Innate Immune Activity Correlates with CD4 T Cell-Associated HIV-1 DNA Decline during Latency-Reversing Treatment with Panobinostat.
about
How to Define the Latent Reservoir: Tools of the TradeEpigenetic control of HIV-1 post integration latency: implications for therapySpecific Elimination of Latently HIV-1 Infected Cells Using HIV-1 Protease-Sensitive Toxin Nanocapsules.Human Galectin-9 Is a Potent Mediator of HIV Transcription and ReactivationCRISPR/gRNA-directed synergistic activation mediator (SAM) induces specific, persistent and robust reactivation of the HIV-1 latent reservoirsThe differential short- and long-term effects of HIV-1 latency-reversing agents on T cell functionHIV-1 Reservoirs During Suppressive Therapy.Specific and Stable Suppression of HIV Provirus Expression In Vitro by Chimeric Zinc Finger DNA Methyltransferase 1.Residual inflammation and viral reservoirs: alliance against an HIV cure.Promising Role of Toll-Like Receptor 8 Agonist in Concert with Prostratin for Activation of Silent HIV.Current views on interferon therapy for HIV.In vivo analysis of the effect of panobinostat on cell-associated HIV RNA and DNA levels and latent HIV infection.A Novel Toll-Like Receptor 9 Agonist, MGN1703, Enhances HIV-1 Transcription and NK Cell-Mediated Inhibition of HIV-1-Infected Autologous CD4+ T Cells.Heat Shock Protein 90 Facilitates Latent HIV Reactivation through Maintaining the Function of Positive Transcriptional Elongation Factor b (p-TEFb) under Proteasome Inhibition.HIV Latency-Reversing Agents Have Diverse Effects on Natural Killer Cell Function.Administration of Panobinostat Is Associated with Increased IL-17A mRNA in the Intestinal Epithelium of HIV-1 Patients.Control of the HIV-1 DNA Reservoir Is Associated In Vivo and In Vitro with NKp46/NKp30 (CD335 CD337) Inducibility and Interferon Gamma Production by Transcriptionally Unique NK Cells.Pegylated IFN-α-induced NK cell activation is associated with HIV-1 DNA decline in ART-treated HIV-1/HCV co-infected patients.Treatment of HIV-Infected Individuals with the Histone Deacetylase Inhibitor Panobinostat Results in Increased Numbers of Regulatory T Cells and Limits Ex Vivo Lipopolysaccharide-Induced Inflammatory Responses.The potential role for romidepsin as a component in early HIV-1 curative efforts.CD161 Defines a Functionally Distinct Subset of Pro-Inflammatory Natural Killer Cells.Distinct biomarker signatures in HIV acute infection associate with viral dynamics and reservoir size.HIV-Specific, Ex Vivo Expanded T Cell Therapy: Feasibility, Safety, and Efficacy in ART-Suppressed HIV-Infected IndividualsInterleukin-15-Stimulated Natural Killer Cells Clear HIV-1-Infected Cells following Latency ReversalExposure to Prostratin but Not Bryostatin-1 Improves Natural Killer Cell Functions Including Killing of CD4 T Cells Harboring Reactivated Human Immunodeficiency Virus
P2860
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P2860
Innate Immune Activity Correlates with CD4 T Cell-Associated HIV-1 DNA Decline during Latency-Reversing Treatment with Panobinostat.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@ast
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@en
type
label
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@ast
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@en
prefLabel
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@ast
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@en
P2093
P2860
P50
P356
P1433
P1476
Innate Immune Activity Correla ...... g Treatment with Panobinostat.
@en
P2093
Arman Bashirova
Christel R Brinkmann
Mathias Lichterfeld
Sarah Palmer
Selena Vigano
Zhengyu Ouyang
P2860
P304
10176-10189
P356
10.1128/JVI.01484-15
P407
P50
P577
2015-07-29T00:00:00Z