Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing. - Test2 - elhamkhani - TriplyDB

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

http://www.wikidata.org/entity/Q35747388

about

Genomic analysis and differential expression of HMG and S100A family in human arthritis: upregulated expression of chemokines, IL-8 and nitric oxide by HMGB1 Angiogenesis in bone regeneration.A mineral-rich extract from the red marine algae Lithothamnion calcareum preserves bone structure and function in female mice on a Western-style diet.Fractures in geriatric mice show decreased callus expansion and bone volume Substrate stiffness and oxygen as regulators of stem cell differentiation during skeletal tissue regeneration: a mechanobiological model.Disruption of thrombospondin-2 accelerates ischemic fracture healing.Mechanical stability affects angiogenesis during early fracture healing.The thrombospondins.Type III collagen modulates fracture callus bone formation and early remodeling.The convergence of fracture repair and stem cells: interplay of genes, aging, environmental factors and disease.Osteoblast response to ovariectomy is enhanced in intrinsically high aerobic-capacity rats.Two molecular weight species of thrombospondin-2 are present in bone and differentially modulated in fractured and nonfractured tibiae in a murine model of bone healing.Thrombospondin-2 deficiency in growing mice alters bone collagen ultrastructure and leads to a brittle bone phenotype.Use of micro-computed tomography to nondestructively characterize biomineral coatings on solid freeform fabricated poly (L-lactic acid) and poly ((ε-caprolactone) scaffolds in vitro and in vivo.Systemic inhibition of canonical Notch signaling results in sustained callus inflammation and alters multiple phases of fracture healing.THBS2 Is a Candidate Modifier of Liver Disease Severity in Alagille Syndrome Thrombospondin-2 and SPARC/osteonectin are critical regulators of bone remodeling.Cyp1B1 expression promotes angiogenesis by suppressing NF-κB activity.Fracture healing with alendronate treatment in the Brtl/+ mouse model of osteogenesis imperfecta Antiangiogenic and anticancer molecules in cartilage.Controlled release strategies for bone, cartilage, and osteochondral engineering--Part II: challenges on the evolution from single to multiple bioactive factor delivery.Thrombospondin-2 and extracellular matrix assembly.Biological perspectives of delayed fracture healing.Mechanical regulation of bone regeneration: theories, models, and experiments.Bone Marrow Aspirate Concentrate-Enhanced Marrow Stimulation of Chondral Defects.Stimulating Fracture Healing in Ischemic Environments: Does Oxygen Direct Stem Cell Fate during Fracture Healing?Notch signaling components are upregulated during both endochondral and intramembranous bone regeneration.Regionally Restricted Hox Function in Adult Bone Marrow Multipotent Mesenchymal Stem/Stromal Cells.The role of oxygen as a regulator of stem cell fate during fracture repair in TSP2-null mice.Improved Mobilization of Exogenous Mesenchymal Stem Cells to Bone for Fracture Healing and Sex Difference.Loss of Dnmt3b in Chondrocytes Leads to Delayed Endochondral Ossification and Fracture Repair.Thrombospondin-2 secreted by human umbilical cord blood-derived mesenchymal stem cells promotes chondrogenic differentiation.A computational model to explore the role of angiogenic impairment on endochondral ossification during fracture healing.Exendin-4, a glucagon-like peptide-1 receptor agonist, prevents osteopenia by promoting bone formation and suppressing bone resorption in aged ovariectomized rats.A Preliminary Study of Contrast-Enhanced Ultrasound (CEUS) and Cytokine Expression Analysis (CEA) as Early Predictors for the Outcome of Tibial Non-Union Therapy

P2860

Q28655057-139070BB-05E3-44EC-BFA2-FC70E5D2C54D Q30467055-2A74FF09-EAE6-4B1C-AF31-9E4CA8CD0C66 Q33881006-DD33D2F3-06A6-4F3C-AF9D-EAD05441A540 Q34276979-0ABD9543-E41A-4E91-816A-D17C90D751CD Q34387499-2C285572-2E8E-41FB-8869-C90F4E44E006 Q34529091-78C1262F-E77D-41CB-B63B-5226BF77B09C Q35197798-F7E7B085-DDB6-4B22-B6F0-3F78D0761C30 Q35230013-5295A0E0-2A61-41A2-BF76-D9FBEB24477C Q35528722-F5063145-A81A-4ADE-A948-0AFD150C2896 Q35683050-A6C0767E-1E35-4B32-B73C-60B6FA43199B Q35814776-E83D5590-E2CB-4223-A54E-A44A12F3BAE1 Q36031214-2309166A-B60E-47B0-B4D6-91E17FC46AC5 Q36174758-160B909A-D898-415C-8334-2CDA70999EDC Q36870384-0AADE3B9-FC3B-4485-9CF7-9BC9532EE4A9 Q36981367-EFF16DCA-C9CD-4D30-AFF5-018CA2AB8B11 Q37297770-3D9A00D0-93B7-4CB1-AA6B-4FAB8CF965A8 Q37424213-D1CB4F0D-7DD4-4FAE-A08B-5CFBE28F8442 Q37439850-144ADD8B-7EB7-476D-90E4-06C2DCE58506 Q37722288-FB9F9696-2A95-490D-9DF0-72E4637D0DDB Q38007565-136FCD57-CE59-4E5A-BD91-A171516B2BAA Q38068244-B4E728FE-543A-41DF-ADDD-A468E2CA7BEA Q38179711-30DDCB80-0955-459B-8170-3CEDFFAC98B6 Q38214253-A7E1F219-4D0C-4E0A-BD9B-99B854E2FEE6 Q38300586-75970E52-46DE-4949-A1B7-49680DDFE53C Q38728665-42DB6A7B-DEB9-466E-84E3-69B00BACF3F0 Q38775706-7D96E471-D71D-4EDD-A11B-0676F96B72B5 Q38985595-775A71E7-DBA3-4D87-ADBA-B89C600C8A9B Q39112663-AB5297AD-7DB8-487C-AB40-835ABE4C29A8 Q46865174-B63DECB5-C725-43B6-B19A-B57B1E280292 Q46898799-CDA6D63C-41FF-4DEC-AD71-698C553EE407 Q47662635-D2EFF800-8ABE-4173-9959-4621F5BCDEED Q48029947-90531FE7-F2B3-4D42-B841-3C9D83840E4E Q50724345-EAE2A5B2-42C5-43A8-8BDE-606A58D53546 Q51272232-186A1B89-454B-4091-9CDB-B403AA137D97 Q58703967-3EEFBD8B-7E61-46C7-A1F8-170714C744DC

P2860

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

http://www.wikidata.org/entity/Q35747388

description

2009 nî lūn-bûn

@nan

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

2009年论文

@zh

2009年论文

@zh-cn

name

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@ast

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@en

type

label

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@ast

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@en

prefLabel

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@ast

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing.

@en

P1433

Q35747388-34ADBE65-2D2F-486D-827B-74D3600D3285

P1476

Q35747388-1327081D-A1E8-42C3-94AC-963101A4B219

P2093

Q35747388-0D39C4DD-F8E6-4DB2-89F5-1CD185B1A7AB

Q35747388-0F2F63E8-893B-4BE8-A737-59ECFDF28A0A

Q35747388-51E3F061-04C9-4A3C-B01C-DB705C4D27B4

Q35747388-79D688A2-2B47-4FA5-A2CC-D7094A939E6D

Q35747388-8A6E1051-92D2-4CE4-850A-EABE72CF7F3E

Q35747388-C0BA82C0-92B0-4A5C-A4D1-A8A3B6F86219

Q35747388-F7C3C256-7D72-425A-85E3-C9F341BD1D57

P2860

Q35747388-00AAC709-3E0F-47F2-B783-A42F1B2802EF

Q35747388-0491006E-8043-4363-836A-BA1991519AA9

Q35747388-145988C8-A81E-49C7-976F-7237D373FE05

Q35747388-14BCA177-1923-42D9-AF57-8E2769E74A9E

Q35747388-15673679-9050-4D89-8EAF-7067198B94F7

Q35747388-206D1319-CE7A-4B3F-9DCB-4745FD023841

Q35747388-262BB818-00A7-440E-B83A-38029677CFBC

Q35747388-2FC23F22-3038-4296-AF8E-5159C8F8BE49

Q35747388-3746AF8C-C353-4CAE-9025-60E244413127

Q35747388-3A3410D9-2731-4D27-8DA6-A56D4D851B73

P304

Q35747388-F83778A2-9EB5-4D1C-9841-7D514C23F49C

P31

Q35747388-F742465E-C439-4D12-A849-26595EAAEEB7

P356

Q35747388-AF6E9F1B-E75B-429F-80F6-C74547C159E8

P433

Q35747388-09FC1B2E-6122-4E8B-83B6-441B837B5A86

P478

Q35747388-3D245A70-F11D-4EC4-A43C-B7EF31A85E42

P50

Q35747388-87D5F74A-33C1-47ED-AA76-72E465D09A32

P577

Q35747388-1A71391A-E7EA-4612-9E8B-5B20E6C670C6

P5875

Q35747388-BD648DDF-5D92-4DDD-9609-3B1D5859C4E8

P698

Q35747388-B1E5A11D-2429-4F71-BC0B-A75240EC0E84

P932

Q35747388-2170402E-40BE-440B-9418-1F6D3266AD53

P356

P1433

Journal of Bone and Mineral Research

P1476

Thrombospondin-2 influences the proportion of cartilage and bone during fracture healing

@en

P2093

Amish Naik

http://www.w3.org/2001/XMLSchema#string

Douglas K Taylor

http://www.w3.org/2001/XMLSchema#string

Jeffrey A Meganck

http://www.w3.org/2001/XMLSchema#string

Rajiv Rajani

http://www.w3.org/2001/XMLSchema#string

Regis J O'Keefe

http://www.w3.org/2001/XMLSchema#string

Shawn Terkhorn

http://www.w3.org/2001/XMLSchema#string

Steven A Goldstein

http://www.w3.org/2001/XMLSchema#string

P2860

Thrombospondin 2 inhibits microvascular endothelial cell proliferation by a caspase-independent mechanism.

Thrombospondins use the VLDL receptor and a nonapoptotic pathway to inhibit cell division in microvascular endothelial cells

Mice that lack thrombospondin 2 display connective tissue abnormalities that are associated with disordered collagen fibrillogenesis, an increased vascular density, and a bleeding diathesis

Accelerated up-regulation of L-Sox5, Sox6, and Sox9 by BMP-2 gene transfer during murine fracture healing

Megakaryocytes require thrombospondin-2 for normal platelet formation and function

Hypoxia and HIF-1 alpha in chondrogenesis

Hypoxia in cartilage: HIF-1alpha is essential for chondrocyte growth arrest and survival

A second, expressed thrombospondin gene (Thbs2) exists in the mouse genome.

The functions of thrombospondin-1 and-2.

Vascular endothelial growth factor stimulates bone repair by promoting angiogenesis and bone turnover.

P304

1043-1054

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1359/JBMR.090101

http://www.w3.org/2001/XMLSchema#string

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

24

http://www.w3.org/2001/XMLSchema#string

P50

Kurt D. Hankenson

P577

2009-06-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

23758958

http://www.w3.org/2001/XMLSchema#string

P698

19123916

http://www.w3.org/2001/XMLSchema#string

P932

3276350

http://www.w3.org/2001/XMLSchema#string