5-HT2A receptor activation is necessary for CO2-induced arousal. - Test2 - elhamkhani - TriplyDB

5-HT2A receptor activation is necessary for CO2-induced arousal.

5-HT2A receptor activation is necessary for CO2-induced arousal.

http://www.wikidata.org/entity/Q35867477

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Sudden unexpected death in epilepsy: Identifying risk and preventing mortality 2014 Epilepsy Benchmarks Area IV: Limit or Prevent Adverse Consequence of Seizures and Their Treatment Across The Lifespan Hypercapnia-induced active expiration increases in sleep and enhances ventilation in unanaesthetized rats.Active sleep unmasks apnea and delayed arousal in infant rat pups lacking central serotonin.Furthering our understanding of SUDEP: the role of animal models.Tourette Syndrome as an Independent Risk Factor for Subsequent Sleep Disorders in Children: A Nationwide Population-Based Case-Control Study.Serotonergic agents act on 5-HT3 receptors in the brain to block seizure-induced respiratory arrest in the DBA/1 mouse model of SUDEP.Dorsal raphe serotonin neurons mediate CO2- induced arousal from sleep.Hypoglossal motoneurons are endogenously activated by serotonin during the active period of circadian cycle.Activation of serotonergic neurons in the medullary caudal raphe shortens the laryngeal chemoreflex in anaesthetized neonatal rats.Impaired central respiratory chemoreflex in an experimental genetic model of epilepsy.

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5-HT2A receptor activation is necessary for CO2-induced arousal.

http://www.wikidata.org/entity/Q35867477

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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5-HT2A receptor activation is necessary for CO2-induced arousal.

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5-HT2A receptor activation is necessary for CO2-induced arousal.

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5-HT2A receptor activation is necessary for CO2-induced arousal.

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5-HT2A receptor activation is necessary for CO2-induced arousal.

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5-HT2A receptor activation is necessary for CO2-induced arousal.

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Journal of Neurophysiology

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5-HT2A receptor activation is necessary for CO2-induced arousal

@en

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Amanda MacAskill

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Haleigh R Smith

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P2860

The sudden infant death syndrome

Control of sleep and wakefulness

Chemoreception and asphyxia-induced arousal.

Sudden unexpected death in epilepsy: fatal post-ictal respiratory and arousal mechanisms

A review of central 5-HT receptors and their function

SB-699551-A (3-cyclopentyl-N-[2-(dimethylamino)ethyl]-N-[(4'-{[(2-phenylethyl)amino]methyl}-4-biphenylyl)methyl]propanamide dihydrochloride), a novel 5-ht5A receptor-selective antagonist, enhances 5-HT neuronal function: Evidence for an autoreceptor

Effects of the serotonin 5-HT2A/2C receptor agonist DOI and of the selective 5-HT2A or 5-HT2C receptor antagonists EMD 281014 and SB-243213, respectively, on sleep and waking in the rat

Lmx1b is required for maintenance of central serotonergic neurons and mice lacking central serotonergic system exhibit normal locomotor activity

Optogenetic stimulation of c1 and retrotrapezoid nucleus neurons causes sleep state-dependent cardiorespiratory stimulation and arousal in rats.

Control of breathing by raphe obscurus serotonergic neurons in mice.

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233-243

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scholarly article

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10.1152/JN.00213.2015

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1

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114

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George B Richerson

Gordon F Buchanan

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2015-04-29T00:00:00Z

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25925320

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4507958

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