Cervical keratinocytes containing stably replicating extrachromosomal HPV-16 are refractory to transformation by oncogenic H-Ras.
about
Human papillomavirus causes an angiogenic switch in keratinocytes which is sufficient to alter endothelial cell behavior.The PDZ binding motif of human papillomavirus type 16 E6 induces PTPN13 loss, which allows anchorage-independent growth and synergizes with ras for invasive growth.Human papillomavirus E7 induces rereplication in response to DNA damageHuman papillomavirus (HPV) type 18 induces extended growth in primary human cervical, tonsillar, or foreskin keratinocytes more effectively than other high-risk mucosal HPVs.Role of Cdc6 in re-replication in cells expressing human papillomavirus E7 oncogeneGallic acid induces apoptosis in human cervical epithelial cells containing human papillomavirus type 16 episomes.Deletion of the PDZ motif of HPV16 E6 preventing immortalization and anchorage-independent growth in human tonsil epithelial cells.
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P2860
Cervical keratinocytes containing stably replicating extrachromosomal HPV-16 are refractory to transformation by oncogenic H-Ras.
description
2006 nî lūn-bûn
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2006年の論文
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Cervical keratinocytes contain ...... sformation by oncogenic H-Ras.
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Aloysius J Klingelhutz
Felicia Barriga
Frederick E Domann
Gideon J Zamba
John H Lee
Kristi L Berger
Lubomir P Turek
Michael J Lace
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P356
10.1016/J.VIROL.2006.07.039
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P577
2006-08-30T00:00:00Z