The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60. - Test2 - elhamkhani - TriplyDB

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

http://www.wikidata.org/entity/Q36228791

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Regulation of immune cell function and differentiation by the NKG2D receptor Crystal Structure of the Cowpox Virus-Encoded NKG2D Ligand OMCP MHC class I D(k) expression in hematopoietic and nonhematopoietic cells confers natural killer cell resistance to murine cytomegalovirus Human cytomegalovirus Fcγ binding proteins gp34 and gp68 antagonize Fcγ receptors I, II and III Modulation of host innate and adaptive immune defenses by cytomegalovirus: timing is everything Genetics, genomics, and evolutionary biology of NKG2D ligands.The herpes simplex virus 1 IgG fc receptor blocks antibody-mediated complement activation and antibody-dependent cellular cytotoxicity in vivo.NKG2D Receptor and Its Ligands in Host Defense The NK cell response to mouse cytomegalovirus infection affects the level and kinetics of the early CD8(+) T-cell response.Zoonotic orthopoxviruses encode a high-affinity antagonist of NKG2D.The role of NKG2D signaling in inhibition of cytotoxic T-lymphocyte lysis by the Murine cytomegalovirus immunoevasin m152/gp40 Down-regulation of NKG2D and NKp80 ligands by Kaposi's sarcoma-associated herpesvirus K5 protects against NK cell cytotoxicity.Dominant-negative FADD rescues the in vivo fitness of a cytomegalovirus lacking an antiapoptotic viral gene.NKG2D receptor signaling enhances cytolytic activity by virus-specific CD8+ T cells: evidence for a protective role in virus-induced encephalitis.Evidence for differential roles for NKG2D receptor signaling in innate host defense against coronavirus-induced neurological and liver disease.The human cytomegalovirus Fc receptor gp68 binds the Fc CH2-CH3 interface of immunoglobulin G Natural killer cells in viral infection: more than just killers.Evolutionary struggles between NK cells and viruses.Immune evasion of natural killer cells by viruses.Ly49P recognition of cytomegalovirus-infected cells expressing H2-Dk and CMV-encoded m04 correlates with the NK cell antiviral response Ly49H signaling through DAP10 is essential for optimal natural killer cell responses to mouse cytomegalovirus infection Differential susceptibility of RAE-1 isoforms to mouse cytomegalovirus.Murine NKG2D ligands: "double, double toil and trouble".Structure and function of murine cytomegalovirus MHC-I-like molecules: how the virus turned the host defense to its advantage.Human herpesvirus 7 U21 tetramerizes to associate with class I major histocompatibility complex molecules.Effect of NKG2D ligand expression on host immune responses Manipulation of NKG2D ligands by cytomegaloviruses: impact on innate and adaptive immune response.MCMV avoidance of recognition and control by NK cells.Cytomegalovirus immune evasion by perturbation of endosomal trafficking.CMV-encoded Fcγ receptors: modulators at the interface of innate and adaptive immunity.NKG2D stimulation of CD8+ T cells during priming promotes their capacity to produce cytokines in response to viral infection in mice.Viral inhibitors of NKG2D ligands for tumor surveillance.Immunoglobulin superfamily members encoded by viruses and their multiple roles in immune evasion.Kaposi's sarcoma-associated herpesvirus ORF54/dUTPase downregulates a ligand for the NK activating receptor NKp44.NCR1-deficiency diminishes the generation of protective murine cytomegalovirus antibodies by limiting follicular helper T-cell maturation.The Natural Selection of Herpesviruses and Virus-Specific NK Cell Receptors.Optimal replication of human cytomegalovirus correlates with endocytosis of glycoprotein gpUL132.The mouse cytomegalovirus glycoprotein m155 inhibits CD40 expression and restricts CD4 T cell responses.Temporal profiling of the coding and noncoding murine cytomegalovirus transcriptomes.Feeling manipulated: cytomegalovirus immune manipulation.

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P2860

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

http://www.wikidata.org/entity/Q36228791

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@ast

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@en

type

label

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@ast

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@en

prefLabel

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@ast

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@en

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Journal of Experimental Medicine

P1476

The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.

@en

P2093

Albert Zimmermann

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Hartmut Hengel

http://www.w3.org/2001/XMLSchema#string

Matthias Budt

http://www.w3.org/2001/XMLSchema#string

Milena Hasan

http://www.w3.org/2001/XMLSchema#string

Tihana Lenac

http://www.w3.org/2001/XMLSchema#string

Ulrich H Koszinowski

http://www.w3.org/2001/XMLSchema#string

Zsolt Ruzsics

http://www.w3.org/2001/XMLSchema#string

P2860

Retinoic acid early inducible genes define a ligand family for the activating NKG2D receptor in mice

Systematic excision of vector sequences from the BAC-cloned herpesvirus genome during virus reconstitution.

Selective down-regulation of the NKG2D ligand H60 by mouse cytomegalovirus m155 glycoprotein

Natural killer cells, viruses and cancer.

Cutting edge: murine UL16-binding protein-like transcript 1: a newly described transcript encoding a high-affinity ligand for murine NKG2D.

Roles of the NKG2D immunoreceptor and its ligands.

The PHD/LAP-domain protein M153R of myxomavirus is a ubiquitin ligase that induces the rapid internalization and lysosomal destruction of CD4

The molecular and functional characterization of a dominant minor H antigen, H60.

Natural killer cells and viral infections.

Antibodies are not essential for the resolution of primary cytomegalovirus infection but limit dissemination of recurrent virus.

P304

1843-1850

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scholarly article

P356

10.1084/JEM.20060514

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P433

8

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P478

203

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Martin Messerle

Astrid Krmpotic

Jurica Arapovic

P577

2006-07-10T00:00:00Z

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P5875

6953556

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16831899

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P932

2118374

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