Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation. - Test2 - elhamkhani - TriplyDB

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

http://www.wikidata.org/entity/Q35842036

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Cell Type-Specific Regulation of Immunological Synapse Dynamics by B7 Ligand Recognition Sequential administration of nivolumab and ipilimumab with a planned switch in patients with advanced melanoma (CheckMate 064): an open-label, randomised, phase 2 trial.CD86 is an activation receptor for NK cell cytotoxicity against tumor cells Pyroptosis and adaptive immunity mechanisms are promptly engendered in mesenteric lymph-nodes during pig infections with Salmonella enterica serovar Typhimurium.Atypical Manifestation of LPS-Responsive Beige-Like Anchor Deficiency Syndrome as an Autoimmune Endocrine Disorder without Enteropathy and Immunodeficiency Comparison of the intracellular trafficking itinerary of ctla-4 orthologues Vitamin D Antagonises the Suppressive Effect of Inflammatory Cytokines on CTLA-4 Expression and Regulatory Function.A transendocytosis model of CTLA-4 function predicts its suppressive behavior on regulatory T cells.One, two or three? Probing the stoichiometry of membrane proteins by single-molecule localization microscopy Coinhibitory Pathways in the B7-CD28 Ligand-Receptor Family.Co-targeting EGFR and survivin with a bivalent aptamer-dual siRNA chimera effectively suppresses prostate cancer Allogeneic T cell responses are regulated by a specific miRNA-mRNA network.Compartmentalization of signaling by vesicular trafficking: a shared building design for the immune synapse and the primary cilium.Understanding the CD28/CTLA-4 (CD152) pathway and its implications for costimulatory blockade.Confusing signals: recent progress in CTLA-4 biology Genomics of Immune Diseases and New Therapies.Identifying functional defects in patients with immune dysregulation due to LRBA and CTLA-4 mutations.CHAI and LATAIE: new genetic diseases of CTLA-4 checkpoint insufficiency.Immune deficiency and autoimmunity in patients with CTLA-4 (CD152) mutations.Glutamate Receptor Interacting Protein 1 Regulates CD4(+) CTLA-4 Expression and Transplant Rejection.Treatment of Infantile Inflammatory Bowel Disease and Autoimmunity by Allogeneic Stem Cell Transplantation in LPS-Responsive Beige-Like Anchor Deficiency.PAG/Cbp suppression reveals a contribution of CTLA-4 to setting the activation threshold in T cells CD28 costimulation: walking the immunological tightrope.Role of the EHD Family of Endocytic Recycling Regulators for TCR Recycling and T Cell Function.Myeloma skews regulatory T and pro-inflammatory T helper 17 cell balance in favor of a suppressive state.Prospects for chimeric antigen receptor-modified T cell therapy for solid tumors.Checkpoint Inhibitors: Applications for Autoimmunity.Interferon-γ Signaling in Melanocytes and Melanoma Cells Regulates Expression of CTLA-4.IMMUNOLOGY. Moving CTLA-4 from the trash to recycling.Characterization of CTLA4 Trafficking and Implications for Its Function

P2860

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P2860

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

http://www.wikidata.org/entity/Q35842036

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

@zh

2012年论文

@zh-cn

name

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@ast

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@en

type

label

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@ast

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@en

prefLabel

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@ast

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@en

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JBC.M111.304329

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Journal of Biological Chemistry

P1476

Constitutive clathrin-mediated endocytosis of CTLA-4 persists during T cell activation.

@en

P2093

Anna K Willox

http://www.w3.org/2001/XMLSchema#string

Joshua Z Rappoport

http://www.w3.org/2001/XMLSchema#string

Louisa E Jeffery

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Natalie S Poulter

http://www.w3.org/2001/XMLSchema#string

Omar S Qureshi

http://www.w3.org/2001/XMLSchema#string

Rupert Kenefeck

http://www.w3.org/2001/XMLSchema#string

Tie Zheng Hou

http://www.w3.org/2001/XMLSchema#string

Zoe Briggs

http://www.w3.org/2001/XMLSchema#string

P2860

Exocytosis of CTLA-4 is dependent on phospholipase D and ADP ribosylation factor-1 and stimulated during activation of regulatory T cells

Interaction of the cytoplasmic tail of CTLA-4 (CD152) with a clathrin-associated protein is negatively regulated by tyrosine phosphorylation

Study of the interaction of the medium chain mu 2 subunit of the clathrin-associated adapter protein complex 2 with cytotoxic T-lymphocyte antigen 4 and CD28

Trans-endocytosis of CD80 and CD86: a molecular basis for the cell-extrinsic function of CTLA-4

Interaction of CTLA-4 with the clathrin-associated protein AP50 results in ligand-independent endocytosis that limits cell surface expression

Lymphoproliferative disorders with early lethality in mice deficient in Ctla-4

Mechanisms of endocytosis

Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4

Dynasore, a cell-permeable inhibitor of dynamin

A screen for endocytic motifs.

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9429-9440

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10.1074/JBC.M111.304329

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12

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287

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Stephen J. Royle

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2012-01-19T00:00:00Z

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221764347

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22262842

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3308817

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