Chronic carbamazepine administration reduces N-methyl-D-aspartate receptor-initiated signaling via arachidonic acid in rat brain - Test2 - elhamkhani - TriplyDB

Chronic carbamazepine administration reduces N-methyl-D-aspartate receptor-initiated signaling via arachidonic acid in rat brain

Chronic carbamazepine administration reduces N-methyl-D-aspartate receptor-initiated signaling via arachidonic acid in rat brain

http://www.wikidata.org/entity/Q36253556

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Bipolar disorder and mechanisms of action of mood stabilizers Lithium and the other mood stabilizers effective in bipolar disorder target the rat brain arachidonic acid cascade Chronic NMDA administration to rats increases brain pro-apoptotic factors while decreasing anti-Apoptotic factors and causes cell death.Mood-stabilizers target the brain arachidonic acid cascade.Increased excitotoxicity and neuroinflammatory markers in postmortem frontal cortex from bipolar disorder patients.Extracellular-derived calcium does not initiate in vivo neurotransmission involving docosahexaenoic acid.Mode of action of mood stabilizers: is the arachidonic acid cascade a common target?Altered arachidonic acid cascade enzymes in postmortem brain from bipolar disorder patients Chronic valproate treatment blocks D2-like receptor-mediated brain signaling via arachidonic acid in rats.Lamotrigine blocks NMDA receptor-initiated arachidonic acid signalling in rat brain: implications for its efficacy in bipolar disorder RETRACTED: Dysregulated glutamate and dopamine transporters in postmortem frontal cortex from bipolar and schizophrenic patients.Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Effects of chronic clozapine administration on markers of arachidonic acid cascade and synaptic integrity in rat brain Aging is associated with altered inflammatory, arachidonic acid cascade, and synaptic markers, influenced by epigenetic modifications, in the human frontal cortex.Chronic administration of valproic acid reduces brain NMDA signaling via arachidonic acid in unanesthetized rats.Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation.Imaging elevated brain arachidonic acid signaling in unanesthetized serotonin transporter (5-HTT)-deficient mice Intracellular- and extracellular-derived Ca(2+) influence phospholipase A(2)-mediated fatty acid release from brain phospholipids.Chronic carbamazepine administration attenuates dopamine D2-like receptor-initiated signaling via arachidonic acid in rat brain.Cuneus and fusiform cortices thickness is reduced in trigeminal neuralgia.Effect of prolonged phenytoin administration on rat brain gene expression assessed by DNA microarrays.

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P2860

Chronic carbamazepine administration reduces N-methyl-D-aspartate receptor-initiated signaling via arachidonic acid in rat brain

http://www.wikidata.org/entity/Q36253556

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J.BIOPSYCH.2007.04.021

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Biological Psychiatry

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Chronic carbamazepine administ ...... arachidonic acid in rat brain

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Jane M Bell

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Mei Chen

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Mireille Basselin

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Nelly E Villacreses

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P2860

Thirty years of clinical experience with carbamazepine in the treatment of bipolar illness: principles and practice.

Acute mania is accompanied by elevated glutamate/glutamine levels within the left dorsolateral prefrontal cortex

A simple method for the isolation and purification of total lipides from animal tissues

5'-flanking region surrounding a human cytosolic phospholipase A2 gene

Increased NMDA current and spine density in mice lacking the NMDA receptor subunit NR3A

Analysis of the convulsant-potentiating effects of lithium in rats

Modulation of synaptic plasticity by antimanic agents: the role of AMPA glutamate receptor subunit 1 synaptic expression

Chronic lithium downregulates cyclooxygenase-2 activity and prostaglandin E(2) concentration in rat brain

Chronic administration of carbamazepine down-regulates AP-2 DNA-binding activity and AP-2alpha protein expression in rat frontal cortex

Rapid phosphorylation of histone H2A.X following ionotropic glutamate receptor activation

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934-943

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