Oral inflammatory diseases and systemic inflammation: role of the macrophage.
about
So depression is an inflammatory disease, but where does the inflammation come from?Thioredoxin Interacting Protein (TXNIP) and Pathogenesis of Diabetic RetinopathyPeriodontitis and bone metabolism in patients with advanced heart failure and after heart transplantation.Are proteinase 3 and cathepsin C enzymes related to pathogenesis of periodontitis?Bisgma stimulates prostaglandin E2 production in macrophages via cyclooxygenase-2, cytosolic phospholipase A2, and mitogen-activated protein kinases family.Subgingival microbial communities in Leukocyte Adhesion Deficiency and their relationship with local immunopathologyActivation and resolution of periodontal inflammation and its systemic impact.Porphyromonas gingivalis virulence factors involved in subversion of leukocytes and microbial dysbiosisMacrophage inducible C-type lectin (Mincle) recognizes glycosylated surface (S)-layer of the periodontal pathogen Tannerella forsythia.Transcriptome analysis reveals mucin 4 to be highly associated with periodontitis and identifies pleckstrin as a link to systemic diseases.Human Periodontal Stem Cells Release Specialized Proresolving Mediators and Carry Immunomodulatory and Prohealing Properties Regulated by LipoxinsDiet-Induced Obesity and Its Differential Impact on Periodontal Bone Loss.Role of inflammation in the pathogenesis of osteoarthritis: latest findings and interpretationsDietary arginine silicate inositol complex inhibits periodontal tissue loss in rats with ligature-induced periodontitis.Epigenetic Modulation in Periodontitis: Interaction of Adiponectin and JMJD3-IRF4 Axis in Macrophages.Macrophage subsets and osteoimmunology: tuning of the immunological recognition and effector systems that maintain alveolar bone.Immuno-pathogenesis of Periodontal Disease: Current and Emerging ParadigmsBlack tea theaflavins attenuate Porphyromonas gingivalis virulence properties, modulate gingival keratinocyte tight junction integrity and exert anti-inflammatory activity.Modulation of Osteoblastic Cell Efferocytosis by Bone Marrow Macrophages.The innate host response in caries and periodontitis.Enhanced Activity of the Macrophage M1/M2 Phenotypes and Phenotypic Switch to M1 in Periodontal Infection.Assessment of the cytotoxicity of a mineral trioxide aggregate-based sealer with respect to macrophage activity.Cytokine response patterns to complex biofilms by mononuclear cells discriminate patient disease status and biofilm dysbiosisTea polyphenols inhibit the activation of NF-κB and the secretion of cytokines and matrix metalloproteinases by macrophages stimulated with Fusobacterium nucleatum.Macrophages in inflammation and its resolutionThe effects of a mineral trioxide aggregate-based sealer on the production of reactive oxygen species, nitrogen species and cytokines by two macrophage subtypes.Effects of areca nut extract on lipopolysaccharides-enhanced adhesion and migration of human mononuclear leukocytes.Macrophage polarization alters post-phagocytosis survivability of the commensal Streptococcus gordonii.The Role of NrF2 in the Regulation of Periodontal Health and Disease.Emerging Concepts in the Resolution of Periodontal Inflammation: A Role for Resolvin E1.Postoperative Weight Loss Masks Metabolic Impacts of Periodontitis in Obese Rodents.Increased Root Canal Endotoxin Levels are Associated with Chronic Apical Periodontitis, Increased Oxidative and Nitrosative Stress, Major Depression, Severity of Depression, and a Lowered Quality of Life.The Anti-Inflammatory Properties of Citrus wilsonii Tanaka Extract in LPS-Induced RAW 264.7 and Primary Mouse Bone Marrow-Derived Dendritic Cells.Microvascular imaging and monitoring of human oral cavity lesions in vivo by swept-source OCT-based angiography.Dual action of highbush blueberry proanthocyanidins on Aggregatibacter actinomycetemcomitans and the host inflammatory response.Recognition of Candida albicans by gingival fibroblasts: The role of TLR2, TLR4/CD14, and MyD88.In vitro stimulatory effect of N-acetyl tryptophan-glucopyranoside against gamma radiation induced immunosuppression.N-acetyl tryptophan glucopyranoside (NATG) as a countermeasure against gamma radiation-induced immunosuppression in murine macrophage J774A.1 cells.A scientific survey on 1550 cases of oral lesions diagnosed in a Brazilian referral center.Metal ions from S-PRG filler have the potential to prevent periodontal disease.
P2860
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P2860
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@ast
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@en
type
label
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@ast
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@en
prefLabel
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@ast
Oral inflammatory diseases and systemic inflammation: role of the macrophage.
@en
P2860
P356
P1476
Oral inflammatory diseases and systemic inflammation: role of the macrophage
@en
P2093
Alpdogan Kantarci
Hatice Hasturk
P2860
P356
10.3389/FIMMU.2012.00118
P577
2012-05-16T00:00:00Z