p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
about
Generation of membrane structures during phagocytosis and chemotaxis of macrophages: role and regulation of the actin cytoskeletonPhosphoinositides: tiny lipids with giant impact on cell regulationHyperactivation of p21ras and PI3K cooperate to alter murine and human neurofibromatosis type 1-haploinsufficient osteoclast functionsCommon genetic denominators for Ca++-based skeleton in Metazoa: role of osteoclast-stimulating factor and of carbonic anhydrase in a calcareous spongeMacrophage motility is driven by frontal-towing with a force magnitude dependent on substrate stiffnessCSF-1 receptor signaling in myeloid cellsGenetic and cellular evidence of vascular inflammation in neurofibromin-deficient mice and humans.ROCK1 functions as a suppressor of inflammatory cell migration by regulating PTEN phosphorylation and stability.Genetic analysis of the role of the PI3K-Akt pathway in lipopolysaccharide-induced cytokine and tissue factor gene expression in monocytes/macrophages.IQGAP1 is involved in post-ischemic neovascularization by regulating angiogenesis and macrophage infiltrationInnate immune regulation by STAT-mediated transcriptional mechanisms.Nf1-/- Schwann cell-conditioned medium modulates mast cell degranulation by c-Kit-mediated hyperactivation of phosphatidylinositol 3-kinase.Essential role for focal adhesion kinase in regulating stress hematopoiesis.The phosphatidylinositol-3-kinase inhibitor PX-866 overcomes resistance to the epidermal growth factor receptor inhibitor gefitinib in A-549 human non-small cell lung cancer xenograftsErk1 positively regulates osteoclast differentiation and bone resorptive activityalphavbeta3 and macrophage colony-stimulating factor: partners in osteoclast biology.Alveolar epithelial cells are critical in protection of the respiratory tract by secretion of factors able to modulate the activity of pulmonary macrophages and directly control bacterial growth.PI3K/Akt activation is critical for early hepatic regeneration after partial hepatectomyThe p85alpha subunit of class IA phosphatidylinositol 3-kinase regulates the expression of multiple genes involved in osteoclast maturation and migration.ROCK1 via LIM kinase regulates growth, maturation and actin based functions in mast cells.An arrestin-dependent multi-kinase signaling complex mediates MIP-1beta/CCL4 signaling and chemotaxis of primary human macrophages.FAK and p38-MAP kinase-dependent activation of apoptosis and caspase-3 in retinal endothelial cells by alpha1(IV)NC1Molecular mechanisms of liver regeneration and protection for treatment of liver dysfunction and diseases.Two-dimensional motility of a macrophage cell line on microcontact-printed fibronectin.rna interference targeting p110β reduces tumor necrosis factor-alpha production in cellular response to wear particles in vitro and osteolysis in vivo.Inhibition of the PI3K/AKT pathway reduces tumor necrosis factor-alpha production in the cellular response to wear particles in vitro.Essential roles of PIKfyve and PTEN on phagosomal phosphatidylinositol 3-phosphate dynamics.Phosphoinositide 3-kinase signalling regulates early development and developmental haemopoiesis.PI3K p85α Subunit-deficient Macrophages Protect Mice from Acute Colitis due to the Enhancement of IL-10 Production.Genetic reduction of class IA PI-3 kinase activity alters fetal hematopoiesis and competitive repopulating ability of hematopoietic stem cells in vivo.Impaired function of Fanconi anemia type C-deficient macrophages.Dendritic cell maturation occurs through the inhibition of GSK-3β.Metabolic Reprogramming Mediated by the mTORC2-IRF4 Signaling Axis Is Essential for Macrophage Alternative Activation.Inhibitory receptor FcγRIIb mediates the effects of IgG on a phagosome acidification and a sequential dephosphorylation system comprising SHIPs and Inpp4a.Cytokine mediators of chronic graft-versus-host disease.Suppression of the PI3K subunit p85α delays embryoid body development and inhibits cell adhesion.M-CSF Regulates the Cytoskeleton via Recruitment of a Multimeric Signaling Complex to c-Fms Tyr-559/697/721
P2860
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P2860
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@ast
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@en
type
label
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@ast
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@en
prefLabel
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@ast
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@en
P2093
P2860
P1433
P1476
p85alpha subunit of class IA PI-3 kinase is crucial for macrophage growth and migration.
@en
P2093
David A Ingram
Jovencio Borneo
Reuben Kapur
Veerendra Munugalavadla
P2860
P304
P356
10.1182/BLOOD-2004-10-4041
P407
P577
2005-03-15T00:00:00Z