A switch region determines the cell type-specific positive or negative action of YY1 on the activity of the human papillomavirus type 18 promoter.
about
YY1-binding sites provide central switch functions in the PARP-1 gene expression networkDifferential expression of the human ST5 gene in HeLa-fibroblast hybrid cell lines mediated by YY1: evidence that YY1 plays a part in tumor suppressionRepression of human immunodeficiency virus type 1 through the novel cooperation of human factors YY1 and LSFThe ability of POU family transcription factors to activate or repress gene expression is dependent on the spacing and context of their specific response elementsAdenovirus E1A243 disrupts the ATF/CREB-YY1 complex at the mouse c-fos promoter.Induction of p53, p21 and apoptosis by silencing the NF90/NF45 complex in human papilloma virus-transformed cervical carcinoma cells.Silencing of integrated human papillomavirus type 18 oncogene transcription in cells expressing SerpinB2.Variant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Novel binding sites for regulatory factors in the human papillomavirus type 18 enhancer and promoter identified by in vivo footprintingOverexpression of C/EBPbeta represses human papillomavirus type 18 upstream regulatory region activity in HeLa cells by interfering with the binding of TATA-binding protein.Genetic analysis of cis regulatory elements within the 5' region of the human papillomavirus type 31 upstream regulatory region during different stages of the viral life cycleYY1 represses human papillomavirus type 16 transcription by quenching AP-1 activity.A novel C/EBP beta-YY1 complex controls the cell-type-specific activity of the human papillomavirus type 18 upstream regulatory region.CCAAT displacement protein, a regulator of differentiation-specific gene expression, binds a negative regulatory element within the 5' end of the human papillomavirus type 6 long control region.The cellular YY1 transcription factor binds a cis-acting, negatively regulating element in the Epstein-Barr virus BRLF1 promoter.YY1 is a novel potential therapeutic target for the treatment of HPV infection-induced cervical cancer by arsenic trioxide.Genetic and biochemical analysis of cis regulatory elements within the keratinocyte enhancer region of the human papillomavirus type 31 upstream regulatory region during different stages of the viral life cycle.Transcription factor YY1 represses cell-free replication from human papillomavirus origins.The transcriptional activation and repression domains of RFX1, a context-dependent regulator, can mutually neutralize their activities.Cell-type-specific separate regulation of the E6 and E7 promoters of human papillomavirus type 6a by the viral transcription factor E2YY1 and NF1 both activate the human p53 promoter by alternatively binding to a composite element, and YY1 and E1A cooperate to amplify p53 promoter activityMultiple mechanisms of transcriptional repression by YY1.
P2860
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P2860
A switch region determines the cell type-specific positive or negative action of YY1 on the activity of the human papillomavirus type 18 promoter.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
A switch region determines the ...... pillomavirus type 18 promoter.
@ast
A switch region determines the ...... pillomavirus type 18 promoter.
@en
type
label
A switch region determines the ...... pillomavirus type 18 promoter.
@ast
A switch region determines the ...... pillomavirus type 18 promoter.
@en
prefLabel
A switch region determines the ...... pillomavirus type 18 promoter.
@ast
A switch region determines the ...... pillomavirus type 18 promoter.
@en
P2093
P2860
P1433
P1476
A switch region determines the ...... pillomavirus type 18 promoter.
@en
P2093
H Zur Hausen
T Bauknecht
P2860
P407
P577
1995-01-01T00:00:00Z