APOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer. - Test2 - elhamkhani - TriplyDB

APOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer.

APOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer.

http://www.wikidata.org/entity/Q36133842

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APOBEC3B, a molecular driver of mutagenesis in human cancers.Mutation Processes in 293-Based Clones Overexpressing the DNA Cytosine Deaminase APOBEC3B.The 29.5 kb APOBEC3B Deletion Polymorphism Is Not Associated with Clinical Outcome of Breast Cancer.Progressive APOBEC3B mRNA expression in distant breast cancer metastases.Co-transcriptional R-loops are the main cause of estrogen-induced DNA damage.The DNA cytosine deaminase APOBEC3H haplotype I likely contributes to breast and lung cancer mutagenesis.The DNA cytosine deaminase APOBEC3B promotes tamoxifen resistance in ER-positive breast cancer.Genomic modelling of the ESR1 Y537S mutation for evaluating function and new therapeutic approaches for metastatic breast cancer.Physiological functions of programmed DNA breaks in signal-induced transcription.WeSME: uncovering mutual exclusivity of cancer drivers and beyond.APOBEC3G Expression Correlates with T-Cell Infiltration and Improved Clinical Outcomes in High-grade Serous Ovarian Carcinoma Assessment of an APOBEC3B truncating mutation, c.783delG, in patients with breast cancer.APOBEC3B is an enzymatic source of molecular alterations in esophageal squamous cell carcinoma.p53 controls expression of the DNA deaminase APOBEC3B to limit its potential mutagenic activity in cancer cells.Enzyme cycling contributes to efficient induction of genome mutagenesis by the cytidine deaminase APOBEC3B.GIGGLE: a search engine for large-scale integrated genome analysis.Viral subversion of APOBEC3s: Lessons for anti-tumor immunity and tumor immunotherapy.Genome-wide analysis reveals a role for TDG in estrogen receptor-mediated enhancer RNA transcription and 3-dimensional reorganization.Dependence receptor UNC5A restricts luminal to basal breast cancer plasticity and metastasis.Transcription-associated events affecting genomic integrity.APOBEC3B Activity Is Prevalent in Urothelial Carcinoma Cells and Only Slightly Affected by LINE-1 Expression

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APOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer.

http://www.wikidata.org/entity/Q36133842

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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APOBEC3B-Mediated Cytidine Dea ...... eptor Action in Breast Cancer.

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APOBEC3B-Mediated Cytidine Dea ...... eptor Action in Breast Cancer.

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APOBEC3B-Mediated Cytidine Dea ...... eptor Action in Breast Cancer.

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Anna Maria Ochocka

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Chun-Fui Lai

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Ekaterina Nevedomskaya

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Frances Fuller-Pace

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Hetal Patel

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Jason S Carroll

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Judit Remenyi

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Laki Buluwela

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Manikandan Periyasamy

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Naveenan Navaratnam

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Model-based analysis of ChIP-Seq (MACS)

An integrated encyclopedia of DNA elements in the human genome

A cooperative activation loop among SWI/SNF, gamma-H2AX and H3 acetylation for DNA double-strand break repair

ZNF366 is an estrogen receptor corepressor that acts through CtBP and histone deacetylases

ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks

Differential oestrogen receptor binding is associated with clinical outcome in breast cancer

Mutational processes molding the genomes of 21 breast cancers

DNA demethylation dynamics

T:G mismatch-specific thymine-DNA glycosylase (TDG) as a coregulator of transcription interacts with SRC1 family members through a novel tyrosine repeat motif.

Association of CBP/p300 acetylase and thymine DNA glycosylase links DNA repair and transcription

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108-121

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