Sat1 is dispensable for active oxalate secretion in mouse duodenum.
about
An update on the role of the inflammasomes in the pathogenesis of kidney diseases.In female rats, ethylene glycol treatment elevates protein expression of hepatic and renal oxalate transporter sat-1 (Slc26a1) without inducing hyperoxaluria.NALP3-mediated inflammation is a principal cause of progressive renal failure in oxalate nephropathy.The role of intestinal oxalate transport in hyperoxaluria and the formation of kidney stones in animals and man.Loss of Cystic Fibrosis Transmembrane Regulator Impairs Intestinal Oxalate Secretion.Extracellular Cl(-) regulates human SO4 (2-)/anion exchanger SLC26A1 by altering pH sensitivity of anion transport.
P2860
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@ast
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@en
type
label
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@ast
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@en
prefLabel
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@ast
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@en
P2093
P2860
P1476
Sat1 is dispensable for active oxalate secretion in mouse duodenum.
@en
P2093
Daniel Markovich
Felix Knauf
Peter S Aronson
Zhirong Jiang
P2860
P356
10.1152/AJPCELL.00385.2011
P577
2012-04-18T00:00:00Z