Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements - Test2 - elhamkhani - TriplyDB

Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements

Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements

http://www.wikidata.org/entity/Q36013719

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Acute pulmonary dose-responses to inhaled multi-walled carbon nanotubes The cell biology of asthma S. mansoni bolsters anti-viral immunity in the murine respiratory tract Muc5b is the major polymeric mucin in mucus from thoroughbred horses with and without airway mucus accumulation Munc18b is an essential gene in mice whose expression is limiting for secretion by airway epithelial and mast cells Muc5b is required for airway defence Tissue distibution of murine Muc19/smgc gene products Allergic asthma is distinguished by sensitivity of allergen-specific CD4+ T cells and airway structural cells to type 2 inflammation.Enhanced interferon-gamma gene expression in T Cells and reduced ovalbumin-dependent lung eosinophilia in hypoxia-inducible factor-1-alpha-deficient mice Airway mucus function and dysfunction.Targeting insulin-like growth factor-I and insulin-like growth factor-binding protein-3 signaling pathways. A novel therapeutic approach for asthma Chronic intermittent hypoxia decreases pulmonary clearance of 99mTc-labelled particulate matter in mice.HIF-VEGF pathways are critical for chronic otitis media in Junbo and Jeff mouse mutants.Interleukin-12p40 modulates human metapneumovirus-induced pulmonary disease in an acute mouse model of infection JNK activation is responsible for mucus overproduction in smoke inhalation injury Muc5ac: a critical component mediating the rejection of enteric nematodes Effects of scutellarin on MUC5AC mucin production induced by human neutrophil elastase or interleukin 13 on airway epithelial cells The polymeric mucin Muc5ac is required for allergic airway hyperreactivity.Mucin production during prenatal and postnatal murine lung development.Hypoxia-mediated mechanism of MUC5AC production in human nasal epithelia and its implication in rhinosinusitis.NF-κB mediates IL-1β- and IL-17A-induced MUC5B expression in airway epithelial cells.IFN-γ acts on the airway epithelium to inhibit local and systemic pathology in allergic airway disease.Gli regulates MUC5AC transcription in human gastrointestinal cells Endogenous airway mucins carry glycans that bind Siglec-F and induce eosinophil apoptosis.Secretory phospholipases A2 are secreted from ciliated cells and increase mucin and eicosanoid secretion from goblet cells.Epinephrine Activation of the β2-Adrenoceptor Is Required for IL-13-Induced Mucin Production in Human Bronchial Epithelial Cells.Silver nanoparticles modify VEGF signaling pathway and mucus hypersecretion in allergic airway inflammation.Repeated Activation of Lung Invariant NKT Cells Results in Chronic Obstructive Pulmonary Disease-Like Symptoms.SNAP23 is selectively expressed in airway secretory cells and mediates baseline and stimulated mucin secretion.Regulation of airway mucin gene expression Overexpressing mouse model demonstrates the protective role of Muc5ac in the lungs.Chronic exposure to beta-blockers attenuates inflammation and mucin content in a murine asthma model.Molecular Mechanisms of Airway Hyperresponsiveness in a Murine Model of Steroid-Resistant Airway Inflammation.Acute inflammation induces insulin-like growth factor-1 to mediate Bcl-2 and Muc5ac expression in airway epithelial cells.Haemophilus influenzae lysate induces aspects of the chronic obstructive pulmonary disease phenotype Stimulation of lung innate immunity protects against lethal pneumococcal pneumonia in mice Increased mitochondrial arginine metabolism supports bioenergetics in asthma.Regulated mucin secretion from airway epithelial cells MUC5AC and inflammatory mediators associated with respiratory outcomes in the British 1946 birth cohort Interleukin-4, interleukin-13, signal transducer and activator of transcription factor 6, and allergic asthma.

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P2860

Central role of Muc5ac expression in mucous metaplasia and its regulation by conserved 5' elements

http://www.wikidata.org/entity/Q36013719

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2007 nî lūn-bûn

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Central role of Muc5ac express ...... ation by conserved 5' elements

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Central role of Muc5ac express ...... ation by conserved 5' elements

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Central role of Muc5ac express ...... ation by conserved 5' elements

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RCMB.2005-0460OC

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American Journal of Respiratory Cell and Molecular Biology

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Central role of Muc5ac express ...... ation by conserved 5' elements

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Alberto Suárez

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Christopher M Evans

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Divay Chandra

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Guillermina Pérez

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Hays W J Young

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Lindsey K Bellinghausen

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Michael J Tuvim

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Michelle G Roy

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Olatunji W Williams

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Samantha N Alexander

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P2860

Cellular response to hypoxia involves signaling via Smad proteins

Effects of reduced mucus oxygen concentration in airway Pseudomonas infections of cystic fibrosis patients

Regulation of Smad signaling through a differential recruitment of coactivators and corepressors by ZEB proteins

Targeting HIF-1 for cancer therapy

Disruption of dimerization and substrate phosphorylation inhibit factor inhibiting hypoxia-inducible factor (FIH) activity

Loss of PTEN facilitates HIF-1-mediated gene expression

Carboxyl-terminal transactivation activity of hypoxia-inducible factor 1 alpha is governed by a von Hippel-Lindau protein-independent, hydroxylation-regulated association with p300/CBP.

Mucins in cancer: protection and control of the cell surface

Interleukin-13: central mediator of allergic asthma

Nmyc plays an essential role during lung development as a dosage-sensitive regulator of progenitor cell proliferation and differentiation

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273-290

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10.1165/RCMB.2005-0460OC

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3

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37

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Burton F Dickey

Michael R. Blackburn

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2007-04-26T00:00:00Z

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17463395

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1994232

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