Distinguishing the contribution of type 1 pili from that of other QseB-misregulated factors when QseC is absent during urinary tract infection.
about
The arsenal of pathogens and antivirulence therapeutic strategies for disarming themPathogen espionage: multiple bacterial adrenergic sensors eavesdrop on host communication systemsAdhesive fiber stratification in uropathogenic Escherichia coli biofilms unveils oxygen-mediated control of type 1 piliBacterial Adrenergic Sensors Regulate Virulence of Enteric Pathogens in the GutPilicide ec240 disrupts virulence circuits in uropathogenic Escherichia coli.Aberrant community architecture and attenuated persistence of uropathogenic Escherichia coli in the absence of individual IHF subunits.QseC inhibitors as an antivirulence approach for Gram-negative pathogens.Human Urine Decreases Function and Expression of Type 1 Pili in Uropathogenic Escherichia coli.Adenylate cyclase and the cyclic AMP receptor protein modulate stress resistance and virulence capacity of uropathogenic Escherichia coli.Drug and Vaccine Development for the Treatment and Prevention of Urinary Tract Infections.Genomic diversity and fitness of E. coli strains recovered from the intestinal and urinary tracts of women with recurrent urinary tract infectionMetabolic Requirements of Escherichia coli in Intracellular Bacterial Communities during Urinary Tract Infection PathogenesisFrom physiology to pharmacy: developments in the pathogenesis and treatment of recurrent urinary tract infectionsStrong cross-system interactions drive the activation of the QseB response regulator in the absence of its cognate sensor.QseBC, a two-component bacterial adrenergic receptor and global regulator of virulence in Enterobacteriaceae and Pasteurellaceae.Purine Biosynthesis Metabolically Constrains Intracellular Survival of Uropathogenic Escherichia coli.Suppression of type 1 pilus assembly in uropathogenic Escherichia coli by chemical inhibition of subunit polymerization.The yin-yang driving urinary tract infection and how proteomics can enhance research, diagnostics, and treatment.Small Molecules That Sabotage Bacterial Virulence.The Histidine Residue of QseC Is Required for Canonical Signaling between QseB and PmrB in Uropathogenic Escherichia coli.Signaling by two-component system noncognate partners promotes intrinsic tolerance to polymyxin B in uropathogenic Escherichia coli.Functional role of the type 1 pilus rod structure in mediating host-pathogen interactions.
P2860
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P2860
Distinguishing the contribution of type 1 pili from that of other QseB-misregulated factors when QseC is absent during urinary tract infection.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Distinguishing the contributio ...... uring urinary tract infection.
@ast
Distinguishing the contributio ...... uring urinary tract infection.
@en
type
label
Distinguishing the contributio ...... uring urinary tract infection.
@ast
Distinguishing the contributio ...... uring urinary tract infection.
@en
prefLabel
Distinguishing the contributio ...... uring urinary tract infection.
@ast
Distinguishing the contributio ...... uring urinary tract infection.
@en
P2093
P2860
P50
P356
P1476
Distinguishing the contributio ...... during urinary tract infection
@en
P2093
Corinne K Cusumano
Maria Kostakioti
Scott J Hultgren
P2860
P304
P356
10.1128/IAI.00283-12
P407
P577
2012-06-04T00:00:00Z