Gestational bisphenol A exposure and testis development.
about
Environmental factors, epigenetics, and developmental origin of reproductive disordersBisphenol A induces DSB-ATM-p53 signaling leading to cell cycle arrest, senescence, autophagy, stress response, and estrogen release in human fetal lung fibroblasts.DNA methylation and transcriptome aberrations mediated by ERα in mouse seminal vesicles following developmental DES exposure.
P2860
Gestational bisphenol A exposure and testis development.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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2014年论文
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2014年论文
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name
Gestational bisphenol A exposure and testis development.
@ast
Gestational bisphenol A exposure and testis development.
@en
type
label
Gestational bisphenol A exposure and testis development.
@ast
Gestational bisphenol A exposure and testis development.
@en
prefLabel
Gestational bisphenol A exposure and testis development.
@ast
Gestational bisphenol A exposure and testis development.
@en
P2860
P356
P1476
Gestational bisphenol A exposure and testis development
@en
P2093
Cory Teuscher
Maria Bondesson
P2860
P356
10.4161/ENDO.29088
P577
2014-01-01T00:00:00Z