Possible pathomechanisms of sudden infant death syndrome: key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants.
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Toxoplasmosis--a global threat. Correlation of latent toxoplasmosis with specific disease burden in a set of 88 countriesAnimal models for assessment of infection and inflammation: contributions to elucidating the pathophysiology of sudden infant death syndromeInner ear insult ablates the arousal response to hypoxia and hypercarbia.Maternal hypoxia increases the activity of MMPs and decreases the expression of TIMPs in the brain of neonatal ratsHigh-protein diet in lactation leads to a sudden infant death-like syndrome in miceMaternal nicotinic exposure produces a depressed hypoxic ventilatory response and subsequent death in postnatal ratsRhesus factor modulation of effects of smoking and age on psychomotor performance, intelligence, personality profile, and health in Czech soldiers.Response of smooth bronchial musculature in bronchoconstrictor substances in newborn with lung atelectasis at the respiratory distress syndrome (RDS).Both very low- and very high in vitro cytokine responses were associated with infant death in low-birth-weight children from Guinea BissauLaryngeal inflammation in the sudden infant death syndromeThe induced prostaglandin E2 pathway is a key regulator of the respiratory response to infection and hypoxia in neonates.Filamin C: a novel component of the KCNE2 interactome during hypoxia.Hematologic profile of the fetus with systemic inflammatory response syndromeAugmented hypothalamic corticotrophin-releasing hormone mRNA and corticosterone responses to stress in adult rats exposed to perinatal hypoxia.Cardiac ion channelopathies and the sudden infant death syndromeThe evolutionary significance of depression in Pathogen Host Defense (PATHOS-D).Relative trends in hospitalizations and mortality among infants by the number of vaccine doses and age, based on the Vaccine Adverse Event Reporting System (VAERS), 1990-2010Toxoplasmosis-associated difference in intelligence and personality in men depends on their Rhesus blood group but not ABO blood group.Tobacco use increases susceptibility to bacterial infection.Intrathoracic petechial hemorrhages in sudden infant death syndrome and other infant deaths: time for re-examination.Secondhand smoke exposure and serum cytokine levels in healthy children.Serotonin gene variants are unlikely to play a significant role in the pathogenesis of the sudden infant death syndrome.Migraine associated with patent foramen ovale may be caused by reactivation of cerebral toxoplasmosis triggered by arterial blood oxygen desaturation.Proposal for mechanisms of protection of supine sleep against sudden infant death syndrome: an integrated mechanism review.Peptide sharing between Bordetella pertussis proteome and human sudden death proteins: a hypothesis for a causal link.Microglia modulate respiratory rhythm generation and autoresuscitation.SIDS-CDF hypothesis revisited: explaining hypoxia in SIDS.Ventilatory response to hypoxia during endotoxemia in young rats: role of nitric oxide.Differential expression of endothelial nitric oxide synthase in coronary and cardiac tissue in hypoxic fetal guinea pig hearts.Metabolomic profiling of brain from infants who died from Sudden Infant Death Syndrome reveals novel predictive biomarkers.Respiratory inhibition after crying or gastroesophageal reflux and feeding hypoxemia in infants.Asymmetric intrauterine growth restriction is a risk factor for respiratory inhibition after crying in infants.Postmortem tandem mass spectrometry profiling for detection of infection in unexpected infant death
P2860
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P2860
Possible pathomechanisms of sudden infant death syndrome: key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Possible pathomechanisms of su ...... netically predisposed infants.
@ast
Possible pathomechanisms of su ...... netically predisposed infants.
@en
type
label
Possible pathomechanisms of su ...... netically predisposed infants.
@ast
Possible pathomechanisms of su ...... netically predisposed infants.
@en
prefLabel
Possible pathomechanisms of su ...... netically predisposed infants.
@ast
Possible pathomechanisms of su ...... netically predisposed infants.
@en
P1476
Possible pathomechanisms of su ...... netically predisposed infants.
@en
P2093
Joseph Prandota
P304
P356
10.1097/01.MJT.0000140648.30948.BD
P577
2004-11-01T00:00:00Z