Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
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Transcriptomic and proteomic analyses of rhabdomyosarcoma cells reveal differential cellular gene expression in response to enterovirus 71 infectionPersistent expression of chemokine and chemokine receptor RNAs at primary and latent sites of herpes simplex virus 1 infectionNeuroinvasion and Inflammation in Viral Central Nervous System InfectionsNanoparticles as potential new generation broad spectrum antimicrobial agentsNumerous conserved and divergent microRNAs expressed by herpes simplex viruses 1 and 2.A limited innate immune response is induced by a replication-defective herpes simplex virus vector following delivery to the murine central nervous systemA common neuronal response to alphaherpesvirus infection.Virus and cell RNAs expressed during Epstein-Barr virus replicationPersistent infection by HSV-1 is associated with changes in functional architecture of iPSC-derived neurons and brain activation patterns underlying working memory performanceFunctional genomic analysis of herpes simplex virus type 1 counteraction of the host innate response.Chronic progressive deficits in neuron size, density and number in the trigeminal ganglia of mice latently infected with herpes simplex virus.Diffusion tensor magnetic resonance imaging of trigeminal nerves in relapsing herpetic keratouveitis.Insights into host responses against pathogens from transcriptional profiling.Herpes simplex virus 1 microRNAs expressed abundantly during latent infection are not essential for latency in mouse trigeminal gangliaA Herpesviral Lytic Protein Regulates the Structure of Latent Viral Chromatin.Host cell gene expression during human immunodeficiency virus type 1 latency and reactivation and effects of targeting genes that are differentially expressed in viral latency.Molecular analysis of human cancer cells infected by an oncolytic HSV-1 reveals multiple upregulated cellular genes and a role for SOCS1 in virus replication.The nucleocapsid protein of severe acute respiratory syndrome-coronavirus inhibits the activity of cyclin-cyclin-dependent kinase complex and blocks S phase progression in mammalian cells.Herpes simplex virus type-1 latency-associated transcript-induced immunoreactivity of substance P in trigeminal neurons is reversed by bone morphogenetic protein-7.Allelic association of sequence variants in the herpes virus entry mediator-B gene (PVRL2) with the severity of multiple sclerosis.Herpes Simplex Virus Type 1 Infects Enteric Neurons and Triggers Gut Dysfunction via Macrophage Recruitment.
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P2860
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
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2003年论文
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name
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@ast
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@en
type
label
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@ast
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@en
prefLabel
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@ast
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@en
P2093
P2860
P1433
P1476
Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.
@en
P2093
David M Knipe
Donald M Coen
Holly Holman
Martha F Kramer
W James Cook
P2860
P304
P356
10.1128/JVI.77.17.9533-9541.2003
P407
P577
2003-09-01T00:00:00Z