E-cadherin expression in melanoma cells restores keratinocyte-mediated growth control and down-regulates expression of invasion-related adhesion receptors.
about
Melanoma: from melanocyte to genetic alterations and clinical optionsDiscovery of a selective inhibitor of oncogenic B-Raf kinase with potent antimelanoma activitySmall dsRNAs induce transcriptional activation in human cellsMyosins as fundamental components during tumorigenesis: diverse and indispensablePushing tumor cells towards a malignant phenotype: stimuli from the microenvironment, intercellular communications and alternative roadsLgl2 executes its function as a tumor suppressor by regulating ErbB signaling in the zebrafish epidermisCancer metastases: challenges and opportunitiesDownregulation of E-cadherin and Desmoglein 1 by autocrine hepatocyte growth factor during melanoma developmentCapturing the biological impact of CDKN2A and MC1R genes as an early predisposing event in melanoma and non melanoma skin cancercis Interaction of the cell adhesion molecule CEACAM1 with integrin beta(3)Evaluation of molecular markers of mesenchymal phenotype in melanoma.Aggressive melanoma cells escape from BMP7-mediated autocrine growth inhibition through coordinated Noggin upregulation.Multiple types of data are required to identify the mechanisms influencing the spatial expansion of melanoma cell colonies.Genetic factors in metastatic progression of cutaneous melanoma: the future role of circulating melanoma cells in prognosis and management.Subtypes of melanocytes and melanoma cells distinguished by their intercellular contacts: heterotypic adherens junctions, adhesive associations, and dispersed desmoglein 2 glycoproteins.Precursor N-cadherin mediates glial cell line-derived neurotrophic factor-promoted human malignant glioma.Dormancy of metastatic melanomaDown-regulation of TWIST decreases migration and invasion of laryngeal carcinoma Hep-2 cells by regulating the E-cadherin, N-cadherin expression.The epithelial-mesenchymal transition (EMT) regulatory factor SLUG (SNAI2) is a downstream target of SPARC and AKT in promoting melanoma cell invasionCadherins in neural crest cell development and transformation.Surface expression of precursor N-cadherin promotes tumor cell invasionAdhesion, migration and communication in melanocytes and melanoma.miR-200c inhibits invasion, migration and proliferation of bladder cancer cells through down-regulation of BMI-1 and E2F3Artificial skin in perspective: concepts and applications.Dynamics of cell interactions and communications during melanoma development.Melanocyte transformation associated with substrate adhesion impediment.Up-regulated expression of zonula occludens protein-1 in human melanoma associates with N-cadherin and contributes to invasion and adhesion.Desmoglein 2 depletion leads to increased migration and upregulation of the chemoattractant secretoneurin in melanoma cells.Generation of a tumor spheroid in a microgravity environment as a 3D model of melanoma.Melanoma: Molecular Pathogenesis and Therapeutic ManagementFunction and regulation of melanoma-stromal fibroblast interactions: when seeds meet soil.The basic biology of malignant melanoma: molecular mechanisms of disease progression and comparative aspects.Dysregulation of claudin-7 leads to loss of E-cadherin expression and the increased invasion of esophageal squamous cell carcinoma cellsPrognostic significance of cadherin-based adhesion molecules in cutaneous malignant melanomaExpression of connexin 43 and E-cadherin in choroidal melanoma.EMT-associated factors promote invasive properties of uveal melanoma cellsCCN3 controls 3D spatial localization of melanocytes in the human skin through DDR1.Protein kinase C inhibitor Gö6976 but not Gö6983 induces the reversion of E- to N-cadherin switch and metastatic phenotype in melanoma: identification of the role of protein kinase D1BRAF Inhibition Generates a Host-Tumor Niche that Mediates Therapeutic Escape.The use of an in vitro 3D melanoma model to predict in vivo plasmid transfection using electroporation.
P2860
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P2860
E-cadherin expression in melanoma cells restores keratinocyte-mediated growth control and down-regulates expression of invasion-related adhesion receptors.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
E-cadherin expression in melan ...... on-related adhesion receptors.
@ast
E-cadherin expression in melan ...... on-related adhesion receptors.
@en
type
label
E-cadherin expression in melan ...... on-related adhesion receptors.
@ast
E-cadherin expression in melan ...... on-related adhesion receptors.
@en
prefLabel
E-cadherin expression in melan ...... on-related adhesion receptors.
@ast
E-cadherin expression in melan ...... on-related adhesion receptors.
@en
P2093
P2860
P1476
E-cadherin expression in melan ...... on-related adhesion receptors.
@en
P2093
P Van Belle
P2860
P304
P356
10.1016/S0002-9440(10)65023-7
P407
P577
2000-05-01T00:00:00Z