Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
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Bone loss. Therapeutic approaches for preventing bone loss in inflammatory arthritisAnti-TNF-alpha therapy as a clinical intervention for periprosthetic osteolysis.In vivo RANK signaling blockade using the receptor activator of NF-kappaB:Fc effectively prevents and ameliorates wear debris-induced osteolysis via osteoclast depletion without inhibiting osteogenesisIn vivo imaging of particle-induced inflammation and osteolysis in the calvariae of NFκB/luciferase transgenic micePrevention of wear particle-induced osteolysis by a novel V-ATPase inhibitor saliphenylhalamide through inhibition of osteoclast bone resorptionPyrroloquinoline quinine inhibits RANKL-mediated expression of NFATc1 in part via suppression of c-Fos in mouse bone marrow cells and inhibits wear particle-induced osteolysis in miceIn vitro reactivity to implant metals demonstrates a person-dependent association with both T-cell and B-cell activation.Direct bone formation during distraction osteogenesis does not require TNFalpha receptors and elevated serum TNFalpha fails to inhibit bone formation in TNFR1 deficient mice.Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interactionSurveillance of systemic trafficking of macrophages induced by UHMWPE particles in nude mice by noninvasive imagingMechanisms balancing skeletal matrix synthesis and degradation.Involvement of T-lymphocytes in periodontal disease and in direct and indirect induction of bone resorption.Nanostructured diamond coatings for orthopaedic applicationsThe role of osteoclast differentiation in aseptic loosening.Interaction of Materials and Biology in Total Joint Replacement - Successes, Challenges and Future Directions.Cobalt-alloy implant debris induce HIF-1α hypoxia associated responses: a mechanism for metal-specific orthopedic implant failure.Osteoclasts; culprits in inflammatory osteolysis.Independent and cooperative roles of tumor necrosis factor-alpha, nuclear factor-kappaB, and bone morphogenetic protein-2 in regulation of metastasis and osteomimicry of prostate cancer cells and differentiation and mineralization of MC3T3-E1 osteobA TNF variant that associates with susceptibility to musculoskeletal disease modulates thyroid hormone receptor binding to control promoter activationHO-1 Induced by Cilostazol Protects Against TNF-α-associated Cytotoxicity via a PPAR-γ-dependent Pathway in Human Endothelial CellsThe roles of lipid oxidation products and receptor activator of nuclear factor-κB signaling in atherosclerotic calcificationThe aetiology of otosclerosis: a review of the literature.Restoration of regenerative osteoblastogenesis in aged mice: modulation of TNF.Inhibitory effect of (-)-epigallocatechin gallate on titanium particle-induced TNF-α release and in vivo osteolysis.Biological response to prosthetic debris.Actin and ERK1/2-CEBPβ signaling mediates phagocytosis-induced innate immune response of osteoprogenitor cells.Aseptic loosening.In vitro studies on the effect of particle size on macrophage responses to nanodiamond wear debris.Does endotoxin contribute to aseptic loosening of orthopedic implants?Comparison of the roles of IL-1, IL-6, and TNFalpha in cell culture and murine models of aseptic loosening.Cobalt Alloy Implant Debris Induces Inflammation and Bone Loss Primarily through Danger Signaling, Not TLR4 Activation: Implications for DAMP-ening Implant Related Inflammation.Lentivirus-mediated short hairpin RNA interference targeting TNF-alpha in macrophages inhibits particle-induced inflammation and osteolysis in vitro and in vivoSurgical Modification of the Murine Calvaria Osteolysis ModelMechanisms of bone loss in rheumatoid arthritis.Aggravation of inflammatory response by costimulation with titanium particles and mechanical perturbations in osteoblast- and macrophage-like cells.Icariin attenuates titanium-particle inhibition of bone formation by activating the Wnt/β-catenin signaling pathway in vivo and in vitro.Anti-oxidation treatment of ultra high molecular weight polyethylene components to decrease periprosthetic osteolysis: evaluation of osteolytic and osteogenic properties of wear debris particles in a murine calvaria model.The central role of wear debris in periprosthetic osteolysis.Development of polymeric nanocarrier system for early detection and targeted therapeutic treatment of peri-implant osteolysis.Direct subcutaneous injection of polyethylene particles over the murine calvaria results in dramatic osteolysis
P2860
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P2860
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@ast
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@en
type
label
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@ast
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@en
prefLabel
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@ast
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.
@en
P2093
P2860
P1476
Tumor necrosis factor-alpha mediates orthopedic implant osteolysis
@en
P2093
J M Erdmann
K D Merkel
K P McHugh
Y Abu-Amer
P2860
P304
P356
10.1016/S0002-9440(10)65266-2
P407
P577
1999-01-01T00:00:00Z