Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
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Toxoplasma gondii-altered host behaviour: clues as to mechanism of actionPulmonary exposure to single-walled carbon nanotubes does not affect the early immune response against Toxoplasma gondiiToxoplasma gondii seropositivity and suicide rates in womenBioactive lipids in Trypanosoma cruzi infectionNeospora caninum Activates p38 MAPK as an Evasion Mechanism against Innate ImmunityCellular renewal and improvement of local cell effector activity in peritoneal cavity in response to infectious stimuli.Immunological interactions between 2 common pathogens, Th1-inducing protozoan Toxoplasma gondii and the Th2-inducing helminth Fasciola hepaticaStrain- and Dose-Dependent Reduction of Toxoplasma gondii Burden in Pigs Is Associated with Interferon-Gamma Production by CD8+ Lymphocytes in a Heterologous Challenge Model.Host control of Mycobacterium tuberculosis is regulated by 5-lipoxygenase-dependent lipoxin production.Profiling of the perturbed metabolomic state of mouse spleen during acute and chronic toxoplasmosis.Efficient activation of T cells by human monocyte-derived dendritic cells (HMDCs) pulsed with Coxiella burnetii outer membrane protein Com1 but not by HspB-pulsed HMDCsMolecular diagnosis of toxoplasmosis in immunocompromised patients: a 3-year multicenter retrospective study.Toxoplasma gondii cyclophilin 18 regulates the proliferation and migration of murine macrophages and spleen cells.The rat Toxo1 locus directs toxoplasmosis outcome and controls parasite proliferation and spreading by macrophage-dependent mechanisms.Environmental exposures and the risk of multiple sclerosis investigated in a Norwegian case-control study.SOCS2-induced proteasome-dependent TRAF6 degradation: a common anti-inflammatory pathway for control of innate immune responsesNALP1 influences susceptibility to human congenital toxoplasmosis, proinflammatory cytokine response, and fate of Toxoplasma gondii-infected monocytic cells.Lipoxin A₄ and 15-epi-lipoxin A₄ protect against experimental cerebral malaria by inhibiting IL-12/IFN-γ in the brainToxoplasma gondii protein disulfide isomerase (TgPDI) is a novel vaccine candidate against toxoplasmosisB7RP-1-ICOS interactions are required for optimal infection-induced expansion of CD4+ Th1 and Th2 responsesVaccination with Recombinant Microneme Proteins Confers Protection against Experimental Toxoplasmosis in MiceNative and aspirin-triggered lipoxins control innate immunity by inducing proteasomal degradation of TRAF6.Macrophage migration inhibitory factor (MIF) is critical for the host resistance against Toxoplasma gondiiVaccination against murine toxoplasmosis using recombinant Toxoplasma gondii SAG3 antigen alone or in combination with Quil A.Adaptive immunity to the obligate intracellular pathogen Coxiella burnetiiReduction of Foxp3+ cells by depletion with the PC61 mAb induces mortality in resistant BALB/c mice infected with Toxoplasma gondii.Host cholesterol and inflammation as common key regulators of toxoplasmosis and artherosclerosis development.The importance of toxoplasma gondii infection in diseases presenting with headaches. Headaches and aseptic meningitis may be manifestations of the Jarisch-Herxheimer reaction.Patterns of Toxoplasma gondii cyst distribution in the forebrain associate with individual variation in predator odor avoidance and anxiety-related behavior in male Long-Evans ratsMigraine associated with patent foramen ovale may be caused by reactivation of cerebral toxoplasmosis triggered by arterial blood oxygen desaturation.Modulation of dendritic cell responses by parasites: a common strategy to survive.Toxoplasma gondii and the blood-brain barrier.Resolution of inflammation: therapeutic potential of pro-resolving lipids in type 2 diabetes mellitus and associated renal complications.Toxoplasma gondii prevents neuron degeneration by interferon-gamma-activated microglia in a mechanism involving inhibition of inducible nitric oxide synthase and transforming growth factor-beta1 production by infected microglia.Slc11a1 (Nramp-1) gene modulates immune-inflammation genes in macrophages during pristane-induced arthritis in mice.Infectious immunity in the central nervous system and brain function.Advances in understanding immunity to Toxoplasma gondii.The correlation of Tim-3 and IFN-γ expressions in mice infected with Toxoplasma gondii during gestation.Diacylglycerol kinase zeta regulates microbial recognition and host resistance to Toxoplasma gondii.Expansion of CD8+CD57+ T Cells in an Immunocompetent Patient with Acute Toxoplasmosis
P2860
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P2860
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@ast
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@en
type
label
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@ast
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@en
prefLabel
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@ast
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@en
P2860
P356
P1476
Host persistence: exploitation of anti-inflammatory pathways by Toxoplasma gondii.
@en
P2093
Julio Aliberti
P2860
P304
P356
10.1038/NRI1547
P577
2005-02-01T00:00:00Z