A3 adenosine receptor agonist prevents the development of paclitaxel-induced neuropathic pain by modulating spinal glial-restricted redox-dependent signaling pathways.
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Mechanisms involved in the development of chemotherapy-induced neuropathyPathophysiology of Chemotherapy-Induced Peripheral Neuropathy.In vivo phenotypic screening for treating chronic neuropathic pain: modification of C2-arylethynyl group of conformationally constrained A3 adenosine receptor agonists.Engagement of the GABA to KCC2 signaling pathway contributes to the analgesic effects of A3AR agonists in neuropathic painMAPK signaling downstream to TLR4 contributes to paclitaxel-induced peripheral neuropathy.New Insights into Potential Prevention and Management Options for Chemotherapy-Induced Peripheral Neuropathy.Characterisation of Immune and Neuroinflammatory Changes Associated with Chemotherapy-Induced Peripheral Neuropathy.Activity-triggered tetrapartite neuron-glial interactions following peripheral injuryIdentification of A3 adenosine receptor agonists as novel non-narcotic analgesics.CD8+ T Cells and Endogenous IL-10 Are Required for Resolution of Chemotherapy-Induced Neuropathic Pain.Cancer and orofacial pain.Regulator Versus Effector Paradigm: Interleukin-10 as Indicator of the Switching Response.Caffeine as an opioid analgesic adjuvant in fibromyalgiaTransmission pathways and mediators as the basis for clinical pharmacology of pain.Nitroxidative Signaling Mechanisms in Pathological Pain.Targeting the S1P/S1PR1 axis mitigates cancer-induced bone pain and neuroinflammation.DRG Voltage-gated Sodium Channel 1.7 is Up-regulated in Paclitaxel-Induced Neuropathy in Rats and in Humans with Neuropathic Pain.Adenosine A3 receptor agonists: do recent findings offer new hope in chronic pain treatment?The Role of Adenosine Receptors in Psychostimulant Addiction.Chemotherapy-induced pain is promoted by enhanced spinal adenosine kinase levels via astrocyte-dependent mechanisms.Role of A3 adenosine receptor in diabetic neuropathy.Beyond symptomatic relief for chemotherapy-induced peripheral neuropathy: Targeting the source.Ferulic acid dimer as a non-opioid therapeutic for acute pain.
P2860
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P2860
A3 adenosine receptor agonist prevents the development of paclitaxel-induced neuropathic pain by modulating spinal glial-restricted redox-dependent signaling pathways.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@ast
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@en
type
label
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@ast
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@en
prefLabel
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@ast
A3 adenosine receptor agonist ...... -dependent signaling pathways.
@en
P2093
P2860
P1433
P1476
A3 adenosine receptor agonist ...... x-dependent signaling pathways
@en
P2093
Daniela Salvemini
Dillip K Tosh
Emanuela Esposito
Kali Janes
Timothy Doyle
P2860
P304
P356
10.1016/J.PAIN.2014.09.016
P407
P577
2014-09-19T00:00:00Z