeneity and myogenic potential.The role of Notch signaling in muscle progenitor cell depletion and the rapid onset of histopathology in muscular dystrophy.Switching roles: the functional plasticity of adult tissue stem cells.Hey bHLH Proteins Interact with a FBXO45 Containing SCF Ubiquitin Ligase Complex and Induce Its Translocation into the Nucleus Evidence of Notch-Hesr-Nrf2 Axis in Muscle Stem Cells, but Absence of Nrf2 Has No Effect on Their Quiescent and Undifferentiated State A Disintegrin and Metalloprotease 10 (ADAM10) Is Indispensable for Maintenance of the Muscle Satellite Cell Pool.Muscle Satellite Cell Protein Teneurin-4 Regulates Differentiation During Muscle Regeneration.Tissue-Specific Cultured Human Pericytes: Perivascular Cells from Smooth Muscle Tissue Have Restricted Mesodermal Differentiation Ability Notch Signaling Mediates Skeletal Muscle Atrophy in Cancer Cachexia Caused by Osteosarcoma.Notch signaling during cell fate determination in the inner ear.Isolation, characterization, and molecular regulation of muscle stem cells.Loss of niche-satellite cell interactions in syndecan-3 null mice alters muscle progenitor cell homeostasis improving muscle regeneration.Stage-specific effects of Notch activation during skeletal myogenesis.Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways.CCAAT/enhancer binding protein β is required for satellite cell self-renewal.Molecular Regulation of Muscle Satellite Cell Self-Renewal.Discovery of two GLP-1/Notch target genes that account for the role of GLP-1/Notch signaling in stem cell maintenance.Satellite Cells and Skeletal Muscle Regeneration.Loss of Ptpn11 (Shp2) drives satellite cells into quiescence Impaired fetal muscle development and JAK-STAT activation mark disease onset and progression in a mouse model for merosin-deficient congenital muscular dystrophy.Keep Your Friends Close: Cell-Cell Contact and Skeletal Myogenesis.Mimicking Muscle Stem Cell Quiescence in Culture: Methods for Synchronization in Reversible Arrest.KLF7 Regulates Satellite Cell Quiescence in Response to Extracellular Signaling.Reciprocal interaction between TRAF6 and notch signaling regulates adult myofiber regeneration upon injury Treadmill running induces satellite cell activation in diabetic mice.Constitutive Notch activation upregulates Pax7 and promotes the self-renewal of skeletal muscle satellite cells.Chemokine (C-X-C motif) ligand 1 is a myokine induced by palmitate and is required for myogenesis in mouse satellite cells.Molecular Regulation of Cellular Quiescence: A Perspective from Adult Stem Cells and Its Niches.BMP signaling regulates satellite cell-dependent postnatal muscle growth.Notch1 and Notch2 Coordinately Regulate Stem Cell Function in the Quiescent and Activated States of Muscle Satellite Cells.

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P2860

Hesr1 and Hesr3 are essential to generate undifferentiated quiescent satellite cells and to maintain satellite cell numbers.

http://www.wikidata.org/entity/Q35693118

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@ast

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@en

type

label

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@ast

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@en

prefLabel

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@ast

Hesr1 and Hesr3 are essential ...... intain satellite cell numbers.

@en

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P1476

Hesr1 and Hesr3 are essential ...... aintain satellite cell numbers

@en

P2093

Akiyoshi Uezumi

http://www.w3.org/2001/XMLSchema#string

Anna Zolkiewska

http://www.w3.org/2001/XMLSchema#string

Hiroki Kokubo

http://www.w3.org/2001/XMLSchema#string

Hiroshi Yamamoto

http://www.w3.org/2001/XMLSchema#string

Kazutake Tsujikawa

http://www.w3.org/2001/XMLSchema#string

Masahiko Yamaguchi

http://www.w3.org/2001/XMLSchema#string

Miroslav M Matev

http://www.w3.org/2001/XMLSchema#string

Norio Motohashi

http://www.w3.org/2001/XMLSchema#string

Randy L Johnson

http://www.w3.org/2001/XMLSchema#string

Ryo Ogawa

http://www.w3.org/2001/XMLSchema#string

P2860

Hey genes: a novel subfamily of hairy- and Enhancer of split related genes specifically expressed during mouse embryogenesis

Cellular and molecular regulation of muscle regeneration

Notch signaling: control of cell communication and cell fate

Hesr, a mediator of the Notch signaling, functions in heart and vessel development

Mouse hesr1 and hesr2 genes are redundantly required to mediate Notch signaling in the developing cardiovascular system

Combined loss of Hey1 and HeyL causes congenital heart defects because of impaired epithelial to mesenchymal transition

Premature myogenic differentiation and depletion of progenitor cells cause severe muscle hypotrophy in Delta1 mutants

The Notch target genes Hey1 and Hey2 are required for embryonic vascular development

HRT1, HRT2, and HRT3: a new subclass of bHLH transcription factors marking specific cardiac, somitic, and pharyngeal arch segments

Pax7 is required for the specification of myogenic satellite cells

P304

4609-4619

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

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10.1242/DEV.067165

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P407

P433

21

http://www.w3.org/2001/XMLSchema#string

P478

138

http://www.w3.org/2001/XMLSchema#string

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So-ichiro Fukada

P577

2011-11-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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51709492

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21989910

http://www.w3.org/2001/XMLSchema#string

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3265560

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