Progression of Behavioral and CNS Deficits in a Viable Murine Model of Chronic Neuronopathic Gaucher Disease.
about
Previously unrecognized behavioral phenotype in Gaucher disease type 3Glucosylsphingosine promotes α-synuclein pathology in mutant GBA-associated Parkinson's disease.Induced Pluripotent Stem Cell Modeling of Gaucher's Disease: What Have We Learned?A convenient approach to facilitate monitoring Gaucher disease progression and therapeutic response.
P2860
Progression of Behavioral and CNS Deficits in a Viable Murine Model of Chronic Neuronopathic Gaucher Disease.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@ast
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@en
type
label
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@ast
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@en
prefLabel
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@ast
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@en
P2093
P2860
P1433
P1476
Progression of Behavioral and ...... Neuronopathic Gaucher Disease.
@en
P2093
Benjamin Liou
Brittany Swope
Gregory A Grabowski
Venette Inskeep
Wujuan Zhang
Xiaohong Wang
P2860
P304
P356
10.1371/JOURNAL.PONE.0162367
P407
P577
2016-09-06T00:00:00Z