The predominant virus antigen burden is present in macrophages in Theiler's murine encephalomyelitis virus-induced demyelinating disease.
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Leader (L) and L* proteins of Theiler's murine encephalomyelitis virus (TMEV) and their regulation of the virus' biological activitiesThe role of infections in autoimmune diseaseTheiler's virus infection: a model for multiple sclerosisInflammatory monocytes and the pathogenesis of viral encephalitisPro-inflammatory functions of astrocytes correlate with viral clearance and strain-dependent protection from TMEV-induced demyelinating diseaseInfluence of the Theiler's virus L* protein on macrophage infection, viral persistence, and neurovirulence.An attenuated variant of the GDVII strain of Theiler's virus does not persist and does not infect the white matter of the central nervous systemAnti-CCL2 treatment inhibits Theiler's murine encephalomyelitis virus-induced demyelinating diseaseHLA-DQ polymorphism influences progression of demyelination and neurologic deficits in a viral model of multiple sclerosisDirect comparison of demyelinating disease induced by the Daniel's strain and BeAn strain of Theiler's murine encephalomyelitis virus.Three immune-mediated disease models induced by Theiler's virus: Multiple sclerosis, seizures and myocarditis.Theiler's virus infection of perforin-deficient mice.The neurovirulence of the DA and GDVII strains of Theiler's virus correlates with their ability To infect cultured neuronsL* protein of the DA strain of Theiler's murine encephalomyelitis virus is important for virus growth in a murine macrophage-like cell line.Characterization of and functional antigen presentation by central nervous system mononuclear cells from mice infected with Theiler's murine encephalomyelitis virusA protein critical for a Theiler's virus-induced immune system-mediated demyelinating disease has a cell type-specific antiapoptotic effect and a key role in virus persistence.L* protein of Theiler's murine encephalomyelitis virus is required for virus growth in a murine macrophage-like cell line.The GDVII strain of Theiler's virus spreads via axonal transport.Depletion of blood-borne macrophages does not reduce demyelination in mice infected with a neurotropic coronavirus.Macrophage infiltration, but not apoptosis, is correlated with immune-mediated demyelination following murine infection with a neurotropic coronavirus.Direct activation of innate and antigen-presenting functions of microglia following infection with Theiler's virus.High numbers of viral RNA copies in the central nervous system of mice during persistent infection with Theiler's virus.Endogenous presentation of self myelin epitopes by CNS-resident APCs in Theiler's virus-infected miceCentral neuroinvasion and demyelination by inflammatory macrophages after peripheral virus infection is controlled by SHP-1.Heterogeneity in oligodendroglia: Is it relevant to mouse models and human disease?Theiler's virus infection: Pathophysiology of demyelination and neurodegeneration.Non-AUG-initiated internal translation of the L* protein of Theiler's virus and importance of this protein for viral persistenceTheiler's virus infection of primary cultures of bone marrow-derived monocytes/macrophagesCD154 blockade results in transient reduction in Theiler's murine encephalomyelitis virus-induced demyelinating disease.Theiler's virus L protein is targeted to the mitochondrial outer membrane.Evasion of antiviral innate immunity by Theiler's virus L* protein through direct inhibition of RNase LIdentification of a novel neuropathogenic Theiler's murine encephalomyelitis virus.Infectious RNA isolated from the spinal cords of mice chronically infected with Theiler's murine encephalomyelitis virus.The antiapoptotic protein Mcl-1 controls the type of cell death in Theiler's virus-infected BHK-21 cells.Theiler's murine encephalomyelitis virus kills restrictive but not permissive cells by apoptosis.Role of VP2 amino acid 141 in tropism of Theiler's virus within the central nervous system.Theiler's murine encephalomyelitis virus subgroup strain-specific infection in a murine macrophage-like cell line.Role of macrophages during Theiler's virus infection.The immune system preferentially clears Theiler's virus from the gray matter of the central nervous system.Infection of macrophages by Theiler's murine encephalomyelitis virus is highly dependent on their activation or differentiation state.
P2860
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P2860
The predominant virus antigen burden is present in macrophages in Theiler's murine encephalomyelitis virus-induced demyelinating disease.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
The predominant virus antigen ...... induced demyelinating disease.
@ast
The predominant virus antigen ...... induced demyelinating disease.
@en
type
label
The predominant virus antigen ...... induced demyelinating disease.
@ast
The predominant virus antigen ...... induced demyelinating disease.
@en
prefLabel
The predominant virus antigen ...... induced demyelinating disease.
@ast
The predominant virus antigen ...... induced demyelinating disease.
@en
P2093
P2860
P1433
P1476
The predominant virus antigen ...... -induced demyelinating disease
@en
P2093
H L Lipton
M L Jelachich
P2860
P304
P407
P577
1995-04-01T00:00:00Z