microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice. - Test2 - elhamkhani - TriplyDB

microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice.

microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice.

http://www.wikidata.org/entity/Q36005429

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Neuromuscular Junctions as Key Contributors and Therapeutic Targets in Spinal Muscular Atrophy lncRNA/MicroRNA interactions in the vasculature New insights into the epigenetic control of satellite cells Noncoding RNAs, Emerging Regulators of Skeletal Muscle Development and Diseases HDAC4-myogenin axis as an important marker of HD-related skeletal muscle atrophy MicroRNA-206: a potential circulating biomarker candidate for amyotrophic lateral sclerosis From Nutrient to MicroRNA: a Novel Insight into Cell Signaling Involved in Skeletal Muscle Development and Disease Transcriptional and Chromatin Dynamics of Muscle Regeneration after Severe Trauma Overexpression of NF90-NF45 Represses Myogenic MicroRNA Biogenesis, Resulting in Development of Skeletal Muscle Atrophy and Centronuclear Muscle Fibers Myogenic Progenitor Cells Control Extracellular Matrix Production by Fibroblasts during Skeletal Muscle Hypertrophy The H19 long noncoding RNA gives rise to microRNAs miR-675-3p and miR-675-5p to promote skeletal muscle differentiation and regeneration.The Signature of MicroRNA Dysregulation in Muscle Paralyzed by Spinal Cord Injury Includes Downregulation of MicroRNAs that Target Myostatin Signaling Selective release of muscle-specific, extracellular microRNAs during myogenic differentiation miR-26a is required for skeletal muscle differentiation and regeneration in mice.Combined isosorbide dinitrate and ibuprofen as a novel therapy for muscular dystrophies: evidence from Phase I studies in healthy volunteers.MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms Circulating Plasma and Exosomal microRNAs as Indicators of Drug-Induced Organ Injury in Rodent Models Concise review: new frontiers in microRNA-based tissue regeneration.microRNA and skeletal muscle function: novel potential roles in exercise, diseases, and aging.miRNA transcriptome of hypertrophic skeletal muscle with overexpressed myostatin propeptide.Adult-specific functions of animal microRNAs.Exercise training in hypertension: Role of microRNAs.Epigenetic mechanisms in respiratory muscle dysfunction of patients with chronic obstructive pulmonary disease Three novel serum biomarkers, miR-1, miR-133a, and miR-206 for Limb-girdle muscular dystrophy, Facioscapulohumeral muscular dystrophy, and Becker muscular dystrophy.MicroRNAs 206 and 21 cooperate to promote RAS-extracellular signal-regulated kinase signaling by suppressing the translation of RASA1 and SPRED1 The miR-206/133b cluster is dispensable for development, survival and regeneration of skeletal muscle.Thyroid hormone regulates muscle fiber type conversion via miR-133a1 miR-206 represses hypertrophy of myogenic cells but not muscle fibers via inhibition of HDAC4 Dystromirs as serum biomarkers for monitoring the disease severity in Duchenne muscular Dystrophy.MiR-206, a key modulator of skeletal muscle development and disease.miR-30 family microRNAs regulate myogenic differentiation and provide negative feedback on the microRNA pathway.MicroRNA-26a induced by hypoxia targets HDAC6 in myogenic differentiation of embryonic stem cells.Monitoring the spatiotemporal activities of miRNAs in small animal models using molecular imaging modalities.Stem cell activation in skeletal muscle regeneration.Expression of Muscle-Specific MiRNA 206 in the Progression of Disease in a Murine SMA Model Acupuncture plus Low-Frequency Electrical Stimulation (Acu-LFES) Attenuates Diabetic Myopathy by Enhancing Muscle Regeneration.MiRNA inhibition in tissue engineering and regenerative medicine.MicroRNA delivery for regenerative medicine.miR-431 promotes differentiation and regeneration of old skeletal muscle by targeting Smad4.Identification in GRMD dog muscle of critical miRNAs involved in pathophysiology and effects associated with MuStem cell transplantation.

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P2860

microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice.

http://www.wikidata.org/entity/Q36005429

description

2012 nî lūn-bûn

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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microRNA-206 promotes skeletal ...... ne muscular dystrophy in mice.

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P2093

Andrew H Williams

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Eric N Olson

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James A Richardson

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Johanna M Maxeiner

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John M Shelton

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Ning Liu

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Rhonda Bassel-Duby

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Svetlana Bezprozvannaya

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P2860

Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy

microRNA-1 and microRNA-206 regulate skeletal muscle satellite cell proliferation and differentiation by repressing Pax7

A long noncoding RNA controls muscle differentiation by functioning as a competing endogenous RNA

Short telomeres and stem cell exhaustion model Duchenne muscular dystrophy in mdx/mTR mice

MicroRNAs: genomics, biogenesis, mechanism, and function

Switching from repression to activation: microRNAs can up-regulate translation

Cellular and molecular regulation of muscle regeneration

Absence of alpha 7 integrin in dystrophin-deficient mice causes a myopathy similar to Duchenne muscular dystrophy

Distinct target-derived signals organize formation, maturation, and maintenance of motor nerve terminals

Insulin-like growth factor-binding protein 5 (Igfbp5) compromises survival, growth, muscle development, and fertility in mice

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2054-2065

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10.1172/JCI62656

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6

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122

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22546853

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